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NOD-like receptors: ancient sentinels of the innate immune system.
Rosenstiel P, Jacobs G, Till A, Schreiber S. Rosenstiel P, et al. Among authors: schreiber s. Cell Mol Life Sci. 2008 May;65(9):1361-77. doi: 10.1007/s00018-008-7502-y. Cell Mol Life Sci. 2008. PMID: 18204816 Review.
Inflammatory bowel disease: immunologic concepts.
Schreiber S. Schreiber S. Hepatogastroenterology. 2000 Jan-Feb;47(31):15-28. Hepatogastroenterology. 2000. PMID: 10690582 Review.
., the monoclonal antibody infliximab), have been introduced into the therapeutic management of Crohn's disease. With our increasing knowledge of disease pathophysiology and the identification of key mediators, it is to be expected that the efficacy of anti-inflammatory th …
., the monoclonal antibody infliximab), have been introduced into the therapeutic management of Crohn's disease. With our increasing …
[Genetics and the environment. Has the picture become clearer?].
Schreiber S, Hampe J, Grebe J, Nikolaus S, Stoll M, Fölsch UR. Schreiber S, et al. Internist (Berl). 2002 Nov;43(11):1335-41. doi: 10.1007/s00108-002-0716-4. Internist (Berl). 2002. PMID: 12524914 Review. German. No abstract available.
TNF-alpha and IFN-gamma regulate the expression of the NOD2 (CARD15) gene in human intestinal epithelial cells.
Rosenstiel P, Fantini M, Bräutigam K, Kühbacher T, Waetzig GH, Seegert D, Schreiber S. Rosenstiel P, et al. Among authors: schreiber s. Gastroenterology. 2003 Apr;124(4):1001-9. doi: 10.1053/gast.2003.50157. Gastroenterology. 2003. PMID: 12671897
BACKGROUND & AIMS: NOD2, a member of the NOD1/Apaf-1 family, was recently identified as the first susceptibility gene for Crohn's disease. ...
BACKGROUND & AIMS: NOD2, a member of the NOD1/Apaf-1 family, was recently identified as the first susceptibility gene for Crohn's
Genetics of Crohn disease, an archetypal inflammatory barrier disease.
Schreiber S, Rosenstiel P, Albrecht M, Hampe J, Krawczak M. Schreiber S, et al. Nat Rev Genet. 2005 May;6(5):376-88. doi: 10.1038/nrg1607. Nat Rev Genet. 2005. PMID: 15861209 Review.
Association study of a functional Toll-like receptor 4 polymorphism with susceptibility to gastric mucosa-associated lymphoid tissue lymphoma.
Hellmig S, Fischbach W, Goebeler-Kolve ME, Fölsch UR, Hampe J, Schreiber S. Hellmig S, et al. Among authors: schreiber s. Leuk Lymphoma. 2005 Jun;46(6):869-72. doi: 10.1080/1042819050086451. Leuk Lymphoma. 2005. PMID: 16019531
Heterozygote genotype was significantly less frequent in patients with gastric MALT lymphoma compared to H. pylori-infected controls (4.6% vs. 11.6%, Fischer's exact P=0.019, odds ratio=0.37, 95% confidence interval 0.13-1.03). ...
Heterozygote genotype was significantly less frequent in patients with gastric MALT lymphoma compared to H. pylori-infected controls (4.6% v …
Genetic variants in matrix metalloproteinase genes are associated with development of gastric ulcer in H. Pylori infection.
Hellmig S, Ott S, Rosenstiel P, Robert Fölsch U, Hampe J, Schreiber S. Hellmig S, et al. Among authors: schreiber s. Am J Gastroenterol. 2006 Jan;101(1):29-35. doi: 10.1111/j.1572-0241.2005.00348.x. Am J Gastroenterol. 2006. PMID: 16405530
A short isoform of NOD2/CARD15, NOD2-S, is an endogenous inhibitor of NOD2/receptor-interacting protein kinase 2-induced signaling pathways.
Rosenstiel P, Huse K, Till A, Hampe J, Hellmig S, Sina C, Billmann S, von Kampen O, Waetzig GH, Platzer M, Seegert D, Schreiber S. Rosenstiel P, et al. Among authors: schreiber s. Proc Natl Acad Sci U S A. 2006 Feb 28;103(9):3280-5. doi: 10.1073/pnas.0505423103. Epub 2006 Feb 21. Proc Natl Acad Sci U S A. 2006. PMID: 16492792 Free PMC article.
NOD2-S is preferentially expressed in the human colon and is up-regulated by the antiinflammatory cytokine IL-10. Overexpression of NOD2-S down-regulates NOD2-induced NF-kappaB activation and IL-8 release. Moreover, NOD2-S also interferes with the maturation …
NOD2-S is preferentially expressed in the human colon and is up-regulated by the antiinflammatory cytokine IL-10. Overexpression of N …
Extreme heterogeneity in CARD15 and DLG5 Crohn disease-associated polymorphisms between German and Norwegian populations.
Medici V, Mascheretti S, Croucher PJ, Stoll M, Hampe J, Grebe J, Sturniolo GC, Solberg C, Jahnsen J, Moum B, Schreiber S, Vatn MH. Medici V, et al. Among authors: schreiber s. Eur J Hum Genet. 2006 Apr;14(4):459-68. doi: 10.1038/sj.ejhg.5201576. Eur J Hum Genet. 2006. PMID: 16493449
[The German competence network inflammatory bowel disease (KNCED) -- network research leads to the identification of the cause of disease and to the improvement in patient care].
Sina C, Schreiber S, Hoffmann JC, Rogler G, Schölmerich J, Zeitz M, Fölsch UR. Sina C, et al. Among authors: schreiber s. Med Klin (Munich). 2006 Feb 15;101(2):161-5. doi: 10.1007/s00063-006-1022-z. Med Klin (Munich). 2006. PMID: 16501914 German.
Aim of the KN-CED is to investigate, in their complexity, the incurable chronic diseases ulcerative colitis and Crohn's disease, particularly with regard to the causes of disease, the establishment of new therapy standards as well as patient care. ...Thus, two of the three …
Aim of the KN-CED is to investigate, in their complexity, the incurable chronic diseases ulcerative colitis and Crohn's disease, part …
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