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Relationship of Borrelia burgdorferi to its arthropod vectors.
Burgdorfer W, Anderson JF, Gern L, Lane RS, Piesman J, Spielman A. Burgdorfer W, et al. Scand J Infect Dis Suppl. 1991;77:35-40. Scand J Infect Dis Suppl. 1991. PMID: 1947809
At the IV International Conference on Lyme Borreliosis, a workshop was held to identify the unique development of the Lyme disease spirochete, Borrelia burgdorferi, in its established and suspected arthropod vectors. The following is a summary of the panel's discuss …
At the IV International Conference on Lyme Borreliosis, a workshop was held to identify the unique development of the Lyme disease sp …
Borrelia burgdorferi and Babesia microti: efficiency of transmission from reservoirs to vector ticks (Ixodes dammini).
Mather TN, Telford SR 3rd, Moore SI, Spielman A. Mather TN, et al. Exp Parasitol. 1990 Jan;70(1):55-61. doi: 10.1016/0014-4894(90)90085-q. Exp Parasitol. 1990. PMID: 2295326
In the laboratory, a similarly disproportionate rate of infection was observed among nymphal ticks, feeding as larvae, on either B. burgdorferi- or B. microti-infected mice. ...These data suggest that the efficiency of acquisition and transstadial passage of B. burgdorferi …
In the laboratory, a similarly disproportionate rate of infection was observed among nymphal ticks, feeding as larvae, on either B. b …
Norway rats as reservoir hosts for Lyme disease spirochetes on Monhegan Island, Maine.
Smith RP Jr, Rand PW, Lacombe EH, Telford SR 3rd, Rich SM, Piesman J, Spielman A. Smith RP Jr, et al. J Infect Dis. 1993 Sep;168(3):687-91. doi: 10.1093/infdis/168.3.687. J Infect Dis. 1993. PMID: 8354910
Genospecies diversity of Lyme disease spirochetes in rodent reservoirs.
Richter D, Endepols S, Ohlenbusch A, Eiffert H, Spielman A, Matuschka FR. Richter D, et al. Emerg Infect Dis. 1999 Mar-Apr;5(2):291-6. doi: 10.3201/eid0502.990218. Emerg Infect Dis. 1999. PMID: 10221886 Free PMC article.
Longitudinal assessment of the clinical and epidemiological features of Lyme disease in a defined population.
Steere AC, Taylor E, Wilson ML, Levine JF, Spielman A. Steere AC, et al. J Infect Dis. 1986 Aug;154(2):295-300. doi: 10.1093/infdis/154.2.295. J Infect Dis. 1986. PMID: 3722867
A minority of individuals, mostly children, had arthritis alone. Of 121 asymptomatic residents who gave blood samples, 10 adults (8%) had high titers of IgG antibodies to the Lyme disease spirochete; these titers sometimes persisted for years. ...
A minority of individuals, mostly children, had arthritis alone. Of 121 asymptomatic residents who gave blood samples, 10 adults (8%)
Characteristics of Lyme disease spirochetes in archived European ticks.
Matuschka FR, Ohlenbusch A, Eiffert H, Richter D, Spielman A. Matuschka FR, et al. J Infect Dis. 1996 Aug;174(2):424-6. doi: 10.1093/infdis/174.2.424. J Infect Dis. 1996. PMID: 8699081
Tick DNA could be amplified, even when ticks had been stored under museum conditions for nearly a century. Spirochetal DNA was detected by polymerase chain reaction in 6 ticks preserved for as long as a century; the oldest was collected in 1884. ...
Tick DNA could be amplified, even when ticks had been stored under museum conditions for nearly a century. Spirochetal DNA was detect …
Etiology of the acrodermatitis chronica atrophicans lesion in Lyme disease.
Ohlenbusch A, Matuschka FR, Richter D, Christen HJ, Thomssen R, Spielman A, Eiffert H. Ohlenbusch A, et al. J Infect Dis. 1996 Aug;174(2):421-3. doi: 10.1093/infdis/174.2.421. J Infect Dis. 1996. PMID: 8699080
Spirochete diversity in acrodermatitis chronica atrophicans lesions in a closely defined central European site was compared to that in the local vector population, in human erythema migrans lesions, and in cerebrospinal fluid by amplifying and sequencing a segment o …
Spirochete diversity in acrodermatitis chronica atrophicans lesions in a closely defined central European site was compared to that i …
Nondifferentiation between Lyme disease spirochetes from vector ticks and human cerebrospinal fluid.
Eiffert H, Ohlenbusch A, Christen HJ, Thomssen R, Spielman A, Matuschka FR. Eiffert H, et al. J Infect Dis. 1995 Feb;171(2):476-9. doi: 10.1093/infdis/171.2.476. J Infect Dis. 1995. PMID: 7844394
The primary structure of the outer surface protein A served as the marker of variation; a fragment of the corresponding gene was amplified by nested polymerase chain reaction and the products sequenced. ...Spirochetal neuropathology in children in our study site doe …
The primary structure of the outer surface protein A served as the marker of variation; a fragment of the corresponding gene w …
Ingestion of Lyme disease spirochetes by ticks feeding on infected hosts.
Nakayama Y, Spielman A. Nakayama Y, et al. J Infect Dis. 1989 Jul;160(1):166-7. doi: 10.1093/infdis/160.1.166. J Infect Dis. 1989. PMID: 2732513 No abstract available.
Hypersensitivity to ticks and Lyme disease risk.
Burke G, Wikel SK, Spielman A, Telford SR, McKay K, Krause PJ; Tick-borne Infection Study Group. Burke G, et al. Emerg Infect Dis. 2005 Jan;11(1):36-41. doi: 10.3201/eid1101.040303. Emerg Infect Dis. 2005. PMID: 15705320 Free PMC article.
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