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Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1958 1
1963 1
1964 5
1965 5
1966 2
1967 4
1968 4
1969 9
1970 6
1971 10
1972 11
1973 11
1974 13
1975 11
1976 16
1977 7
1978 4
1979 8
1980 9
1981 7
1982 11
1983 14
1984 11
1985 6
1986 14
1987 13
1988 15
1989 7
1990 20
1991 14
1992 18
1993 23
1994 19
1995 18
1996 28
1997 28
1998 23
1999 18
2000 18
2001 20
2002 30
2003 34
2004 34
2005 34
2006 39
2007 35
2008 38
2009 36
2010 41
2011 51
2012 50
2013 60
2014 50
2015 68
2016 74
2017 84
2018 89
2019 87
2020 64
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1,358 results
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Citations

3 articles found by citation matching

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Page 1
Macrophage Immunometabolism: Where Are We (Going)?
Van den Bossche J, O'Neill LA, Menon D. Van den Bossche J, et al. Trends Immunol. 2017 Jun;38(6):395-406. doi: 10.1016/j.it.2017.03.001. Epub 2017 Apr 7. Trends Immunol. 2017. PMID: 28396078 Review.
Metabolic pathways are closely interconnected and recent literature demonstrates the need for glucose metabolism in anti-inflammatory as well as inflammatory macrophages. Moreover, fatty acid oxidation (FAO) not only supports anti-inflammatory response …
Metabolic pathways are closely interconnected and recent literature demonstrates the need for glucose metabolism in anti-inflammatory …
The Warburg Effect and the Hallmarks of Cancer.
Schwartz L, Supuran CT, Alfarouk KO. Schwartz L, et al. Anticancer Agents Med Chem. 2017;17(2):164-170. doi: 10.2174/1871520616666161031143301. Anticancer Agents Med Chem. 2017. PMID: 27804847 Review.
As a result of the metabolic impairment of the oxidative phosphorylation, there is a decrease in ATP concentration. To compensate the reduced energy yield, there is massive glucose uptake, anaerobic glycolysis, with an up-regulation of the Pentose Phosphate P …
As a result of the metabolic impairment of the oxidative phosphorylation, there is a decrease in ATP concentration. To compensate the …
Fatty acid oxidation is required for the respiration and proliferation of malignant glioma cells.
Lin H, Patel S, Affleck VS, Wilson I, Turnbull DM, Joshi AR, Maxwell R, Stoll EA. Lin H, et al. Neuro Oncol. 2017 Jan;19(1):43-54. doi: 10.1093/neuonc/now128. Epub 2016 Jun 29. Neuro Oncol. 2017. PMID: 27365097 Free PMC article.
Gliomas, like many tumors, are thought to primarily metabolize glucose for energy production; however, the reliance upon glycolysis has recently been called into question. ...In addition, the expression of fatty acid oxidation enzymes may provid …
Gliomas, like many tumors, are thought to primarily metabolize glucose for energy production; however, the reliance upon glycolysi
Mitochondrial fatty acid oxidation alterations in heart failure, ischaemic heart disease and diabetic cardiomyopathy.
Fillmore N, Mori J, Lopaschuk GD. Fillmore N, et al. Br J Pharmacol. 2014 Apr;171(8):2080-90. doi: 10.1111/bph.12475. Br J Pharmacol. 2014. PMID: 24147975 Free PMC article. Review.
Specific metabolic changes include a relative increase in cardiac fatty acid oxidation rates and an uncoupling of glycolysis from glucose oxidation. ...In both of these conditions, residual mitochondrial fatty acid oxidat
Specific metabolic changes include a relative increase in cardiac fatty acid oxidation rates and an uncoupling of gl
Mitochondria: The ketogenic diet--A metabolism-based therapy.
Vidali S, Aminzadeh S, Lambert B, Rutherford T, Sperl W, Kofler B, Feichtinger RG. Vidali S, et al. Int J Biochem Cell Biol. 2015 Jun;63:55-9. doi: 10.1016/j.biocel.2015.01.022. Epub 2015 Feb 7. Int J Biochem Cell Biol. 2015. PMID: 25666556 Review.
