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The ATM gene and protein: possible roles in genome surveillance, checkpoint controls and cellular defence against oxidative stress.
Rotman G, Shiloh Y. Rotman G, et al. Cancer Surv. 1997;29:285-304. Cancer Surv. 1997. PMID: 9338105 Review.
ATM deficiency and oxidative stress: a new dimension of defective response to DNA damage.
Barzilai A, Rotman G, Shiloh Y. Barzilai A, et al. Among authors: rotman g. DNA Repair (Amst). 2002 Jan 22;1(1):3-25. doi: 10.1016/s1568-7864(01)00007-6. DNA Repair (Amst). 2002. PMID: 12509294 Review.
Accumulation of DNA damage and reduced levels of nicotine adenine dinucleotide in the brains of Atm-deficient mice.
Stern N, Hochman A, Zemach N, Weizman N, Hammel I, Shiloh Y, Rotman G, Barzilai A. Stern N, et al. Among authors: rotman g. J Biol Chem. 2002 Jan 4;277(1):602-8. doi: 10.1074/jbc.M106798200. Epub 2001 Oct 25. J Biol Chem. 2002. PMID: 11679583
Nuclear retention of ATM at sites of DNA double strand breaks.
Andegeko Y, Moyal L, Mittelman L, Tsarfaty I, Shiloh Y, Rotman G. Andegeko Y, et al. Among authors: rotman g. J Biol Chem. 2001 Oct 12;276(41):38224-30. doi: 10.1074/jbc.M102986200. Epub 2001 Jul 13. J Biol Chem. 2001. PMID: 11454856
Elevated Cu/Zn-SOD exacerbates radiation sensitivity and hematopoietic abnormalities of Atm-deficient mice.
Peter Y, Rotman G, Lotem J, Elson A, Shiloh Y, Groner Y. Peter Y, et al. Among authors: rotman g. EMBO J. 2001 Apr 2;20(7):1538-46. doi: 10.1093/emboj/20.7.1538. EMBO J. 2001. PMID: 11285218 Free PMC article.
Increased oxidative stress in ataxia telangiectasia evidenced by alterations in redox state of brains from Atm-deficient mice.
Kamsler A, Daily D, Hochman A, Stern N, Shiloh Y, Rotman G, Barzilai A. Kamsler A, et al. Among authors: rotman g. Cancer Res. 2001 Mar 1;61(5):1849-54. Cancer Res. 2001. PMID: 11280737
We previously hypothesized that some aspects of A-T, such as the preferential loss of certain neurons, could result from a continuous state of increased oxidative stress (G. Rotman and Y. Shiloh, Cancer Surv., 29: 285-304, 1997; G. Rotman and Y. ...
We previously hypothesized that some aspects of A-T, such as the preferential loss of certain neurons, could result from a continuous state …
ATM is required for IkappaB kinase (IKKk) activation in response to DNA double strand breaks.
Li N, Banin S, Ouyang H, Li GC, Courtois G, Shiloh Y, Karin M, Rotman G. Li N, et al. Among authors: rotman g. J Biol Chem. 2001 Mar 23;276(12):8898-903. doi: 10.1074/jbc.M009809200. Epub 2000 Dec 12. J Biol Chem. 2001. PMID: 11114307
Ataxia telangiectasia-mutated phosphorylates Chk2 in vivo and in vitro.
Matsuoka S, Rotman G, Ogawa A, Shiloh Y, Tamai K, Elledge SJ. Matsuoka S, et al. Among authors: rotman g. Proc Natl Acad Sci U S A. 2000 Sep 12;97(19):10389-94. doi: 10.1073/pnas.190030497. Proc Natl Acad Sci U S A. 2000. PMID: 10973490 Free PMC article.
ATM: a mediator of multiple responses to genotoxic stress.
Rotman G, Shiloh Y. Rotman G, et al. Oncogene. 1999 Nov 1;18(45):6135-44. doi: 10.1038/sj.onc.1203124. Oncogene. 1999. PMID: 10557105 Review.
Selective loss of dopaminergic nigro-striatal neurons in brains of Atm-deficient mice.
Eilam R, Peter Y, Elson A, Rotman G, Shiloh Y, Groner Y, Segal M. Eilam R, et al. Among authors: rotman g. Proc Natl Acad Sci U S A. 1998 Oct 13;95(21):12653-6. doi: 10.1073/pnas.95.21.12653. Proc Natl Acad Sci U S A. 1998. PMID: 9770541 Free PMC article.
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