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Clonal populations of hematopoietic cells with paroxysmal nocturnal hemoglobinuria genotype and phenotype are present in normal individuals.
Araten DJ, Nafa K, Pakdeesuwan K, Luzzatto L. Araten DJ, et al. Proc Natl Acad Sci U S A. 1999 Apr 27;96(9):5209-14. doi: 10.1073/pnas.96.9.5209. Proc Natl Acad Sci U S A. 1999. PMID: 10220445 Free PMC article.
PNH red blood cells also were identified at a frequency of eight per million. Thus, small clones with PIG-A mutations exist commonly in normal individuals, showing clearly that PIG-A gene mutations are not sufficient for the development of PNH. Because …
PNH red blood cells also were identified at a frequency of eight per million. Thus, small clones with PIG-A mutations e …
[Immune pathophysiology of refractory anemias].
Nakao S. Nakao S. Nihon Rinsho. 2008 Mar;66(3):453-9. Nihon Rinsho. 2008. PMID: 18326316 Review. Japanese.
The significance of detecting small populations of such paroxysmal nocturnal hemoglobinuria (PNH)-type cells was substantiated by a recent observation that PNH-type cells arose from a donor-derived hematopoietic stem cell with a …
The significance of detecting small populations of such paroxysmal nocturnal hemoglobinuria (PNH)-type cells was substa …
Frequent HPRT mutations in paroxysmal nocturnal haemoglobinuria reflect T cell clonal expansion, not genomic instability.
Chen G, Zeng W, Green S, Young NS. Chen G, et al. Br J Haematol. 2004 May;125(3):383-91. doi: 10.1111/j.1365-2141.2004.04912.x. Br J Haematol. 2004. PMID: 15086421
Paroxysmal nocturnal haemoglobinuria (PNH) results from acquired mutations in the PIG-A gene of an haematopoietic stem cell, leading to defective biosynthesis of glycosylphosphatidylinositol (GPI) anchors and deficient expression of GPI-anchored protei …
Paroxysmal nocturnal haemoglobinuria (PNH) results from acquired mutations in the PIG-A gene of an haematopoietic stem
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