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2006 2
2016 1
2020 0
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Page 1
Borrelia burgdorferi Induces TLR1 and TLR2 in human microglia and peripheral blood monocytes but differentially regulates HLA-class II expression.
Cassiani-Ingoni R, Cabral ES, Lünemann JD, Garza Z, Magnus T, Gelderblom H, Munson PJ, Marques A, Martin R. Cassiani-Ingoni R, et al. J Neuropathol Exp Neurol. 2006 Jun;65(6):540-8. doi: 10.1097/00005072-200606000-00002. J Neuropathol Exp Neurol. 2006. PMID: 16783164
Ureaplasma diversum Genome Provides New Insights about the Interaction of the Surface Molecules of This Bacterium with the Host.
Marques LM, Rezende IS, Barbosa MS, Guimarães AM, Martins HB, Campos GB, do Nascimento NC, Dos Santos AP, Amorim AT, Santos VM, Farias ST, Barrence FÂ, de Souza LM, Buzinhani M, Arana-Chavez VE, Zenteno ME, Amarante-Mendes GP, Messick JB, Timenetsky J. Marques LM, et al. PLoS One. 2016 Sep 7;11(9):e0161926. doi: 10.1371/journal.pone.0161926. eCollection 2016. PLoS One. 2016. PMID: 27603136 Free PMC article.
As with viable bacteria, non-viable bacteria were capable of promoting a significant inflammatory response by activation of Toll like receptor 2 (TLR2), indicating that surface molecules are important for the activation of inflammatory response. ...
As with viable bacteria, non-viable bacteria were capable of promoting a significant inflammatory response by activation of Toll like recept …
Borrelia burgdorferi lipoprotein-mediated TLR2 stimulation causes the down-regulation of TLR5 in human monocytes.
Cabral ES, Gelderblom H, Hornung RL, Munson PJ, Martin R, Marques AR. Cabral ES, et al. J Infect Dis. 2006 Mar 15;193(6):849-59. doi: 10.1086/500467. Epub 2006 Feb 8. J Infect Dis. 2006. PMID: 16479520
Lipoproteins from Borrelia burgdorferi, the agent of Lyme disease, activate inflammatory cells through TLR2 and TLR1. We show that stimulation of human monocytes with B. burgdorferi lysate, lipidated outer surface protein A, and triacylated lipopeptide Pam3CysSerLys4 resul …
Lipoproteins from Borrelia burgdorferi, the agent of Lyme disease, activate inflammatory cells through TLR2 and TLR1. We show that st …
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