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Antiproteinase 3 antineutrophil cytoplasmic antibodies and disease activity in Wegener granulomatosis.
Finkielman JD, Merkel PA, Schroeder D, Hoffman GS, Spiera R, St Clair EW, Davis JC Jr, McCune WJ, Lears AK, Ytterberg SR, Hummel AM, Viss MA, Peikert T, Stone JH, Specks U; WGET Research Group. Finkielman JD, et al. Among authors: viss ma. Ann Intern Med. 2007 Nov 6;147(9):611-9. doi: 10.7326/0003-4819-147-9-200711060-00005. Ann Intern Med. 2007. PMID: 17975183 Clinical Trial.
ANCA are detectable in nearly all patients with active severe Wegener's granulomatosis.
Finkielman JD, Lee AS, Hummel AM, Viss MA, Jacob GL, Homburger HA, Peikert T, Hoffman GS, Merkel PA, Spiera R, St Clair EW, Davis JC Jr, McCune WJ, Tibbs AK, Ytterberg SR, Stone JH, Specks U; WGET Research Group. Finkielman JD, et al. Among authors: viss ma. Am J Med. 2007 Jul;120(7):643.e9-14. doi: 10.1016/j.amjmed.2006.08.016. Am J Med. 2007. PMID: 17602941
Glycosylation of proteinase 3 (PR3) is not required for its reactivity with antineutrophil cytoplasmic antibodies (ANCA) in Wegener's granulomatosis.
Finkielman JD, Merkel PA, Schroeder D, Hoffman GS, Spiera R, St Clair EW, Davis JC Jr, McCune WJ, Lears A, Ytterberg SR, Hummel AM, Viss MA, Peikert T, Stone JH, Specks U; WGET Research Group. Finkielman JD, et al. Among authors: viss ma. Clin Exp Rheumatol. 2009 Jan-Feb;27(1 Suppl 52):S45-52. Clin Exp Rheumatol. 2009. PMID: 19646346 Free PMC article. Clinical Trial.
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