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1914 3
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1918 2
1920 2
1921 2
1922 2
1923 3
1924 7
1925 4
1926 4
1927 6
1928 4
1929 5
1930 2
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1932 4
1933 2
1934 5
1935 3
1936 7
1937 3
1938 1
1939 4
1940 3
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1942 5
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1945 24
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1950 124
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1953 163
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1968 671
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1971 711
1972 743
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1974 769
1975 960
1976 1011
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1978 1098
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1980 1196
1981 1218
1982 1292
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1984 1570
1985 1634
1986 1718
1987 1613
1988 1755
1989 1896
1990 1997
1991 2026
1992 2128
1993 2316
1994 2410
1995 2564
1996 2605
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1998 2665
1999 2710
2000 2801
2001 2795
2002 2764
2003 2841
2004 3019
2005 3209
2006 3380
2007 3393
2008 3442
2009 3477
2010 3641
2011 3886
2012 3873
2013 4184
2014 4137
2015 4253
2016 4021
2017 3890
2018 3846
2019 4012
2020 4489
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145,668 results

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Page 1
Did you mean hyperplasia OR hyperplasia (145,625 results)?
[Adrenocortical tumours].
Saeger W. Saeger W. Pathologe. 2003 Jul;24(4):272-9. doi: 10.1007/s00292-003-0621-1. Epub 2003 May 6. Pathologe. 2003. PMID: 14513274 Review. German.
Adrenocortical tumours have to be differentiated from cortical hyperplasias, medullary tumours and extra-adrenal tumours or metastases. These diagnoses are mostly possible using routine paraffin sections. In some cases, immunostaining for differing cortical and medullary t …
Adrenocortical tumours have to be differentiated from cortical hyperplasias, medullary tumours and extra-adrenal tumours or metastase …
Adrenocortical tumorigenesis: Lessons from genetics.
Kamilaris CDC, Hannah-Shmouni F, Stratakis CA. Kamilaris CDC, et al. Best Pract Res Clin Endocrinol Metab. 2020 May;34(3):101428. doi: 10.1016/j.beem.2020.101428. Epub 2020 May 23. Best Pract Res Clin Endocrinol Metab. 2020. PMID: 32507359 Free PMC article. Review.
Genetic alterations in GNAS, PRKAR1A, PRKACA, PRKACB, PDE11A, and PDE8B, that lead to aberrant cyclic adenosine monophosphate-protein (cAMP) kinase A signaling, were found to play a major role in the development of benign cortisol-producing adrenocortical tumors and/or hyperpl …
Genetic alterations in GNAS, PRKAR1A, PRKACA, PRKACB, PDE11A, and PDE8B, that lead to aberrant cyclic adenosine monophosphate-protein (cAMP) …
Adrenal hyperplasias in childhood: An update.
Pitsava G, Stratakis CA. Pitsava G, et al. Front Endocrinol (Lausanne). 2022 Aug 3;13:937793. doi: 10.3389/fendo.2022.937793. eCollection 2022. Front Endocrinol (Lausanne). 2022. PMID: 35992119 Free PMC article. Review.
Most cases are due to defects in the cyclic AMP/protein kinase A (cAMP/PKA) pathway, although a few cases remain without an identified genetic defect. Another cause of adrenal hyperplasia in childhood is congenital adrenal hyperplasia, a group of autosomal recessive …
Most cases are due to defects in the cyclic AMP/protein kinase A (cAMP/PKA) pathway, although a few cases remain without an identified genet …
Disordered CYP11B2 Expression in Primary Aldosteronism.
Gomez-Sanchez CE, Kuppusamy M, Reincke M, Williams TA. Gomez-Sanchez CE, et al. Horm Metab Res. 2017 Dec;49(12):957-962. doi: 10.1055/s-0043-122238. Epub 2017 Dec 4. Horm Metab Res. 2017. PMID: 29202495 Free article. Review.
PA is mainly caused by unilateral hyperaldosteronism due to an aldosterone-producing adenoma, unilateral hyperplasia with or without micronodules or bilateral zona glomerulosa hyperplasias with or without macro or micronodules. ...Approximately 30% of patients with …
PA is mainly caused by unilateral hyperaldosteronism due to an aldosterone-producing adenoma, unilateral hyperplasia with or without …
Adrenocorticotropic hormone-independent Cushing's syndrome.
