White FJ - Search Results

1

Excitability of dopamine neurons: modulation and physiological consequences.

Marinelli M, Rudick CN, Hu XT, White FJ. Marinelli M, et al. Among authors: white fj. CNS Neurol Disord Drug Targets. 2006 Feb;5(1):79-97. doi: 10.2174/187152706784111542. CNS Neurol Disord Drug Targets. 2006. PMID: 16613555 Review.

2

Adaptations in the mesoaccumbens dopamine system resulting from repeated administration of dopamine D1 and D2 receptor-selective agonists: relevance to cocaine sensitization.

Henry DJ, Hu XT, White FJ. Henry DJ, et al. Among authors: white fj. Psychopharmacology (Berl). 1998 Nov;140(2):233-42. doi: 10.1007/s002130050762. Psychopharmacology (Berl). 1998. PMID: 9860115

3

Neuroadaptations involved in amphetamine and cocaine addiction.

White FJ, Kalivas PW. White FJ, et al. Drug Alcohol Depend. 1998 Jun-Jul;51(1-2):141-53. doi: 10.1016/s0376-8716(98)00072-6. Drug Alcohol Depend. 1998. PMID: 9716936 Review. No abstract available.

4

Both glutamate receptor antagonists and prefrontal cortex lesions prevent induction of cocaine sensitization and associated neuroadaptations.

Li Y, Hu XT, Berney TG, Vartanian AJ, Stine CD, Wolf ME, White FJ. Li Y, et al. Among authors: white fj. Synapse. 1999 Dec;34(3):169-80. doi: 10.1002/(SICI)1098-2396(19991201)34:3<169::AID-SYN1>3.0.CO;2-C. Synapse. 1999. PMID: 10523754

5

Enhanced vulnerability to cocaine self-administration is associated with elevated impulse activity of midbrain dopamine neurons.

Marinelli M, White FJ. Marinelli M, et al. Among authors: white fj. J Neurosci. 2000 Dec 1;20(23):8876-85. doi: 10.1523/JNEUROSCI.20-23-08876.2000. J Neurosci. 2000. PMID: 11102497 Free PMC article.

6

L-type calcium channels modulate glutamate-driven bursting activity in the nucleus accumbens in vivo.

Cooper DC, White FJ. Cooper DC, et al. Among authors: white fj. Brain Res. 2000 Oct 13;880(1-2):212-8. doi: 10.1016/s0006-8993(00)02868-7. Brain Res. 2000. PMID: 11033010

7

Altered responsiveness of medial prefrontal cortex neurons to glutamate and dopamine after withdrawal from repeated amphetamine treatment.

Peterson JD, Wolf ME, White FJ. Peterson JD, et al. Among authors: white fj. Synapse. 2000 Jun 15;36(4):342-4. doi: 10.1002/(SICI)1098-2396(20000615)36:4<342::AID-SYN11>3.0.CO;2-9. Synapse. 2000. PMID: 10819912

8

Alterations in dopamine release but not dopamine autoreceptor function in dopamine D3 receptor mutant mice.

Koeltzow TE, Xu M, Cooper DC, Hu XT, Tonegawa S, Wolf ME, White FJ. Koeltzow TE, et al. Among authors: white fj. J Neurosci. 1998 Mar 15;18(6):2231-8. doi: 10.1523/JNEUROSCI.18-06-02231.1998. J Neurosci. 1998. PMID: 9482807 Free PMC article. Review.

9

Whole-cell plasticity in cocaine withdrawal: reduced sodium currents in nucleus accumbens neurons.

Zhang XF, Hu XT, White FJ. Zhang XF, et al. Among authors: white fj. J Neurosci. 1998 Jan 1;18(1):488-98. doi: 10.1523/JNEUROSCI.18-01-00488.1998. J Neurosci. 1998. PMID: 9412525 Free PMC article.

10

Increased responsiveness of ventral tegmental area dopamine neurons to glutamate after repeated administration of cocaine or amphetamine is transient and selectively involves AMPA receptors.

Zhang XF, Hu XT, White FJ, Wolf ME. Zhang XF, et al. Among authors: white fj. J Pharmacol Exp Ther. 1997 May;281(2):699-706. J Pharmacol Exp Ther. 1997. PMID: 9152375

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