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Pathophysiology and aetiology of impaired fasting glycaemia and impaired glucose tolerance: does it matter for prevention and treatment of type 2 diabetes?
Faerch K, Borch-Johnsen K, Holst JJ, Vaag A. Faerch K, et al. Diabetologia. 2009 Sep;52(9):1714-23. doi: 10.1007/s00125-009-1443-3. Epub 2009 Jul 10. Diabetologia. 2009. PMID: 19590846
A better understanding of the aetiology and pathophysiology of the prediabetic states might give a basis for the development of individualised prevention and treatment strategies for type 2 diabetes. ...Individuals developing combined IFG/IGT exhibit severe defects
A better understanding of the aetiology and pathophysiology of the prediabetic states might give a basis for the development o
Effects of prolonged Acipimox treatment on glucose and lipid metabolism and on in vivo insulin sensitivity in patients with non-insulin dependent diabetes mellitus.
Vaag AA, Beck-Nielsen H. Vaag AA, et al. Acta Endocrinol (Copenh). 1992 Oct;127(4):344-50. doi: 10.1530/acta.0.1270344. Acta Endocrinol (Copenh). 1992. PMID: 1449046 Clinical Trial.
The effect of prolonged treatment with Acipimox on in vivo peripheral insulin sensitivity, and on glucose and lipid metabolism, was investigated in patients with NIDDM in a double-blind study. ...
The effect of prolonged treatment with Acipimox on in vivo peripheral insulin sensitivity, and on glucose and lipid metabolism, was investig …
Hyperglycaemia compensates for the defects in insulin-mediated glucose metabolism and in the activation of glycogen synthase in the skeletal muscle of patients with type 2 (non-insulin-dependent) diabetes mellitus.
Vaag A, Damsbo P, Hother-Nielsen O, Beck-Nielsen H. Vaag A, et al. Diabetologia. 1992 Jan;35(1):80-8. doi: 10.1007/BF00400856. Diabetologia. 1992. PMID: 1541385
Insulin resistance and a defective insulin activation of the enzyme glycogen synthase in skeletal muscle during euglycaemia may have important pathophysiological implications in Type 2 (non-insulin-dependent) diabetes mellitus. Hyperglycaemia may serve to compensate for th …
Insulin resistance and a defective insulin activation of the enzyme glycogen synthase in skeletal muscle during euglycaemia may have …
Insulin resistance in skeletal muscles in patients with NIDDM.
Beck-Nielsen H, Vaag A, Damsbo P, Handberg A, Nielsen OH, Henriksen JE, Thye-Rønn P. Beck-Nielsen H, et al. Diabetes Care. 1992 Mar;15(3):418-29. doi: 10.2337/diacare.15.3.418. Diabetes Care. 1992. PMID: 1559409 Review.
However, it does not appear to be a defect in the enzyme itself, but rather a defect in the covalent activation of the enzyme system. ...In conclusion, we hypothesize that insulin resistance in skeletal muscles may be a primary genetic defect preceding the di …
However, it does not appear to be a defect in the enzyme itself, but rather a defect in the covalent activation of the enzyme …
Effects of hyperglycaemia on kidney function, atrial natriuretic factor and plasma renin in patients with insulin-dependent diabetes mellitus.
Skøtt P, Vaag A, Hother-Nielsen O, Andersen P, Bruun NE, Giese J, Beck-Nielsen H, Parving HH. Skøtt P, et al. Scand J Clin Lab Invest. 1991 Dec;51(8):715-27. doi: 10.3109/00365519109104586. Scand J Clin Lab Invest. 1991. PMID: 1666932
In normoalbuminuric patients with insulin-dependent diabetes mellitus, plasma atrial natriuretic factor (ANF), cyclic GMP and active renin and the renal clearances of [99Tcm]-diethylenetriaminepentaacetic acid (DTPA) lithium and sodium were studied on a hyperglycaemia day …
In normoalbuminuric patients with insulin-dependent diabetes mellitus, plasma atrial natriuretic factor (ANF), cyclic GMP and active renin a …
Reduced glycogen synthase activity in skeletal muscle from obese patients with and without type 2 (non-insulin-dependent) diabetes mellitus.
