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Table representation of search results timeline featuring number of search results per year.

Year Number of Results
2000 2
2001 1
2003 1
2004 3
2005 1
2006 3
2007 2
2008 8
2009 3
2010 4
2011 8
2012 10
2013 12
2014 18
2015 19
2016 16
2017 20
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2019 29
2020 35
2021 43
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2023 35
2024 42
2025 28
2026 15

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382 results

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Page 1
Neuropathological hallmarks during the chronic phase of ischemic stroke in mice and humans.
Khan R, Guzman G, Do T, Devlin P, Ahn J, Tan C, Venna VR, Marrelli SP, Koochak H, Di Gesù CM, Flores A, McCullough LD, Ritzel RM. Khan R, et al. Exp Neurol. 2026 Apr 16;402:115766. doi: 10.1016/j.expneurol.2026.115766. Online ahead of print. Exp Neurol. 2026. PMID: 41999827 Free article.
These findings support the concept that stroke accelerates brain aging ("inflamm-aging"), characterized by premature cellular senescence, altered microglial states, and ongoing neurodegeneration. ...
These findings support the concept that stroke accelerates brain aging ("inflamm-aging"), characterized by premature ce …
Oral wound healing in the elderly: Mechanisms, challenges, and innovations.
Decker AM, Stuhr SH, Avila-Ortiz G, Pilloni A, Marini L, Castilho R, Wallace BI, Hernandez-Kapila YL. Decker AM, et al. Periodontol 2000. 2026 Mar 25. doi: 10.1111/prd.70027. Online ahead of print. Periodontol 2000. 2026. PMID: 41881503 Review.
Tissue-specific cues in oral mucosa, periodontal ligament, cementum, and bone are also altered with age. Compounding factors-including "inflamm-aging," malnutrition, polypharmacy-induced xerostomia, cognitive decline, and frailty-further impede healing. ...
Tissue-specific cues in oral mucosa, periodontal ligament, cementum, and bone are also altered with age. Compounding factors-including "i
Telomere damage-mediated senescence in alveolar epithelial type II cells but not in macrophages aggravates inflammation in acute lung injury.
Hildebrand CB, Öztürk S, Ye JL, Wedekind D, Maus UA, Bär C, Mühlfeld C, Brandenberger C. Hildebrand CB, et al. Respir Res. 2026 Mar 14;27(1):161. doi: 10.1186/s12931-026-03627-0. Respir Res. 2026. PMID: 41826998 Free PMC article.
Control mice with Trf1 deletion of both Cre-loxP mouse models did not show signs of inflamm-aging. Additional comparison with old mice revealed that global aging causes a much greater severity in ALI than Trf1 deletion in lung cells. ...The results also suggest that …
Control mice with Trf1 deletion of both Cre-loxP mouse models did not show signs of inflamm-aging. Additional comparison with …
Preventing ovarian aging: from redox-targeted strategies to extracellular vesicle-based therapies.
Camerano Spelta Rapini C, Rojo-Fleming CC, Di Berardino C, Peserico A, Capacchietti G, Tosi U, Bernabò N, Mattioli M, Barboni B. Camerano Spelta Rapini C, et al. Front Aging. 2026 Feb 24;7:1707614. doi: 10.3389/fragi.2026.1707614. eCollection 2026. Front Aging. 2026. PMID: 41816543 Free PMC article. Review.
The distinction between physiological aging and pathological processes such as "inflamm-aging" is discussed, with particular attention to redox imbalance and mitochondrial impairment as key drivers of follicular depletion and endocrine dysfunction. ...
The distinction between physiological aging and pathological processes such as "inflamm-aging" is discussed, with particular a …
Lymphocyte-To-Monocyte Ratio is Partially Mediated in Age-Related Cardiovascular Mortality in HFpEF: Immunosenescence, Inflamm-Aging, and Longevity.
Cai X, Liu M, Feng C, Tang S, Qin P, Li Y, Wang T, He L, Lei J, Zhou Y, Guo Y, Zhuang X, Liao X. Cai X, et al. Rev Cardiovasc Med. 2026 Feb 11;27(2):45403. doi: 10.31083/RCM45403. eCollection 2026 Feb. Rev Cardiovasc Med. 2026. PMID: 41789332 Free PMC article.
This study offers a cost-effective predictor for HFpEF and suggests potential mechanisms related to immunosenescence and inflammation-related aging (inflamm-aging)....
This study offers a cost-effective predictor for HFpEF and suggests potential mechanisms related to immunosenescence and inflammation-relate …
Senescent endothelial cells: key commanders of the cellular communication network within atherosclerotic plaques.
Xie F, Xi C, Bao G, Yang B, Zheng X, Fu B, Zheng Z. Xie F, et al. Front Immunol. 2026 Feb 4;17:1712974. doi: 10.3389/fimmu.2026.1712974. eCollection 2026. Front Immunol. 2026. PMID: 41716416 Free PMC article. Review.
Crucially, our analysis emphasizes the necessity of sex-specific therapeutic approaches, distinguishing between inflamm-aging driven pathologies in men and mechanisms centered on metabolic resilience in women....
Crucially, our analysis emphasizes the necessity of sex-specific therapeutic approaches, distinguishing between inflamm-aging
SLIT2 as a key regulator and therapeutic target in liver injury.
Choi YW, Choi JH, Lee YS, Jeong J, Kang E, Park SH, Lee YK, Park SS, Kang HY, Kim YH, Park TJ. Choi YW, et al. Mol Ther. 2026 Apr 1;34(4):2446-2467. doi: 10.1016/j.ymthe.2026.01.010. Epub 2026 Jan 10. Mol Ther. 2026. PMID: 41521561 Free article.
IL-17a induces age-related olfactory dysfunction by impairing regeneration and promoting respiratory metaplasia in mice.
Wang S, Cha X, Xie Y, Xu Z, Cai B, Li F, Li T, Wang Z, Wang T, Yu Y, Liu H, Ren W. Wang S, et al. Nat Commun. 2025 Dec 29;17(1):1036. doi: 10.1038/s41467-025-67786-2. Nat Commun. 2025. PMID: 41461651 Free PMC article.
This process is supported by the globose basal cells (GBC) during the homeostatic state, as well as horizontal basal cells (HBC) during severe damage. Inflamm-aging refers to the low-grade, chronic and progressive state of heightened pro-inflammation associated with …
This process is supported by the globose basal cells (GBC) during the homeostatic state, as well as horizontal basal cells (HBC) during seve …
382 results