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Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1950 1
1951 2
1961 2
1963 9
1964 14
1965 9
1966 14
1967 20
1968 22
1969 30
1970 32
1971 40
1972 35
1973 57
1974 43
1975 64
1976 50
1977 47
1978 44
1979 44
1980 56
1981 45
1982 32
1983 55
1984 53
1985 59
1986 65
1987 48
1988 50
1989 56
1990 63
1991 67
1992 69
1993 79
1994 71
1995 90
1996 96
1997 111
1998 105
1999 128
2000 162
2001 185
2002 222
2003 223
2004 241
2005 304
2006 285
2007 286
2008 294
2009 316
2010 360
2011 399
2012 464
2013 409
2014 477
2015 403
2016 422
2017 410
2018 403
2019 424
2020 431
2021 449
2022 519
2023 508
2024 403
2025 344
2026 245

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10,425 results

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Page 1
Acute liver failure.
Stravitz RT, Lee WM. Stravitz RT, et al. Lancet. 2019 Sep 7;394(10201):869-881. doi: 10.1016/S0140-6736(19)31894-X. Lancet. 2019. PMID: 31498101 Free PMC article. Review.
The absence of existing liver disease distinguishes acute liver failure from decompensated cirrhosis or acute-on-chronic liver failure. Causes of acute liver failure include paracetamol toxicity, hepatic ischaemia, viral and autoimmune hepati
The absence of existing liver disease distinguishes acute liver failure from decompensated cirrhosis or acute-on-chronic
Phenytoin-induced chronic hepatitis.
Roy AK, Mahoney HC, Levine RA. Roy AK, et al. Dig Dis Sci. 1993 Apr;38(4):740-3. doi: 10.1007/BF01316808. Dig Dis Sci. 1993. PMID: 8462373
Assays for hepatitis A, B, and C were negative. Liver biopsy was performed and showed chronic persistent hepatitis. This documentation of phenytoin-induced chronic persistent hepatitis was proven by histology and its etiology confi …
Assays for hepatitis A, B, and C were negative. Liver biopsy was performed and showed chronic persistent hepatitis
Drug-induced immunotoxicity.
Dansette PM, Bonierbale E, Minoletti C, Beaune PH, Pessayre D, Mansuy D. Dansette PM, et al. Eur J Drug Metab Pharmacokinet. 1998 Oct-Dec;23(4):443-51. doi: 10.1007/BF03189993. Eur J Drug Metab Pharmacokinet. 1998. PMID: 10323325 Review.
Immune-related drug responses are one of the most common sources of idiosyncratic toxicity. A number of organs may be the target of such reactions; however, this review concentrates mostly on the liver. Drug-induced hepatitis is generally divide …
Immune-related drug responses are one of the most common sources of idiosyncratic toxicity. A number of organs may be the target of s …
Drug-induced chronic liver injury.
Dakhoul L, Ghabril M, Chalasani N. Dakhoul L, et al. J Hepatol. 2018 Jul;69(1):248-250. doi: 10.1016/j.jhep.2018.01.001. Epub 2018 Mar 26. J Hepatol. 2018. PMID: 29598958 Review. No abstract available.
Ashwagandha-induced liver injury: A case series from Iceland and the US Drug-Induced Liver Injury Network.
Björnsson HK, Björnsson ES, Avula B, Khan IA, Jonasson JG, Ghabril M, Hayashi PH, Navarro V. Björnsson HK, et al. Liver Int. 2020 Apr;40(4):825-829. doi: 10.1111/liv.14393. Epub 2020 Feb 11. Liver Int. 2020. PMID: 31991029 Free PMC article.
Several dietary supplements containing ashwagandha are marketed in the US and Europe, but only one case of drug-induced liver injury (DILI) due to ashwagandha has been published. The aim of this case series was to describe the clinical phenotype of sus …
Several dietary supplements containing ashwagandha are marketed in the US and Europe, but only one case of drug-induced liv
The role of innate immunity in the pathogenesis of preneoplasia in drug-induced chronic hepatitis based on a mouse model.
French SW, Bardag-Gorce F, French BA, Li J, Oliva J. French SW, et al. Exp Mol Pathol. 2011 Dec;91(3):653-9. doi: 10.1016/j.yexmp.2011.07.004. Epub 2011 Jul 28. Exp Mol Pathol. 2011. PMID: 21820428 Free PMC article. Review.
Innate immunity factors such as conversion of the 26S proteasome to form the immunoproteasome and the Toll-like receptor signaling pathways are activated in chronic hepatitis induced by the carcinogenic drug DDC. Over time, preneoplastic hepatocyte phe …
Innate immunity factors such as conversion of the 26S proteasome to form the immunoproteasome and the Toll-like receptor signaling pathways …
Drug-induced hepatotoxicity.
Kaplowitz N, Aw TY, Simon FR, Stolz A. Kaplowitz N, et al. Ann Intern Med. 1986 Jun;104(6):826-39. doi: 10.7326/0003-4819-104-6-826. Ann Intern Med. 1986. PMID: 3518564 Review.
Drug-induced injury to the liver can mimic any form of acute or chronic liver disease. ...Certain drugs produce chronic liver disease that is pathologically identical to chronic active hepatitis, biliary cirrho
Drug-induced injury to the liver can mimic any form of acute or chronic liver disease. ...Certain
Ashwagandha-induced liver injury-A case series from India and literature review.
Philips CA, Valsan A, Theruvath AH, Ravindran R, Oommen TT, Rajesh S, Bishnu S, Augustine P; Liver Research Club India. Philips CA, et al. Hepatol Commun. 2023 Sep 27;7(10):e0270. doi: 10.1097/HC9.0000000000000270. eCollection 2023 Oct 1. Hepatol Commun. 2023. PMID: 37756041 Free PMC article. Review.
Study cohort was male predominant, and cholestatic hepatitis was the commonest presentation. Five patients had underlying chronic liver disease; 3 presented with acute-on-chronic liver failure, and all 3 died on follow-up. ...CONCLUSIONS: Ashwag …
Study cohort was male predominant, and cholestatic hepatitis was the commonest presentation. Five patients had underlying chronic
Nitroimidazole-induced chronic hepatitis.
Ersoz G, Karasu Z, Akarca US, Gunsar F, Yuce G, Batur Y. Ersoz G, et al. Eur J Gastroenterol Hepatol. 2001 Aug;13(8):963-6. doi: 10.1097/00042737-200108000-00015. Eur J Gastroenterol Hepatol. 2001. PMID: 11507363
Drug-induced chronic hepatitis is a rare pathological condition. There is no reported case with chronic hepatitis secondary to nitroimidazole use. We report a patient who developed nitroimidazole-induced chronic hepatitis
Drug-induced chronic hepatitis is a rare pathological condition. There is no reported case with chronic
10,425 results
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