Damsbo P, Vaag A, Hother-Nielsen O, Beck-Nielsen H. Damsbo P, et al. Diabetologia. 1991 Apr;34(4):239-45. doi: 10.1007/BF00405082. Diabetologia. 1991. PMID: 1906024
Insulin stimulation of the glycogen synthase activity at a glucose-6-phosphate concentration of 0.1 mmol/l was absent in both obese groups, while activities increased significantly in the lean control subjects (19.6 +/- 4.2% to 45.6 +/- 6.8%, p less than 0.01). ...We concl …
Insulin stimulation of the glycogen synthase activity at a glucose-6-phosphate concentration of 0.1 mmol/l was absent in both obese g …
Prevalence of micro- and macroalbuminuria, arterial hypertension, retinopathy and large vessel disease in European type 2 (non-insulin-dependent) diabetic patients.
Gall MA, Rossing P, Skøtt P, Damsbo P, Vaag A, Bech K, Dejgaard A, Lauritzen M, Lauritzen E, Hougaard P, et al. Gall MA, et al. Diabetologia. 1991 Sep;34(9):655-61. doi: 10.1007/BF00400995. Diabetologia. 1991. PMID: 1955098
The prevalence of micro- and macroalbuminuria was determined in Type 2 (non-insulin-dependent) diabetic patients, less than 76 years of age, attending a diabetic clinic during 1987. ...This cross-sectional study has revealed a high prevalence of microalbuminuria (27 …
The prevalence of micro- and macroalbuminuria was determined in Type 2 (non-insulin-dependent) diabetic patients, less than 76 years of age, …
Effect of insulin on renal sodium handling in hyperinsulinaemic type 2 (non-insulin-dependent) diabetic patients with peripheral insulin resistance.
Skøtt P, Vaag A, Bruun NE, Hother-Nielsen O, Gall MA, Beck-Nielsen H, Parving HH. Skøtt P, et al. Diabetologia. 1991 Apr;34(4):275-81. doi: 10.1007/BF00405088. Diabetologia. 1991. PMID: 2065862
The renal clearances of 99mTc-DTPA, lithium, sodium and potassium were measured over a basal period of 90 min. Then insulin was infused at a rate of 40 mU.min-1.m-2. After an equilibration period of 90 min, the clearance measurements were repeated during a ne …
The renal clearances of 99mTc-DTPA, lithium, sodium and potassium were measured over a basal period of 90 min. Then insulin was infus …
Impairment of glucose tolerance: mechanism of action and impact on the cardiovascular system.
Beck-Nielsen H, Hother Nielsen O, Damsbo P, Vaag A, Handberg A, Henriksen JE. Beck-Nielsen H, et al. Am J Obstet Gynecol. 1990 Jul;163(1 Pt 2):292-5. doi: 10.1016/0002-9378(90)90570-w. Am J Obstet Gynecol. 1990. PMID: 2115296 Review.
On the basis of these findings, a new syndrome has been postulated-syndrome X. This syndrome may be caused by inherited insulin resistance in skeletal muscles, and secondary to that arterial hypertension, obesity, and dyslipoproteinemia may develop. ...The pill (sex steroi …
On the basis of these findings, a new syndrome has been postulated-syndrome X. This syndrome may be caused by inherited insulin resis …
Intramuscular versus subcutaneous injection of unmodified insulin: consequences for blood glucose control in patients with type 1 diabetes mellitus.
Vaag A, Pedersen KD, Lauritzen M, Hildebrandt P, Beck-Nielsen H. Vaag A, et al. Diabet Med. 1990 May;7(4):335-42. doi: 10.1111/j.1464-5491.1990.tb01401.x. Diabet Med. 1990. PMID: 2140087
When unmodified insulin (Actrapid) was given IM 30 min before breakfast, lunch, and dinner together with intermediate-acting insulin (Protaphane) SC at 2200 h, a more physiological profile of plasma free insulin and a more stable blood glucose profile was obtained t …
When unmodified insulin (Actrapid) was given IM 30 min before breakfast, lunch, and dinner together with intermediate-acting insulin (Protap …
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