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The ERdj5-Sel1L complex facilitates cholera toxin retrotranslocation.
Williams JM, Inoue T, Banks L, Tsai B. Williams JM, et al. Mol Biol Cell. 2013 Mar;24(6):785-95. doi: 10.1091/mbc.E12-07-0522. Epub 2013 Jan 30. Mol Biol Cell. 2013. PMID: 23363602 Free PMC article.
Cholera toxin (CT) traffics from the host cell surface to the endoplasmic reticulum (ER), where the toxin's catalytic CTA1 subunit retrotranslocates to the cytosol to induce toxicity. ...
Cholera toxin (CT) traffics from the host cell surface to the endoplasmic reticulum (ER), where the toxin's catalytic CTA1 subunit re
A deubiquitinase negatively regulates retro-translocation of nonubiquitinated substrates.
Bernardi KM, Williams JM, Inoue T, Schultz A, Tsai B. Bernardi KM, et al. Mol Biol Cell. 2013 Nov;24(22):3545-56. doi: 10.1091/mbc.E13-06-0332. Epub 2013 Sep 25. Mol Biol Cell. 2013. PMID: 24068323 Free PMC article.
In this study, we probed the role of the ERAD-associated DUB, YOD1, during retro-translocation of the nonubiquitinated cholera toxin A1 (CTA1) peptide, a critical intoxication step. Through combining knockdown, overexpression, and binding studies, we demonstrated that YOD1 …
In this study, we probed the role of the ERAD-associated DUB, YOD1, during retro-translocation of the nonubiquitinated cholera toxin …
The nucleotide exchange factors Grp170 and Sil1 induce cholera toxin release from BiP to enable retrotranslocation.
Williams JM, Inoue T, Chen G, Tsai B. Williams JM, et al. Mol Biol Cell. 2015 Jun 15;26(12):2181-9. doi: 10.1091/mbc.E15-01-0014. Epub 2015 Apr 15. Mol Biol Cell. 2015. PMID: 25877869 Free PMC article.
Cholera toxin (CT) intoxicates cells by trafficking from the cell surface to the endoplasmic reticulum (ER), where the catalytic CTA1 subunit hijacks components of the ER-associated degradation (ERAD) machinery to retrotranslocate to the cytosol and induce toxicity. ...
Cholera toxin (CT) intoxicates cells by trafficking from the cell surface to the endoplasmic reticulum (ER), where the catalytic CTA1
The E3 ubiquitin ligases Hrd1 and gp78 bind to and promote cholera toxin retro-translocation.
Bernardi KM, Williams JM, Kikkert M, van Voorden S, Wiertz EJ, Ye Y, Tsai B. Bernardi KM, et al. Mol Biol Cell. 2010 Jan 1;21(1):140-51. doi: 10.1091/mbc.e09-07-0586. Epub 2009 Oct 28. Mol Biol Cell. 2010. PMID: 19864457 Free PMC article.
To cause disease, cholera toxin (CT) is transported from the cell surface to the endoplasmic reticulum (ER) lumen where the catalytic CTA1 subunit retro-translocates to the cytosol to induce pathological water secretion. ...
To cause disease, cholera toxin (CT) is transported from the cell surface to the endoplasmic reticulum (ER) lumen where the catalytic …