Bourdeau I, Lampron A, Costa MH, Tadjine M, Lacroix A. Bourdeau I, et al. Curr Opin Endocrinol Diabetes Obes. 2007 Jun;14(3):219-25. doi: 10.1097/MED.0b013e32814db842. Curr Opin Endocrinol Diabetes Obes. 2007. PMID: 17940443 Review.
Primary Cushing's syndrome is most often secondary to adrenocortical adenomas or carcinomas, and more rarely to bilateral adrenal hyperplasias. Corticotropin-independent cortisol-producing hyperplasia is caused by micronodular diseases, including primary pigmented n …
Primary Cushing's syndrome is most often secondary to adrenocortical adenomas or carcinomas, and more rarely to bilateral adrenal hyperpl …
Hyperplasia in glands with hormone excess.
Marx SJ. Marx SJ. Endocr Relat Cancer. 2016 Jan;23(1):R1-14. doi: 10.1530/ERC-15-0171. Epub 2015 Sep 25. Endocr Relat Cancer. 2016. PMID: 26407873 Free PMC article. Review.
Comparisons were made among this and two other groups of syndromes, forming a continuum of gland staging: predominant oversecretions express little or no hyperplasia; predominant hyperplasias express little or no neoplasia; and predominant neoplasias express nodules …
Comparisons were made among this and two other groups of syndromes, forming a continuum of gland staging: predominant oversecretions express …
Gastrointestinal neuroendocrine cell proliferations.
Lechago J. Lechago J. Hum Pathol. 1994 Nov;25(11):1114-22. doi: 10.1016/0046-8177(94)90028-0. Hum Pathol. 1994. PMID: 7959656 Review.
The gastrointestinal neuroendocrine cell proliferations are comprised of a few hyperplasias and various neoplasias. The better characterized hyperplasias include G-cell hyperplasia, either primary or secondary, enterochromaffin-like (ECL)-cell hyperplasias …
The gastrointestinal neuroendocrine cell proliferations are comprised of a few hyperplasias and various neoplasias. The better charac …
A Gene-Based Classification of Primary Adrenocortical Hyperplasias.
Hannah-Shmouni F, Stratakis CA. Hannah-Shmouni F, et al. Horm Metab Res. 2020 Mar;52(3):133-141. doi: 10.1055/a-1107-2972. Epub 2020 Mar 25. Horm Metab Res. 2020. PMID: 32215884 Review.
Primary or adrenocorticotropin-independent adrenocortical tumors and hyperplasias represent a heterogeneous group of adrenocortical neoplasms that arise from various genetic defects, either in isolation or familial. The traditional classification as adenomas, hyperplasi …
Primary or adrenocorticotropin-independent adrenocortical tumors and hyperplasias represent a heterogeneous group of adrenocortical n …
Role of unilateral adrenalectomy in bilateral adrenal hyperplasias with Cushing's syndrome.
Meloche-Dumas L, Mercier F, Lacroix A. Meloche-Dumas L, et al. Best Pract Res Clin Endocrinol Metab. 2021 Mar;35(2):101486. doi: 10.1016/j.beem.2021.101486. Epub 2021 Jan 13. Best Pract Res Clin Endocrinol Metab. 2021. PMID: 33637447 Review.
Primary bilateral adrenocortical hyperplasias are rare forms of pituitary ACTH-independent Cushing's syndrome (CS). They are divided between primary bilateral macronodular adrenal hyperplasia (PBMAH) and micronodular adrenal hyperplasia (MiBAH), which is subd …
Primary bilateral adrenocortical hyperplasias are rare forms of pituitary ACTH-independent Cushing's syndrome (CS). They are divided …
Epulides in dogs.
Reichart PA, Philipsen HP, Dürr UM. Reichart PA, et al. J Oral Pathol Med. 1989 Feb;18(2):92-6. doi: 10.1111/j.1600-0714.1989.tb00743.x. J Oral Pathol Med. 1989. PMID: 2746522
Eighty-one cases of canine epulides are described clinically and histologically; 73 cases were diagnosed as inflammatory, fibrous hyperplasias of which 37% showed metaplastic bone formation and 22% demonstrated so-called plexiform epithelial hyperplasia which seems …
Eighty-one cases of canine epulides are described clinically and histologically; 73 cases were diagnosed as inflammatory, fibrous hyperpl …
145,668 results
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