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Oxidative stress in ulcerative colitis-associated carcinogenesis.
Roessner A, Kuester D, Malfertheiner P, Schneider-Stock R. Roessner A, et al. Pathol Res Pract. 2008;204(7):511-24. doi: 10.1016/j.prp.2008.04.011. Epub 2008 Jun 20. Pathol Res Pract. 2008. PMID: 18571874 Review.
It has been known for a long time that oxidative stress in inflamed tissue can pave the way for malignant tumors, and that it is a major pathogenetic factor for the well-established correlation between inflammatory diseases and cancer. ...
It has been known for a long time that oxidative stress in inflamed tissue can pave the way for malignant tumors, and that it is a
Inhibitory effects of antisense cathepsin B cDNA transfection on invasion and motility in a human osteosarcoma cell line.
Krueger S, Haeckel C, Buehling F, Roessner A. Krueger S, et al. Among authors: roessner a. Cancer Res. 1999 Dec 1;59(23):6010-4. Cancer Res. 1999. PMID: 10606250
In ELISA assays, all antisense clones showed a significant reduction at the cathepsin B antigen level (about 70%) as compared with the control cell clones and MNNG/HOS. ...In the antisense cell clones, Northern blot analysis and reverse transcription-PCR revealed a
In ELISA assays, all antisense clones showed a significant reduction at the cathepsin B antigen level (about 70%) as compared with th …
Assessment of desmosomal components (desmoglein 1-3, plakoglobin) in cardia mucosa in relation to gastroesophageal reflux disease and Helicobacter pylori infection.
Wex T, Kuester D, Mönkemüller K, Stahr A, Fry LC, Kandulski A, Kropf S, Roessner A, Malfertheiner P. Wex T, et al. Among authors: roessner a. Hum Pathol. 2012 Oct;43(10):1745-54. doi: 10.1016/j.humpath.2011.12.024. Epub 2012 Apr 20. Hum Pathol. 2012. PMID: 22521077
Interleukin 1 beta (IL1B) gene polymorphisms are not associated with gastric carcinogenesis in Germany.
Wex T, Leodolter A, Bornschein J, Kuester D, Kähne T, Kropf S, Albrecht C, Naumann M, Roessner A, Malfertheiner P. Wex T, et al. Among authors: roessner a. Anticancer Res. 2010 Feb;30(2):505-11. Anticancer Res. 2010. PMID: 20332462
Gastroesophageal reflux disease does not lead to changes in the secretory leukocyte protease inhibitor expression in esophageal mucosa.
Wex T, Mönkemüller K, Kuester D, Weise S, Kropf S, Fry LC, Stahr A, Völkel S, Roessner A, Malfertheiner P. Wex T, et al. Among authors: roessner a. Eur J Gastroenterol Hepatol. 2009 Feb;21(2):150-8. doi: 10.1097/MEG.0b013e32830e4905. Eur J Gastroenterol Hepatol. 2009. PMID: 19212204
OBJECTIVES: Secretory leukocyte protease inhibitor (SLPI) serves as a 'defense shield' against serine proteases in inflammation. ...CONCLUSION: GERD, a chemically induced inflammation, does not affect mucosal SLPI expression in gastroesophageal mucosa....
OBJECTIVES: Secretory leukocyte protease inhibitor (SLPI) serves as a 'defense shield' against serine proteases in inflammation. ...C …
The long-term effect of Helicobacter pylori eradication on COX-1/2, 5-LOX and leukotriene receptors in patients with a risk gastritis phenotype--a link to gastric carcinogenesis.