Mitochondria are the energy-producing organelles of the cell, generating ATP via oxidative phosphorylation mainly by using pyruvate derived from glycolytic processing of glucose. Ketone bodies generated by fatty acid oxidation can serve as alter …
Mitochondria are the energy-producing organelles of the cell, generating ATP via oxidative phosphorylation mainly by using pyruvate d …
Myocardial Energy Substrate Metabolism in Heart Failure : from Pathways to Therapeutic Targets.
Fukushima A, Milner K, Gupta A, Lopaschuk GD. Fukushima A, et al. Curr Pharm Des. 2015;21(25):3654-64. doi: 10.2174/1381612821666150710150445. Curr Pharm Des. 2015. PMID: 26166604 Review.
One approach to stimulating myocardial glucose oxidation is to inhibit fatty acid oxidation. This review will overview the physiological regulation of both myocardial fatty acid and glucose oxidation in the heart, and …
One approach to stimulating myocardial glucose oxidation is to inhibit fatty acid oxidation. This review …
Glycolytic Stimulation Is Not a Requirement for M2 Macrophage Differentiation.
Wang F, Zhang S, Vuckovic I, Jeon R, Lerman A, Folmes CD, Dzeja PP, Herrmann J. Wang F, et al. Cell Metab. 2018 Sep 4;28(3):463-475.e4. doi: 10.1016/j.cmet.2018.08.012. Cell Metab. 2018. PMID: 30184486 Free PMC article.
Enhanced glucose uptake and a switch to glycolysis are key traits of M1 macrophages, whereas enhanced fatty acid oxidation and oxidative phosphorylation are the main metabolic characteristics of M2 macrophages. ...While glucose dep …
Enhanced glucose uptake and a switch to glycolysis are key traits of M1 macrophages, whereas enhanced fatty acid
Astragaloside IV alleviates heart failure via activating PPARα to switch glycolysis to fatty acid β-oxidation.
Dong Z, Zhao P, Xu M, Zhang C, Guo W, Chen H, Tian J, Wei H, Lu R, Cao T. Dong Z, et al. Sci Rep. 2017 Jun 2;7(1):2691. doi: 10.1038/s41598-017-02360-5. Sci Rep. 2017. PMID: 28578382 Free PMC article.
In heart failure (HF), energy metabolism pathway in cardiac muscle changes from fatty acid β-oxidation to glycolysis. However, the exact mechanism is unknown. ...In HF, Astragaloside IV switched glycolysis to fatty acid β-oxidat
In heart failure (HF), energy metabolism pathway in cardiac muscle changes from fatty acid β-oxidation to glycolysis
Elucidating cancer metabolic plasticity by coupling gene regulation with metabolic pathways.
Jia D, Lu M, Jung KH, Park JH, Yu L, Onuchic JN, Kaipparettu BA, Levine H. Jia D, et al. Proc Natl Acad Sci U S A. 2019 Feb 26;116(9):3909-3918. doi: 10.1073/pnas.1816391116. Epub 2019 Feb 7. Proc Natl Acad Sci U S A. 2019. PMID: 30733294 Free PMC article.
Metabolic plasticity enables cancer cells to switch their metabolism phenotypes between glycolysis and oxidative phosphorylation (OXPHOS) during tumorigenesis and metastasis. ...Our modeling results demonstrate a direct association between the activities of AMPK and …
Metabolic plasticity enables cancer cells to switch their metabolism phenotypes between glycolysis and oxidative phosphorylati …
How glucose, glutamine and fatty acid metabolism shape blood and lymph vessel development.
Teuwen LA, Geldhof V, Carmeliet P. Teuwen LA, et al. Dev Biol. 2019 Mar 1;447(1):90-102. doi: 10.1016/j.ydbio.2017.12.001. Epub 2017 Dec 7. Dev Biol. 2019. PMID: 29224892 Free article. Review.
So far, the best-characterized metabolic pathways to have an impact on angiogenesis are glycolysis, fatty acid oxidation and glutamine metabolism. ...During lymphangiogenesis, glycolysis and fatty acid oxidation are key meta …
So far, the best-characterized metabolic pathways to have an impact on angiogenesis are glycolysis, fatty acid oxida
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