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TMEM106B risk variant is implicated in the pathologic presentation of Alzheimer disease.
Rutherford NJ, Carrasquillo MM, Li M, Bisceglio G, Menke J, Josephs KA, Parisi JE, Petersen RC, Graff-Radford NR, Younkin SG, Dickson DW, Rademakers R. Rutherford NJ, et al. Neurology. 2012 Aug 14;79(7):717-8. doi: 10.1212/WNL.0b013e318264e3ac. Epub 2012 Aug 1. Neurology. 2012. PMID: 22855871 Free PMC article. No abstract available.
TMEM106B and APOE polymorphisms interact to confer risk for late-onset Alzheimer's disease in Han Chinese.
Lu RC, Wang H, Tan MS, Yu JT, Tan L. Lu RC, et al. J Neural Transm (Vienna). 2014;121(3):283-7. doi: 10.1007/s00702-013-1106-x. Epub 2013 Oct 29. J Neural Transm (Vienna). 2014. PMID: 24166182
Recent large genome-wide association studies have found variants in TMEM106B (top SNP rs1990622) as a strong risk factor for frontotemporal lobar degeneration. Moreover, the TMEM106B risk variant is also implicated in the pathol …
Recent large genome-wide association studies have found variants in TMEM106B (top SNP rs1990622) as a strong risk facto …
Inflammation and neuronal gene expression changes differ in early versus late chronic traumatic encephalopathy brain.
Labadorf A, Agus F, Aytan N, Cherry J, Mez J, McKee A, Stein TD. Labadorf A, et al. BMC Med Genomics. 2023 Mar 9;16(1):49. doi: 10.1186/s12920-023-01471-5. BMC Med Genomics. 2023. PMID: 36895005 Free PMC article.
We then identified genes and biological processes associated with total years of play as a measure of exposure, amount of tau pathology present at time of death, and the presence of APOE and TMEM106B risk variants. ...CONCLUSIONS: Together, thes …
We then identified genes and biological processes associated with total years of play as a measure of exposure, amount of tau pathology …