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2019 3
2020 3
2021 0
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Environmental microcystin targets the microbiome and increases the risk of intestinal inflammatory pathology via NOX2 in underlying murine model of Nonalcoholic Fatty Liver Disease.
Sarkar S, Kimono D, Albadrani M, Seth RK, Busbee P, Alghetaa H, Porter DE, Scott GI, Brooks B, Nagarkatti M, Nagarkatti P, Chatterjee S. Sarkar S, et al. Sci Rep. 2019 Jun 19;9(1):8742. doi: 10.1038/s41598-019-45009-1. Sci Rep. 2019. PMID: 31217465 Free PMC article.
With increased climate change pressures likely to influence harmful algal blooms, exposure to microcystin, a known hepatotoxin and a byproduct of cyanobacterial blooms can be a risk factor for NAFLD associated comorbidities. Using both in vivo and in vitro experiments we s …
With increased climate change pressures likely to influence harmful algal blooms, exposure to microcystin, a known hepatotoxin and a …
Microcystin exposure worsens nonalcoholic fatty liver disease associated ectopic glomerular toxicity via NOX-2-MIR21 axis.
Sarkar S, Alhasson F, Kimono D, Albadrani M, Seth RK, Xiao S, Porter DE, Scott GI, Brooks B, Nagarkatti M, Nagarkatti P, Chatterjee S. Sarkar S, et al. Environ Toxicol Pharmacol. 2020 Jan;73:103281. doi: 10.1016/j.etap.2019.103281. Epub 2019 Oct 20. Environ Toxicol Pharmacol. 2020. PMID: 31706246 Free PMC article.
Extending our studies, we hypothesized that existing inflammatory conditions in NAFLD could make the kidneys more susceptible to environmental toxicity. Our results showed that exposure of Microcystin-LR (MC) in NAFLD mice caused a marked increase in cellular scarri …
Extending our studies, we hypothesized that existing inflammatory conditions in NAFLD could make the kidneys more susceptible to environm
Exogenous PP2A inhibitor exacerbates the progression of nonalcoholic fatty liver disease via NOX2-dependent activation of miR21.
Albadrani M, Seth RK, Sarkar S, Kimono D, Mondal A, Bose D, Porter DE, Scott GI, Brooks B, Raychoudhury S, Nagarkatti M, Nagarkatti P, Jule Y, Diehl AM, Chatterjee S. Albadrani M, et al. Among authors: sarkar s. Am J Physiol Gastrointest Liver Physiol. 2019 Oct 1;317(4):G408-G428. doi: 10.1152/ajpgi.00061.2019. Epub 2019 Aug 8. Am J Physiol Gastrointest Liver Physiol. 2019. PMID: 31393787 Free PMC article.
Though significant progress has been made in unraveling the pathophysiology of the disease, the role of protein phosphatase 2A (PP2A) and its subsequent inhibition by environmental and genetic factors in NAFLD pathophysiology remains unclear. The present report tests the h …
Though significant progress has been made in unraveling the pathophysiology of the disease, the role of protein phosphatase 2A (PP2A) and it …
Early microcystin-LR exposure-linked inflammasome activation in mice causes development of fatty liver disease and insulin resistance.
Al-Badrani M, Saha P, Mondal A, Seth RK, Sarkar S, Kimono D, Bose D, Porter DE, Scott GI, Brooks B, Raychoudhury S, Nagarkatti M, Nagarkatti P, Chatterjee S. Al-Badrani M, et al. Among authors: sarkar s. Environ Toxicol Pharmacol. 2020 Nov;80:103457. doi: 10.1016/j.etap.2020.103457. Epub 2020 Jul 17. Environ Toxicol Pharmacol. 2020. PMID: 32687983
Evidence from pediatric studies show that infants and children are at risk for early exposure to microcystin. The present report tests the hypothesis that early life exposure to microcystin (MC), a principal component of harmful algal blooms followed by a juvenile e …
Evidence from pediatric studies show that infants and children are at risk for early exposure to microcystin. The present report test …
Higher intestinal and circulatory lactate associated NOX2 activation leads to an ectopic fibrotic pathology following microcystin co-exposure in murine fatty liver disease.
Sarkar S, Saha P, Seth RK, Mondal A, Bose D, Kimono D, Albadrani M, Mukherjee A, Porter DE, Scott GI, Xiao S, Brooks B, Ferry J, Nagarkatti M, Nagarkatti P, Chatterjee S. Sarkar S, et al. Comp Biochem Physiol C Toxicol Pharmacol. 2020 Dec;238:108854. doi: 10.1016/j.cbpc.2020.108854. Epub 2020 Aug 8. Comp Biochem Physiol C Toxicol Pharmacol. 2020. PMID: 32781293
Clinical studies implicated an increased risk of intestinal fibrosis in patients with nonalcoholic fatty liver disease (NAFLD). Our previous studies have shown that microcystin-LR (MC-LR) exposure led to altered gut microbiome and increased abundance of lactate producing b …
Clinical studies implicated an increased risk of intestinal fibrosis in patients with nonalcoholic fatty liver disease (NAFLD). Our previous …
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