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Corticosteroids Are Essential for Maintaining Cardiovascular Function in Male Mice.
Cruz-Topete D, Myers PH, Foley JF, Willis MS, Cidlowski JA. Cruz-Topete D, et al. Among authors: willis ms. Endocrinology. 2016 Jul;157(7):2759-71. doi: 10.1210/en.2015-1604. Epub 2016 May 24. Endocrinology. 2016. PMID: 27219275 Free PMC article.
Wnt1/βcatenin injury response activates the epicardium and cardiac fibroblasts to promote cardiac repair.
Duan J, Gherghe C, Liu D, Hamlett E, Srikantha L, Rodgers L, Regan JN, Rojas M, Willis M, Leask A, Majesky M, Deb A. Duan J, et al. Among authors: willis m. EMBO J. 2012 Jan 18;31(2):429-42. doi: 10.1038/emboj.2011.418. Epub 2011 Nov 15. EMBO J. 2012. PMID: 22085926 Free PMC article.
Rib fractures and death from deletion of osteoblast βcatenin in adult mice is rescued by corticosteroids.
Duan J, Lee Y, Jania C, Gong J, Rojas M, Burk L, Willis M, Homeister J, Tilley S, Rubin J, Deb A. Duan J, et al. Among authors: willis m. PLoS One. 2013;8(2):e55757. doi: 10.1371/journal.pone.0055757. Epub 2013 Feb 5. PLoS One. 2013. PMID: 23393600 Free PMC article.
Essential role of stress hormone signaling in cardiomyocytes for the prevention of heart disease.
Oakley RH, Ren R, Cruz-Topete D, Bird GS, Myers PH, Boyle MC, Schneider MD, Willis MS, Cidlowski JA. Oakley RH, et al. Among authors: willis ms. Proc Natl Acad Sci U S A. 2013 Oct 15;110(42):17035-40. doi: 10.1073/pnas.1302546110. Epub 2013 Sep 30. Proc Natl Acad Sci U S A. 2013. PMID: 24082121 Free PMC article.
Kinome and Transcriptome Profiling Reveal Broad and Distinct Activities of Erlotinib, Sunitinib, and Sorafenib in the Mouse Heart and Suggest Cardiotoxicity From Combined Signal Transducer and Activator of Transcription and Epidermal Growth Factor Receptor Inhibition.
Stuhlmiller TJ, Zawistowski JS, Chen X, Sciaky N, Angus SP, Hicks ST, Parry TL, Huang W, Beak JY, Willis MS, Johnson GL, Jensen BC. Stuhlmiller TJ, et al. Among authors: willis ms. J Am Heart Assoc. 2017 Oct 19;6(10):e006635. doi: 10.1161/JAHA.117.006635. J Am Heart Assoc. 2017. PMID: 29051215 Free PMC article.
MuRF1 mono-ubiquitinates TRα to inhibit T3-induced cardiac hypertrophy in vivo.
Wadosky KM, Berthiaume JM, Tang W, Zungu M, Portman MA, Gerdes AM, Willis MS. Wadosky KM, et al. Among authors: willis ms. J Mol Endocrinol. 2016 Apr;56(3):273-90. doi: 10.1530/JME-15-0283. Epub 2016 Feb 9. J Mol Endocrinol. 2016. PMID: 26862156 Free PMC article.
Fenofibrate unexpectedly induces cardiac hypertrophy in mice lacking MuRF1.
Parry TL, Desai G, Schisler JC, Li L, Quintana MT, Stanley N, Lockyer P, Patterson C, Willis MS. Parry TL, et al. Among authors: willis ms. Cardiovasc Pathol. 2016 Mar-Apr;25(2):127-140. doi: 10.1016/j.carpath.2015.09.008. Epub 2015 Oct 29. Cardiovasc Pathol. 2016. PMID: 26764147 Free PMC article.
Non-targeted metabolomics of Brg1/Brm double-mutant cardiomyocytes reveals a novel role for SWI/SNF complexes in metabolic homeostasis.
Banerjee R, Bultman SJ, Holley D, Hillhouse C, Bain JR, Newgard CB, Muehlbauer MJ, Willis MS. Banerjee R, et al. Among authors: willis ms. Metabolomics. 2015 Oct 1;11(5):1287-1301. doi: 10.1007/s11306-015-0786-7. Metabolomics. 2015. PMID: 26392817 Free PMC article.
To determine the underlying defects, we performed nontargeted metabolomics analysis of cardiac tissue by GC/MS from a line of Brg1/Brm double-mutant mice, which lack both Brg1 and Brm in cardiomyocytes in an inducible manner, and two groups of controls. ...
To determine the underlying defects, we performed nontargeted metabolomics analysis of cardiac tissue by GC/MS from a line of Brg1/Br …
MuRF2 regulates PPARγ1 activity to protect against diabetic cardiomyopathy and enhance weight gain induced by a high fat diet.
He J, Quintana MT, Sullivan J, L Parry T, J Grevengoed T, Schisler JC, Hill JA, Yates CC, Mapanga RF, Essop MF, Stansfield WE, Bain JR, Newgard CB, Muehlbauer MJ, Han Y, Clarke BA, Willis MS. He J, et al. Among authors: willis ms. Cardiovasc Diabetol. 2015 Aug 5;14:97. doi: 10.1186/s12933-015-0252-x. Cardiovasc Diabetol. 2015. PMID: 26242235 Free PMC article.
Cardiac energy dependence on glucose increases metabolites related to glutathione and activates metabolic genes controlled by mechanistic target of rapamycin.
Schisler JC, Grevengoed TJ, Pascual F, Cooper DE, Ellis JM, Paul DS, Willis MS, Patterson C, Jia W, Coleman RA. Schisler JC, et al. Among authors: willis ms. J Am Heart Assoc. 2015 Feb 24;4(2):e001136. doi: 10.1161/JAHA.114.001136. J Am Heart Assoc. 2015. PMID: 25713290 Free PMC article.
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