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Mitochondrial dysfunction and lipid homeostasis.
Vamecq J, Dessein AF, Fontaine M, Briand G, Porchet N, Latruffe N, Andreolotti P, Cherkaoui-Malki M. Vamecq J, et al. Curr Drug Metab. 2012 Dec;13(10):1388-400. doi: 10.2174/138920012803762792. Curr Drug Metab. 2012. PMID: 22978394 Review.
In the first part of the review, physiological roles of mitochondria in coordinating lipid metabolism and in controlling reactive oxygen species (ROS), ATP and calcium levels are briefly presented. In a second part, clues for how mitochondria-driven alterations in lipid …
In the first part of the review, physiological roles of mitochondria in coordinating lipid metabolism and in controlling reactive oxy …
[Insulin effect on metabolism in skeletal muscles and the role of muscles in regulation of glucose homeostasis].
Buczkowska EO, Jarosz-Chobot P. Buczkowska EO, et al. Przegl Lek. 2001;58(7-8):782-7. Przegl Lek. 2001. PMID: 11769387 Review. Polish.
Insulin enhances glucose disposal, storage and oxidation in muscles. It control the metabolites required in the muscle and is involved in maintaining glucose homeostasis. ...Patients with diabetes mellitus are characterized by insulin deficiency …
Insulin enhances glucose disposal, storage and oxidation in muscles. It control the metabolites required in the muscle and is …
Lipotoxicity in the pancreatic beta cell: not just survival and function, but proliferation as well?
Sharma RB, Alonso LC. Sharma RB, et al. Curr Diab Rep. 2014 Jun;14(6):492. doi: 10.1007/s11892-014-0492-2. Curr Diab Rep. 2014. PMID: 24740729 Free PMC article. Review.
Free fatty acids (FFAs) exert both positive and negative effects on beta cell survival and insulin secretory function, depending on concentration, duration, and glucose abundance. ...Clinical observations continue to support a role for lipid biology in the ri …
Free fatty acids (FFAs) exert both positive and negative effects on beta cell survival and insulin secretory function, dependi …
Effect of insulin deprivation on muscle mitochondrial ATP production and gene transcript levels in type 1 diabetic subjects.
Karakelides H, Asmann YW, Bigelow ML, Short KR, Dhatariya K, Coenen-Schimke J, Kahl J, Mukhopadhyay D, Nair KS. Karakelides H, et al. Diabetes. 2007 Nov;56(11):2683-9. doi: 10.2337/db07-0378. Epub 2007 Jul 27. Diabetes. 2007. PMID: 17660267 Free article.
RESULTS: Withdrawal of insulin resulted in increased plasma glucose, branched chain amino acids, nonesterified fatty acids, beta-hydroxybutyrate, and urinary nitrogen but no change in bicarbonate. ...CONCLUSIONS: Insulin deficiency and associated metab …
RESULTS: Withdrawal of insulin resulted in increased plasma glucose, branched chain amino acids, nonesterified fatty
Ketone bodies alter dinitrophenol-induced glucose uptake through AMPK inhibition and oxidative stress generation in adult cardiomyocytes.
Pelletier A, Coderre L. Pelletier A, et al. Am J Physiol Endocrinol Metab. 2007 May;292(5):E1325-32. doi: 10.1152/ajpendo.00186.2006. Epub 2007 Jan 16. Am J Physiol Endocrinol Metab. 2007. PMID: 17227964 Free article.
In aerobic conditions, the heart preferentially oxidizes fatty acids. However, during metabolic stress, glucose becomes the major energy source, and enhanced glucose uptake has a protective effect on heart function and cardiomyocyte survi …
In aerobic conditions, the heart preferentially oxidizes fatty acids. However, during metabolic stress, glucose
Diabetic patients without vascular complications display enhanced basal platelet activation and decreased antioxidant status.
Véricel E, Januel C, Carreras M, Moulin P, Lagarde M. Véricel E, et al. Diabetes. 2004 Apr;53(4):1046-51. doi: 10.2337/diabetes.53.4.1046. Diabetes. 2004. PMID: 15047620 Free article.
Thus, the aim of the present study was to evaluate basal arachidonic acid metabolism in relation to the redox status of platelets in both type 1 and type 2 diabetic patients, in the absence of vascular complications, as compared with resp …
Thus, the aim of the present study was to evaluate basal arachidonic acid metabolism in relation to the redox status of platel …
Increased susceptibility to lipid oxidation of low-density lipoproteins and erythrocyte membranes from diabetic patients.
Rabini RA, Fumelli P, Galassi R, Dousset N, Taus M, Ferretti G, Mazzanti L, Curatola G, Solera ML, Valdiguié P. Rabini RA, et al. Metabolism. 1994 Dec;43(12):1470-4. doi: 10.1016/0026-0495(94)90003-5. Metabolism. 1994. PMID: 7990698
The susceptibility to peroxidation was determined by measuring the production of thiobarbituric acid-reactive substances (TBARS) after the incubation. The following parameters were also evaluated: plasma glucose, triglycerides (TG), phospholipids (PL), total and hig …
The susceptibility to peroxidation was determined by measuring the production of thiobarbituric acid-reactive substances (TBARS) afte …
Lipid advanced glycosylation: pathway for lipid oxidation in vivo.
Bucala R, Makita Z, Koschinsky T, Cerami A, Vlassara H. Bucala R, et al. Proc Natl Acad Sci U S A. 1993 Jul 15;90(14):6434-8. doi: 10.1073/pnas.90.14.6434. Proc Natl Acad Sci U S A. 1993. PMID: 8341651 Free PMC article.
To address potential mechanisms for oxidative modification of lipids in vivo, we investigated the possibility that phospholipids react directly with glucose to form advanced glycosylation end products (AGEs) that then initiate lipid oxidation. Phospholipid-li …
To address potential mechanisms for oxidative modification of lipids in vivo, we investigated the possibility that phospholipids reac …
[New concepts in the therapy of diabetic cardiopathies].
Pogátsa G. Pogátsa G. Orv Hetil. 1997 Jun 8;138(23):1491-4. Orv Hetil. 1997. PMID: 9254364 Review. Hungarian.
Most reasonable cause of diabetic cardiopathy might be the impaired energy fuel supply and metabolism producing inevitably a hypotic condition and needing myocardial cytoprotection. ...Therefore, it is worth to stimulate aerobic glycolysis to protect diabetic heart …
Most reasonable cause of diabetic cardiopathy might be the impaired energy fuel supply and metabolism producing inevitably a h …
Increased susceptibility to peroxidation of VLDL from non-insulin-dependent diabetic patients: a possible correlation with fatty acid composition.
Rabini RA, Tesei M, Galeazzi T, Dousset N, Ferretti G, Mazzanti L. Rabini RA, et al. Mol Cell Biochem. 1999 Sep;199(1-2):63-7. doi: 10.1023/a:1006994931023. Mol Cell Biochem. 1999. PMID: 10544953 Clinical Trial.
The aim of the present work was to analyse the susceptibility to in vitro peroxidation of VLDL and HDL from apparently normolipidemic subjects affected by insulin-dependent diabetes mellitus (IDDM) and non-insulin-dependent diabetes mellitus (NIDDM) in …
The aim of the present work was to analyse the susceptibility to in vitro peroxidation of VLDL and HDL from apparently normolipidemic subjec …
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