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Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1964 1
1981 1
1984 1
1985 1
1986 1
1987 2
1989 3
1990 2
1991 4
1992 3
1993 4
1994 1
1995 2
1997 1
1998 1
2002 1
2008 1
2009 2
2014 2
2015 1
2019 3
2020 3
2021 2
2022 1
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39 results
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Page 1
Celiac disease: adenovirus and alpha gliadin.
Kagnoff MF. Kagnoff MF. Curr Top Microbiol Immunol. 1989;145:67-78. doi: 10.1007/978-3-642-74594-2_6. Curr Top Microbiol Immunol. 1989. PMID: 2553337 Review. No abstract available.
Enterovirus Infections Are Associated With the Development of Celiac Disease in a Birth Cohort Study.
Oikarinen M, Puustinen L, Lehtonen J, Hakola L, Simell S, Toppari J, Ilonen J, Veijola R, Virtanen SM, Knip M, Hyöty H. Oikarinen M, et al. Front Immunol. 2021 Feb 2;11:604529. doi: 10.3389/fimmu.2020.604529. eCollection 2020. Front Immunol. 2021. PMID: 33603739 Free PMC article.
Enterovirus and adenovirus infections have been linked to the development of celiac disease. ...No difference was observed in the frequency of adenovirus infections. The findings suggest that enterovirus infections may contribute t …
Enterovirus and adenovirus infections have been linked to the development of celiac disease. ...No difference wa …
Infectious associations of Celiac disease.
Plot L, Amital H. Plot L, et al. Autoimmun Rev. 2009 Feb;8(4):316-9. doi: 10.1016/j.autrev.2008.10.001. Epub 2008 Oct 29. Autoimmun Rev. 2009. PMID: 18973831 Review.
Infectious agents have been implicated in the pathogenesis of many autoimmune diseases. Celiac disease (CD) is an autoimmune disorder affecting patients with a specific genetic predisposition (HLA-DQ2, HLA-DQ8) who are exposed to gluten, the major storage protein of …
Infectious agents have been implicated in the pathogenesis of many autoimmune diseases. Celiac disease (CD) is an autoimmune d …
Possible role for a human adenovirus in the pathogenesis of celiac disease.
Kagnoff MF, Austin RK, Hubert JJ, Bernardin JE, Kasarda DD. Kagnoff MF, et al. J Exp Med. 1984 Nov 1;160(5):1544-57. doi: 10.1084/jem.160.5.1544. J Exp Med. 1984. PMID: 6491604 Free PMC article.
Celiac disease in humans is activated by the dietary ingestion of wheat, rye, triticale, barley, and possibly oats. ...Nonetheless, less than 0.2% of individuals with those serologic HLA specificities develop celiac disease and disease is not al
Celiac disease in humans is activated by the dietary ingestion of wheat, rye, triticale, barley, and possibly oats. ...Nonethe
Review article: exposure to microbes and risk of coeliac disease.
Størdal K, Kahrs C, Tapia G, Agardh D, Kurppa K, Stene LC. Størdal K, et al. Aliment Pharmacol Ther. 2021 Jan;53(1):43-62. doi: 10.1111/apt.16161. Epub 2020 Nov 18. Aliment Pharmacol Ther. 2021. PMID: 33210316 Review.
BACKGROUND: Coeliac disease is an immune-mediated intestinal disease characterised by lifelong intolerance to dietary gluten in genetically predisposed individuals. ...Cohort studies and longitudinal studies that have sampled biological specimens before di
BACKGROUND: Coeliac disease is an immune-mediated intestinal disease characterised by lifelong intolerance to dietary g …
In this issue: autoimmunity and innate immunity.
Bot A. Bot A. Int Rev Immunol. 2014 Jan;33(1):1-2. doi: 10.3109/08830185.2014.877705. Int Rev Immunol. 2014. PMID: 24423193
More specifically, Sanz et al. cover a tantalizing form of death called pyroptosis that leads to inflammation. Samasca et al. provide a brief perspective on celiac disease. Singh and collaborators discuss in detail a newly designed leptin antagonist that could be ap …
More specifically, Sanz et al. cover a tantalizing form of death called pyroptosis that leads to inflammation. Samasca et al. provide a brie …
Antipeptide antibodies to adenovirus E1b protein indicate enhanced risk of celiac disease and dermatitis herpetiformis.
Lähdeaho ML, Lehtinen M, Rissa HR, Hyöty H, Reunala T, Mäki M. Lähdeaho ML, et al. Int Arch Allergy Immunol. 1993;101(3):272-6. doi: 10.1159/000236457. Int Arch Allergy Immunol. 1993. PMID: 8324388
The relationship between adenovirus type 12 (Ad12), celiac disease (CD) and dermatitis herpetiformis (DH) was evaluated by enzyme-linked immunosorbent assay (ELISA). ...
The relationship between adenovirus type 12 (Ad12), celiac disease (CD) and dermatitis herpetiformis (DH) was evaluated …
Is celiac disease due to molecular mimicry between gliadin peptide-HLA class II molecule-T cell interactions and those of some unidentified superantigen?
Barbeau WE, Novascone MA, Elgert KD. Barbeau WE, et al. Mol Immunol. 1997 May;34(7):535-41. doi: 10.1016/s0161-5890(97)00068-0. Mol Immunol. 1997. PMID: 9364219 Review.
This paper presents a new hypothesis for the etiology and pathogenesis of celiac disease (CD). It is our contention that CD is triggered by the binding of one or more gliadin peptides to CD-associated HLA class II molecules. ...Finally, we speculate that molecular m …
This paper presents a new hypothesis for the etiology and pathogenesis of celiac disease (CD). It is our contention that CD is …
Enterovirus infection during pregnancy is not a risk factor for celiac disease in the offspring.
Carlsson AK, Lindberg BA, Bredberg AC, Hyöty H, Ivarsson SA. Carlsson AK, et al. J Pediatr Gastroenterol Nutr. 2002 Nov;35(5):649-52. doi: 10.1097/00005176-200211000-00011. J Pediatr Gastroenterol Nutr. 2002. PMID: 12454580
BACKGROUND: There is a strong genetic influence on the susceptibility to celiac disease but it is also evident that environmental factors play a role in the development of the disease. ...Of the mothers whose children developed celiac disease, 4 …
BACKGROUND: There is a strong genetic influence on the susceptibility to celiac disease but it is also evident that environmen …
Enterovirus as trigger of coeliac disease: nested case-control study within prospective birth cohort.
Kahrs CR, Chuda K, Tapia G, Stene LC, Mårild K, Rasmussen T, Rønningen KS, Lundin KEA, Kramna L, Cinek O, Størdal K. Kahrs CR, et al. BMJ. 2019 Feb 13;364:l231. doi: 10.1136/bmj.l231. BMJ. 2019. PMID: 30760441 Free PMC article.
Adenovirus was not associated with coeliac disease. CONCLUSIONS: In this longitudinal study, a higher frequency of enterovirus, but not adenovirus, during early childhood was associated with later coeliac disease. The finding adds new inf
Adenovirus was not associated with coeliac disease. CONCLUSIONS: In this longitudinal study, a higher frequency of ente
39 results