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[Immunopathogenesis of Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS)].
Yamamura T, Ono H, Sato W. Yamamura T, et al. Brain Nerve. 2018 Jan;70(1):35-40. doi: 10.11477/mf.1416200947. Brain Nerve. 2018. PMID: 29348373 Japanese.
T-T cellular interaction between CD4-CD8- regulatory T cells and T cell clones presenting TCR peptide. Its implication for TCR vaccination against experimental autoimmune encephalomyelitis.
Kozovska MF, Yamamura T, Tabira T. Kozovska MF, et al. Among authors: yamamura t. J Immunol. 1996 Aug 15;157(4):1781-90. J Immunol. 1996. PMID: 8759768
The TCR peptide-specific T cells produced a soluble mediator(s) that is inhibitory for T cell activation and were protective against actively induced experimental autoimmune encephalomyelitis. These results show that V beta 17a50-68 vaccination induces regulatory CD …
The TCR peptide-specific T cells produced a soluble mediator(s) that is inhibitory for T cell activation and were protective a …
Differential expression of NK T cell V alpha 24J alpha Q invariant TCR chain in the lesions of multiple sclerosis and chronic inflammatory demyelinating polyneuropathy.
Illés Z, Kondo T, Newcombe J, Oka N, Tabira T, Yamamura T. Illés Z, et al. Among authors: yamamura t. J Immunol. 2000 Apr 15;164(8):4375-81. doi: 10.4049/jimmunol.164.8.4375. J Immunol. 2000. PMID: 10754338
Human V alpha 24+ NK T cells are a unique subset of lymphocytes expressing the V alpha 24J alpha Q invariant TCR chain. Because they can rapidly produce large amounts of regulatory cytokines, a reduction of NK T cells may lead to the development of certain autoimmun …
Human V alpha 24+ NK T cells are a unique subset of lymphocytes expressing the V alpha 24J alpha Q invariant TCR chain. Because they …
Natural killer type 2 bias in remission of multiple sclerosis.
Takahashi K, Miyake S, Kondo T, Terao K, Hatakenaka M, Hashimoto S, Yamamura T. Takahashi K, et al. Among authors: yamamura t. J Clin Invest. 2001 Mar;107(5):R23-9. doi: 10.1172/JCI11819. J Clin Invest. 2001. PMID: 11238569 Free PMC article.
These features are reminiscent of those of NK type 2 (NK2) cells that can be induced in a condition favoring functional deviation of T cells toward Th2. ...We also found that NK2 cells induced in vitro inhibit induction of Th1 cells, suggesting that the NK2-like cells in v …
These features are reminiscent of those of NK type 2 (NK2) cells that can be induced in a condition favoring functional deviation of T
New cyclooxygenase-2 inhibitors for treatment of experimental autoimmune neuritis.
Miyamoto K, Oka N, Kawasaki T, Miyake S, Yamamura T, Akiguchi I. Miyamoto K, et al. Among authors: yamamura t. Muscle Nerve. 2002 Feb;25(2):280-2. doi: 10.1002/mus.10019. Muscle Nerve. 2002. PMID: 11870698
[Involvement of NK and NKT cells in the pathogenesis of multiple sclerosis].
Yamamura T. Yamamura T. Nihon Rinsho. 2003 Aug;61(8):1329-34. Nihon Rinsho. 2003. PMID: 12962019 Review. Japanese.
Neuropeptide Y (NPY) suppresses experimental autoimmune encephalomyelitis: NPY1 receptor-specific inhibition of autoreactive Th1 responses in vivo.
Bedoui S, Miyake S, Lin Y, Miyamoto K, Oki S, Kawamura N, Beck-Sickinger A, von Hörsten S, Yamamura T. Bedoui S, et al. Among authors: yamamura t. J Immunol. 2003 Oct 1;171(7):3451-8. doi: 10.4049/jimmunol.171.7.3451. J Immunol. 2003. PMID: 14500640
Prior studies have revealed that the sympathetic nervous system regulates the clinical and pathological manifestations of experimental autoimmune encephalomyelitis (EAE), an autoimmune disease model mediated by Th1 T cells. ...We also revealed a significant inhibition of m …
Prior studies have revealed that the sympathetic nervous system regulates the clinical and pathological manifestations of experimental autoi …
Accumulation of Valpha7.2-Jalpha33 invariant T cells in human autoimmune inflammatory lesions in the nervous system.
Illés Z, Shimamura M, Newcombe J, Oka N, Yamamura T. Illés Z, et al. Among authors: yamamura t. Int Immunol. 2004 Feb;16(2):223-30. doi: 10.1093/intimm/dxh018. Int Immunol. 2004. PMID: 14734607
Using single-strand conformation polymorphism clonotype analysis, we investigated the presence of the human AV19-AJ33 T cells (V(alpha)7.2-J(alpha)33 T cells) in autopsy samples from multiple sclerosis (MS) patients as well as in nerve biopsy samples from chronic in …
Using single-strand conformation polymorphism clonotype analysis, we investigated the presence of the human AV19-AJ33 T cells (V(alph …
NKT cell-stimulating synthetic glycolipids as potential therapeutics for autoimmune disease.
Yamamura T, Miyamoto K, Illés Z, Pál E, Araki M, Miyake S. Yamamura T, et al. Curr Top Med Chem. 2004;4(5):561-7. doi: 10.2174/1568026043451221. Curr Top Med Chem. 2004. PMID: 14965307 Review.
Although T cells were previously believed to recognize only peptide antigen associated with the major histocompatibility complex (MHC), recent studies have shown that there are unique T cells specialized for recognition of lipid or glycolipid antigens bound to the M …
Although T cells were previously believed to recognize only peptide antigen associated with the major histocompatibility complex (MHC …
The clinical implication and molecular mechanism of preferential IL-4 production by modified glycolipid-stimulated NKT cells.
Oki S, Chiba A, Yamamura T, Miyake S. Oki S, et al. Among authors: yamamura t. J Clin Invest. 2004 Jun;113(11):1631-40. doi: 10.1172/JCI20862. J Clin Invest. 2004. PMID: 15173890 Free PMC article.
OCH, a sphingosine-truncated analog of alpha-galactosylceramide (alphaGC), is a potential therapeutic reagent for a variety of Th1-mediated autoimmune diseases through its selective induction of Th2 cytokines from natural killer T (NKT) cells. ...Furthermore, IFN-gamma pro …
OCH, a sphingosine-truncated analog of alpha-galactosylceramide (alphaGC), is a potential therapeutic reagent for a variety of Th1-mediated …
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