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36th International Symposium on Intensive Care and Emergency Medicine : Brussels, Belgium. 15-18 March 2016.
Bateman RM, Sharpe MD, Jagger JE, Ellis CG, Solé-Violán J, López-Rodríguez M, Herrera-Ramos E, Ruíz-Hernández J, Borderías L, Horcajada J, González-Quevedo N, Rajas O, Briones M, Rodríguez de Castro F, Rodríguez Gallego C, Esen F, Orhun G, Ergin Ozcan P, Senturk E, Ugur Yilmaz C, Orhan N, Arican N, Kaya M, Kucukerden M, Giris M, Akcan U, Bilgic Gazioglu S, Tuzun E, Riff R, Naamani O, Douvdevani A, Takegawa R, Yoshida H, Hirose T, Yamamoto N, Hagiya H, Ojima M, Akeda Y, Tasaki O, Tomono K, Shimazu T, Ono S, Kubo T, Suda S, Ueno T, Ikeda T, Hirose T, Ogura H, Takahashi H, Ojima M, Kang J, Nakamura Y, Kojima T, Shimazu T, Ikeda T, Suda S, Izutani Y, Ueno T, Ono S, Taniguchi T, O M, Dinter C, Lotz J, Eilers B, Wissmann C, Lott R, Meili MM, Schuetz PS, Hawa H, Sharshir M, Aburageila M, Salahuddin N, Chantziara V, Georgiou S, Tsimogianni A, Alexandropoulos P, Vassi A, Lagiou F, Valta M, Micha G, Chinou E, Michaloudis G, Kodaira A, Ikeda T, Ono S, Ueno T, Suda S, Izutani Y, Imaizumi H, De la Torre-Prados MV, Garcia-De la Torre A, Enguix-Armada A, Puerto-Morlan A, Perez-Valero V, Garcia-Alcantara A, Bolton N, Dudziak J, Bonney S, Tridente A, Nee P, Nicolaes G, Wiewel M, Schultz M, Wildhag… See abstract for full author list ➔ Bateman RM, et al. Crit Care. 2016 Apr 20;20(Suppl 2):94. doi: 10.1186/s13054-016-1208-6. Crit Care. 2016. PMID: 27885969 Free PMC article.
Ben Lakhal P101 - Septic shock is an independent risk factor for colistin-induced severe acute kidney injury: a retrospective cohort study B. Bilgili, M. Haliloglu, F. Gul, I. ...Jovinge P264 - Lung function six months post extra corporeal membrane oxygenation
Ben Lakhal P101 - Septic shock is an independent risk factor for colistin-induced severe acute kidney injury: a retrospective …
Tissue oxygen reactivity and cerebral autoregulation after severe traumatic brain injury.
Lang EW, Czosnyka M, Mehdorn HM. Lang EW, et al. Crit Care Med. 2003 Jan;31(1):267-71. doi: 10.1097/00003246-200301000-00042. Crit Care Med. 2003. PMID: 12545027
OBJECTIVE: To study the relationship between arterial blood pressure, intracranial pressure, directly measured brain tissue oxygenation (PtiO2), and middle cerebral artery blood flow velocity in severely head-injured patients. ...Static cereb
OBJECTIVE: To study the relationship between arterial blood pressure, intracranial pressure, directly measured brain tissue
Deferoxamine reduces endothelial ferroptosis and protects cerebrovascular function after experimental traumatic brain injury.
Liang Y, Wang Y, Sun C, Xiang Y, Deng Y. Liang Y, et al. Brain Res Bull. 2024 Feb;207:110878. doi: 10.1016/j.brainresbull.2024.110878. Epub 2024 Jan 12. Brain Res Bull. 2024. PMID: 38218407 Free article.
Cerebrovascular dysfunction resulting from traumatic brain injury (TBI) significantly contributes to poor patient outcomes. Recent studies revealed the involvement of iron metabolism in neuronal survival, yet its effect on vasculature remains unclear. ...DFO …
Cerebrovascular dysfunction resulting from traumatic brain injury (TBI) significantly contributes to poor patient outco …
Observations on the Cerebral Effects of Refractory Intracranial Hypertension After Severe Traumatic Brain Injury.
Donnelly J, Smielewski P, Adams H, Zeiler FA, Cardim D, Liu X, Fedriga M, Hutchinson P, Menon DK, Czosnyka M. Donnelly J, et al. Neurocrit Care. 2020 Apr;32(2):437-447. doi: 10.1007/s12028-019-00748-x. Neurocrit Care. 2020. PMID: 31240622 Free PMC article.
BACKGROUND: Raised intracranial pressure (ICP) is a prominent cause of morbidity and mortality after severe traumatic brain injury (TBI). However, in the clinical setting, little is known about the cerebral physiological response to severe
BACKGROUND: Raised intracranial pressure (ICP) is a prominent cause of morbidity and mortality after severe traumatic brain
Cerebral hemodynamic effects of acute hyperoxia and hyperventilation after severe traumatic brain injury.
