Van Sande J - Search Results

1

Somatic mutations causing constitutive activity of the thyrotropin receptor are the major cause of hyperfunctioning thyroid adenomas: identification of additional mutations activating both the cyclic adenosine 3',5'-monophosphate and inositol phosphate-Ca2+ cascades.

Parma J, Van Sande J, Swillens S, Tonacchera M, Dumont J, Vassart G. Parma J, et al. Among authors: van sande j. Mol Endocrinol. 1995 Jun;9(6):725-33. doi: 10.1210/mend.9.6.8592518. Mol Endocrinol. 1995. PMID: 8592518

2

Reciprocal negative regulation between thyrotropin/3',5'-cyclic adenosine monophosphate-mediated proliferation and caveolin-1 expression in human and murine thyrocytes.

Costa MJ, Senou M, Van Rode F, Ruf J, Capello M, Dequanter D, Lothaire P, Dessy C, Dumont JE, Many MC, Van Sande J. Costa MJ, et al. Among authors: van rode f, van sande j. Mol Endocrinol. 2007 Apr;21(4):921-32. doi: 10.1210/me.2006-0328. Epub 2007 Jan 3. Mol Endocrinol. 2007. PMID: 17202321

3

Thyroid stimulating immunoglobulins, like thyrotropin activate both the cyclic AMP and the PIP2 cascades in CHO cells expressing the TSH receptor.

Van Sande J, Lejeune C, Ludgate M, Munro DS, Vassart G, Dumont JE, Mockel J. Van Sande J, et al. Mol Cell Endocrinol. 1992 Oct;88(1-3):R1-5. doi: 10.1016/0303-7207(92)90024-z. Mol Cell Endocrinol. 1992. PMID: 1360926

4

Thyrotropin activates both the cyclic AMP and the PIP2 cascades in CHO cells expressing the human cDNA of TSH receptor.

Van Sande J, Raspé E, Perret J, Lejeune C, Maenhaut C, Vassart G, Dumont JE. Van Sande J, et al. Mol Cell Endocrinol. 1990 Nov 12;74(1):R1-6. doi: 10.1016/0303-7207(90)90209-q. Mol Cell Endocrinol. 1990. PMID: 2178105

5

Control of thyroperoxidase and thyroglobulin transcription by cAMP: evidence for distinct regulatory mechanisms.

Gérard CM, Lefort A, Christophe D, Libert F, Van Sande J, Dumont JE, Vassart G. Gérard CM, et al. Among authors: van sande j. Mol Endocrinol. 1989 Dec;3(12):2110-8. doi: 10.1210/mend-3-12-2110. Mol Endocrinol. 1989. PMID: 2560810

6

Dual activation by thyrotropin of the phospholipase C and cyclic AMP cascades in human thyroid.

Laurent E, Mockel J, Van Sande J, Graff I, Dumont JE. Laurent E, et al. Among authors: van sande j. Mol Cell Endocrinol. 1987 Aug;52(3):273-8. doi: 10.1016/0303-7207(87)90055-4. Mol Cell Endocrinol. 1987. PMID: 2820816

7

Somatic and germline mutations of the TSH receptor gene in thyroid diseases.

Van Sande J, Parma J, Tonacchera M, Swillens S, Dumont J, Vassart G. Van Sande J, et al. J Clin Endocrinol Metab. 1995 Sep;80(9):2577-85. doi: 10.1210/jcem.80.9.7673398. J Clin Endocrinol Metab. 1995. PMID: 7673398 Review. No abstract available.

8

In Chinese hamster ovary K1 cells dog and human thyrotropin receptors activate both the cyclic AMP and the phosphatidylinositol 4,5-bisphosphate cascades in the presence of thyrotropin and the cyclic AMP cascade in its absence.

Van Sande J, Swillens S, Gerard C, Allgeier A, Massart C, Vassart G, Dumont JE. Van Sande J, et al. Eur J Biochem. 1995 Apr 15;229(2):338-43. Eur J Biochem. 1995. PMID: 7744056 Free article.

9

Germline mutations in the thyrotropin receptor gene cause non-autoimmune autosomal dominant hyperthyroidism.

Duprez L, Parma J, Van Sande J, Allgeier A, Leclère J, Schvartz C, Delisle MJ, Decoulx M, Orgiazzi J, Dumont J, et al. Duprez L, et al. Among authors: van sande j. Nat Genet. 1994 Jul;7(3):396-401. doi: 10.1038/ng0794-396. Nat Genet. 1994. PMID: 7920658

10

Identification and functional characterization of two new somatic mutations causing constitutive activation of the thyrotropin receptor in hyperfunctioning autonomous adenomas of the thyroid.

Paschke R, Tonacchera M, Van Sande J, Parma J, Vassart G. Paschke R, et al. Among authors: van sande j. J Clin Endocrinol Metab. 1994 Dec;79(6):1785-9. doi: 10.1210/jcem.79.6.7989485. J Clin Endocrinol Metab. 1994. PMID: 7989485

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