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Selenium, selenoproteins and the thyroid gland: interactions in health and disease.
Schomburg L. Schomburg L. Nat Rev Endocrinol. 2011 Oct 18;8(3):160-71. doi: 10.1038/nrendo.2011.174. Nat Rev Endocrinol. 2011. PMID: 22009156 Review.
Thyroid hormones rapidly and stringently regulate the messenger RNA levels of the thyrotropin-releasing hormone (TRH) receptor and the TRH-degrading ectoenzyme.
Schomburg L, Bauer K. Schomburg L, et al. Endocrinology. 1995 Aug;136(8):3480-5. doi: 10.1210/endo.136.8.7628384. Endocrinology. 1995. PMID: 7628384
Gene disruption discloses role of selenoprotein P in selenium delivery to target tissues.
Schomburg L, Schweizer U, Holtmann B, Flohé L, Sendtner M, Köhrle J. Schomburg L, et al. Biochem J. 2003 Mar 1;370(Pt 2):397-402. doi: 10.1042/BJ20021853. Biochem J. 2003. PMID: 12521380 Free PMC article.
Efficient selenium transfer from mother to offspring in selenoprotein-P-deficient mice enables dose-dependent rescue of phenotypes associated with selenium deficiency.
Schweizer U, Michaelis M, Köhrle J, Schomburg L. Schweizer U, et al. Among authors: schomburg l. Biochem J. 2004 Feb 15;378(Pt 1):21-6. doi: 10.1042/BJ20031795. Biochem J. 2004. PMID: 14664694 Free PMC article.
The neurobiology of selenium: lessons from transgenic mice.
Schweizer U, Schomburg L, Savaskan NE. Schweizer U, et al. Among authors: schomburg l. J Nutr. 2004 Apr;134(4):707-10. doi: 10.1093/jn/134.4.707. J Nutr. 2004. PMID: 15051814 Review.
Selenium and selenoproteins in mammals: extraordinary, essential, enigmatic.
Schomburg L, Schweizer U, Köhrle J. Schomburg L, et al. Cell Mol Life Sci. 2004 Aug;61(16):1988-95. doi: 10.1007/s00018-004-4114-z. Cell Mol Life Sci. 2004. PMID: 15316649
Hepatically derived selenoprotein P is a key factor for kidney but not for brain selenium supply.
Schweizer U, Streckfuss F, Pelt P, Carlson BA, Hatfield DL, Köhrle J, Schomburg L. Schweizer U, et al. Among authors: schomburg l. Biochem J. 2005 Mar 1;386(Pt 2):221-6. doi: 10.1042/BJ20041973. Biochem J. 2005. PMID: 15638810 Free PMC article.
Liver-specific inactivation of Trsp, the gene for selenocysteine tRNA, removes SePP (selenoprotein P) from plasma, causing serum selenium levels to fall from 298 microg/l to 50 microg/l and kidney selenium to decrease to 36% of wild-type levels. ...
Liver-specific inactivation of Trsp, the gene for selenocysteine tRNA, removes SePP (selenoprotein P) from plasma, causing serum selenium le …
Hepatic deiodinase activity is dispensable for the maintenance of normal circulating thyroid hormone levels in mice.
Streckfuss F, Hamann I, Schomburg L, Michaelis M, Sapin R, Klein MO, Köhrle J, Schweizer U. Streckfuss F, et al. Among authors: schomburg l. Biochem Biophys Res Commun. 2005 Nov 18;337(2):739-45. doi: 10.1016/j.bbrc.2005.09.102. Epub 2005 Sep 26. Biochem Biophys Res Commun. 2005. PMID: 16202981
Synthesis and metabolism of thyroid hormones is preferentially maintained in selenium-deficient transgenic mice.
Schomburg L, Riese C, Michaelis M, Griebert E, Klein MO, Sapin R, Schweizer U, Köhrle J. Schomburg L, et al. Endocrinology. 2006 Mar;147(3):1306-13. doi: 10.1210/en.2005-1089. Epub 2005 Dec 1. Endocrinology. 2006. PMID: 16322066
New insights into the physiological actions of selenoproteins from genetically modified mice.
Schweizer U, Schomburg L. Schweizer U, et al. Among authors: schomburg l. IUBMB Life. 2005 Nov;57(11):737-44. doi: 10.1080/15216540500364255. IUBMB Life. 2005. PMID: 16511966 Review.
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