Rangel-Castilla L, Lara LR, Gopinath S, Swank PR, Valadka A, Robertson C. Rangel-Castilla L, et al. J Neurotrauma. 2010 Oct;27(10):1853-63. doi: 10.1089/neu.2010.1339. Epub 2010 Sep 17. J Neurotrauma. 2010. PMID: 20684672 Free PMC article.
The purpose of this study was to examine the effects of hyperventilation or hyperoxia on cerebral hemodynamic parameters over time in patients with severe traumatic brain injury (TBI). ...Brain tissue oxygen reactivity
The purpose of this study was to examine the effects of hyperventilation or hyperoxia on cerebral hemodynamic parameters over time in …
Impact of Therapeutic Interventions on Cerebral Autoregulatory Function Following Severe Traumatic Brain Injury: A Secondary Analysis of the BOOST-II Study.
Prasad A, Gilmore EJ, Kim JA, Begunova L, Olexa M, Beekman R, Falcone GJ, Matouk C, Ortega-Gutierrez S, Temkin NR, Barber J, Diaz-Arrastia R, de Havenon A, Petersen NH. Prasad A, et al. Neurocrit Care. 2024 Aug;41(1):91-99. doi: 10.1007/s12028-023-01896-x. Epub 2023 Dec 29. Neurocrit Care. 2024. PMID: 38158481 Clinical Trial.
BACKGROUND: The Brain Oxygen Optimization in Severe Traumatic Brain Injury Phase II randomized controlled trial used a tier-based management protocol based on brain tissue oxygen (PbtO(2)) and intracranial pressure (I …
BACKGROUND: The Brain Oxygen Optimization in Severe Traumatic Brain Injury Phase II randomized con …
Continuous assessment of cerebrovascular autoregulation after traumatic brain injury using brain tissue oxygen pressure reactivity.
Jaeger M, Schuhmann MU, Soehle M, Meixensberger J. Jaeger M, et al. Crit Care Med. 2006 Jun;34(6):1783-8. doi: 10.1097/01.CCM.0000218413.51546.9E. Crit Care Med. 2006. PMID: 16625135
OBJECTIVE: To evaluate whether two newly developed indexes of brain tissue oxygen pressure reactivity (ORx and bPtio2) provide information on the status of cerebrovascular autoregulation after traumatic brain injury. ...PATI …
OBJECTIVE: To evaluate whether two newly developed indexes of brain tissue oxygen pressure reactivity (ORx and b …
The Effect of Red Blood Cell Transfusion on Cerebral Autoregulation in Patients with Severe Traumatic Brain Injury.
Sekhon MS, Griesdale DE, Czosnyka M, Donnelly J, Liu X, Aries MJ, Robba C, Lavinio A, Menon DK, Smielewski P, Gupta AK. Sekhon MS, et al. Neurocrit Care. 2015 Oct;23(2):210-6. doi: 10.1007/s12028-015-0141-x. Neurocrit Care. 2015. PMID: 25894454
BACKGROUND: Red blood cell (RBC) transfusion is associated with inconsistent changes in brain tissue oxygenation (PbO2). ...METHODS: Retrospective analysis of 28 severe traumatic brain injury (TBI) patients from a prospectiv …
BACKGROUND: Red blood cell (RBC) transfusion is associated with inconsistent changes in brain tissue oxygenation
Brain tissue pO2 in relation to cerebral perfusion pressure, TCD findings and TCD-CO2-reactivity after severe head injury.
Dings J, Meixensberger J, Amschler J, Hamelbeck B, Roosen K. Dings J, et al. Acta Neurochir (Wien). 1996;138(4):425-34. doi: 10.1007/BF01420305. Acta Neurochir (Wien). 1996. PMID: 8738393
As a reliable continuous monitoring of cerebral blood flow and/or cerebral oxygen metabolism is necessary to prevent secondary ischaemic events after severe head injury (SHI) the authors introduced brain tissue pO2 (ptiO2) monitori …
As a reliable continuous monitoring of cerebral blood flow and/or cerebral oxygen metabolism is necessary to prevent se …
Decision-making for decompressive craniectomy in traumatic brain injury aided by multimodality monitoring: illustrative case.
Robinson MB, Shin P, Alunday R, Cole C, Torbey MT, Carlson AP. Robinson MB, et al. J Neurosurg Case Lessons. 2021 Jun 21;1(25):CASE2197. doi: 10.3171/CASE2197. eCollection 2021 Jun 21. J Neurosurg Case Lessons. 2021. PMID: 35855080 Free PMC article.
BACKGROUND: Severe traumatic brain injury (TBI) requires individualized, physiology-based management to avoid secondary brain injury. ...OBSERVATIONS: This patient sustained a severe TBI without mass lesion and was monitored with a …
BACKGROUND: Severe traumatic brain injury (TBI) requires individualized, physiology-based management to avoid se …
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