Venerito M, Kuester D, Wex T, Roessner A, Malfertheiner P, Treiber G. Venerito M, et al. Among authors: roessner a. Cancer Lett. 2008 Nov 8;270(2):218-28. doi: 10.1016/j.canlet.2008.05.015. Epub 2008 Jun 20. Cancer Lett. 2008. PMID: 18571838
To identify the role of eicosanoid-mediated pathways for gastric carcinogenesis, the expression of enzymes (COX-1, COX-2, 5-LOX) and receptors (BLT-1, CysLTR(1)) were immunohistochemically studied in H. pylori positive patients with a risk gastritis phenotype and either su …
To identify the role of eicosanoid-mediated pathways for gastric carcinogenesis, the expression of enzymes (COX-1, COX-2, 5-LOX) and recepto …
Helicobacter pylori-induced downregulation of the secretory leukocyte protease inhibitor (SLPI) in gastric epithelial cell lines and its functional relevance for H. pylori-mediated diseases.
Wex T, Treiber G, Venerito M, Leodolter A, Peitz U, Kuester D, Hritz I, Krueger S, Roessner A, Malfertheiner P. Wex T, et al. Among authors: roessner a. Biol Chem. 2006 Jul;387(7):893-901. doi: 10.1515/BC.2006.113. Biol Chem. 2006. PMID: 16913839
The lower SLPI levels in antral biopsies of H. pylori-positive subjects were associated with a 30-fold increase (p<0.01) in neutrophil elastase activity, and a significant negative correlation was demonstrated for both parameters (R=-0.63, p=0.0002). Eradication …
The lower SLPI levels in antral biopsies of H. pylori-positive subjects were associated with a 30-fold increase (p<0.01) in neutro …
Adenocarcinomas at different positions at the gastro-oesophageal junction show distinct association with gastritis and gastric preneoplastic conditions.
Bornschein J, Dingwerth A, Selgrad M, Venerito M, Stuebs P, Frauenschlaeger K, Achilleos A, Roessner A, Malfertheiner P. Bornschein J, et al. Among authors: roessner a. Eur J Gastroenterol Hepatol. 2015 May;27(5):492-500. doi: 10.1097/MEG.0000000000000299. Eur J Gastroenterol Hepatol. 2015. PMID: 25822856
PATIENTS AND METHODS: A total of 520 patients with junctional and nonjunctional gastric cancer were assessed for the presence and degree of intestinal metaplasia, glandular atrophy and inflammation in the stomach. ...
PATIENTS AND METHODS: A total of 520 patients with junctional and nonjunctional gastric cancer were assessed for the presence and deg …
Induction of premalignant host responses by cathepsin x/z-deficiency in Helicobacter pylori-infected mice.
Krueger S, Bernhardt A, Kalinski T, Baldensperger M, Zeh M, Teller A, Adolf D, Reinheckel T, Roessner A, Kuester D. Krueger S, et al. Among authors: roessner a. PLoS One. 2013 Jul 30;8(7):e70242. doi: 10.1371/journal.pone.0070242. Print 2013. PLoS One. 2013. PMID: 23936173 Free PMC article.
To explore the Ctsz-function in the context of chronic inflammation and the development of preneoplastic lesions, we used Ctsz-deficient mice in a H. pylori gastritis model. ...Ctsz deficiency supports H. pylori-dependent development of chronic gastritis up to metaplasia, …
To explore the Ctsz-function in the context of chronic inflammation and the development of preneoplastic lesions, we used Ctsz-deficient mic …
Selective loss of codon 72 proline p53 and frequent mutational inactivation of the retained arginine allele in colorectal cancer.
Schneider-Stock R, Boltze C, Peters B, Szibor R, Landt O, Meyer F, Roessner A. Schneider-Stock R, et al. Among authors: roessner a. Neoplasia. 2004 Sep-Oct;6(5):529-35. doi: 10.1593/neo.04178. Neoplasia. 2004. PMID: 15548361 Free PMC article.
To clarify this phenomenon for colorectal cancer and to find out if this preferential loss might have any functional significance, p53 loss of heterozygosity (LOH) and p53 mutations were investigated in a group of 61 colorectal cancers and 28 liver metastases, and were cor …
To clarify this phenomenon for colorectal cancer and to find out if this preferential loss might have any functional significance, p53 loss …
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