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Year Number of Results
1980 1
1993 2
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2000 3
2003 2
2006 1
2007 1
2008 1
2009 1
2010 1
2011 1
2012 1
2013 1
2014 1
2015 1
2016 1
2020 1
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24 results

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Page 1
Mitochondria in Diabetic Kidney Disease.
Ahmad AA, Draves SO, Rosca M. Ahmad AA, et al. Cells. 2021 Oct 29;10(11):2945. doi: 10.3390/cells10112945. Cells. 2021. PMID: 34831168 Free PMC article. Review.
The pathogenesis of DKD is multifactorial and involves activation of multiple signaling pathways with merging outcomes including thickening of the basement membrane, podocyte loss, mesangial expansion, tubular atrophy, and interstitial inflammation and …
The pathogenesis of DKD is multifactorial and involves activation of multiple signaling pathways with merging outcomes including thickeni
[Nephronophtisis].
Niaudet P, Salomon R. Niaudet P, et al. Nephrol Ther. 2006 Sep;2(4):200-6. doi: 10.1016/j.nephro.2006.06.006. Epub 2006 Aug 1. Nephrol Ther. 2006. PMID: 16966065 Review. French.
It is an autosomal recessive disease which is responsible for a urine concentration defect starting after age 2, failure to thrive and a progressive deterioration of renal function without signs of glomerular disease. Kidney size is normal. Histologic lesions concern tubular
It is an autosomal recessive disease which is responsible for a urine concentration defect starting after age 2, failure to thrive and a pro …
The complex pathology of diabetic nephropathy in humans.
Kriz W, Löwen J, Gröne HJ. Kriz W, et al. Nephrol Dial Transplant. 2023 Sep 29;38(10):2109-2119. doi: 10.1093/ndt/gfad052. Nephrol Dial Transplant. 2023. PMID: 36918205 Free PMC article. Review.
The relevant changes may be derived from two major derangements. First, a failure in the turnover of the glomerular basement membrane (GBM) based on an increased production of GBM components by podocytes and endothelial cells leading to the thickening of the …
The relevant changes may be derived from two major derangements. First, a failure in the turnover of the glomerular basement membr
Nephronophthisis: should we target cysts or fibrosis?
Slaats GG, Lilien MR, Giles RH. Slaats GG, et al. Pediatr Nephrol. 2016 Apr;31(4):545-54. doi: 10.1007/s00467-015-3162-y. Epub 2015 Jul 29. Pediatr Nephrol. 2016. PMID: 26219413 Review.
Ciliopathy nephronophthisis (NPHP), a common cause of end-stage renal disease (ESRD) in children and young adults, is characterized by disintegration of the tubular basement membrane accompanied by irregular thickening and attenuation, interstitial fib …
Ciliopathy nephronophthisis (NPHP), a common cause of end-stage renal disease (ESRD) in children and young adults, is characterized by disin …
Targeting the Pathobiology of Diabetic Kidney Disease.
Thomas MC. Thomas MC. Adv Chronic Kidney Dis. 2021 Jul;28(4):282-289. doi: 10.1053/j.ackd.2021.07.001. Adv Chronic Kidney Dis. 2021. PMID: 34922684 Review.
The pathobiology of diabetic kidney disease (DKD) involves an interplay between all the many different cell types that exist within the kidney and their shared and cumulative dysfunction in response to chronic hyperglycemia. DKD is characteriszed by morphological changes includin …
The pathobiology of diabetic kidney disease (DKD) involves an interplay between all the many different cell types that exist within the kidn …
The Mechanistic Role of Different Mediators in the Pathophysiology of Nephropathy: A Review.
Singh J, Jain A, Bhamra R, Rathi V, Dhingra AK. Singh J, et al. Curr Drug Targets. 2023;24(2):104-117. doi: 10.2174/1389450124666221026152647. Curr Drug Targets. 2023. PMID: 36305150 Review.
Growth factors like VEGF, IGFs, PDGF, EGFR and TGF-beta contribute to excessive extracellular matrix accumulation, together with thickening of the glomerular and tubular basement membranes and an increase in the mesangial matrix, leading to glomerulosclerosis …
Growth factors like VEGF, IGFs, PDGF, EGFR and TGF-beta contribute to excessive extracellular matrix accumulation, together with thickeni
Renal tubular basement membrane and collagen type IV in diabetes mellitus.
Ziyadeh FN. Ziyadeh FN. Kidney Int. 1993 Jan;43(1):114-20. doi: 10.1038/ki.1993.19. Kidney Int. 1993. PMID: 8433550 Free article. Review.
The pathogenesis of the multiple structural lesions in diabetic nephropathy remains debated, and likely is multifactorial. The uniform thickening of the renal basement membranes lining the glomerular and tubular elements appears to be a consequence of the met …
The pathogenesis of the multiple structural lesions in diabetic nephropathy remains debated, and likely is multifactorial. The uniform th
An update to the pathogenesis for monoclonal gammopathy of renal significance.
Zuo C, Zhu Y, Xu G. Zuo C, et al. Crit Rev Oncol Hematol. 2020 May;149:102926. doi: 10.1016/j.critrevonc.2020.102926. Epub 2020 Mar 2. Crit Rev Oncol Hematol. 2020. PMID: 32199132 Review.
The effect of transforming growth factor beta and platelet-derived growth factor-beta on mesangial extracellular matrix is associated with glomerular and tubular basement membrane thickening, nodular glomerulosclerosis, and interstitial fibrosis. ...
The effect of transforming growth factor beta and platelet-derived growth factor-beta on mesangial extracellular matrix is associated with g …
Oxidative stress in diabetic nephropathy.
Kashihara N, Haruna Y, Kondeti VK, Kanwar YS. Kashihara N, et al. Curr Med Chem. 2010;17(34):4256-69. doi: 10.2174/092986710793348581. Curr Med Chem. 2010. PMID: 20939814 Free PMC article. Review.
Its morphologic characteristics include glomerular hypertrophy, basement membrane thickening, mesangial expansion, tubular atrophy, interstitial fibrosis and arteriolar thickening. ...
Its morphologic characteristics include glomerular hypertrophy, basement membrane thickening, mesangial expansion, t
Pathogenesis of the podocytopathy and proteinuria in diabetic glomerulopathy.
Ziyadeh FN, Wolf G. Ziyadeh FN, et al. Curr Diabetes Rev. 2008 Feb;4(1):39-45. doi: 10.2174/157339908783502370. Curr Diabetes Rev. 2008. PMID: 18220694 Review.
Transforming growth factor-beta 1 (TGF-beta1) has emerged as an important downstream mediator for the development of renal hypertrophy and the accumulation of mesangial extracellular matrix components, but there is limited evidence to support its role in the development of albumi …
Transforming growth factor-beta 1 (TGF-beta1) has emerged as an important downstream mediator for the development of renal hypertrophy and t …
24 results