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Splicing regulatory factors in breast cancer hallmarks and disease progression.
Koedoot E, Wolters L, van de Water B, Dévédec SEL. Koedoot E, et al. Among authors: van de Water B. Oncotarget. 2019 Oct 15;10(57):6021-6037. doi: 10.18632/oncotarget.27215. eCollection 2019 Oct 15. Oncotarget. 2019. PMID: 31666932 Free PMC article. Review.
Phospho-proteomic analysis of cellular signaling.
de Graauw M, Hensbergen P, van de Water B. de Graauw M, et al. Among authors: van de Water B. Electrophoresis. 2006 Jul;27(13):2676-86. doi: 10.1002/elps.200600018. Electrophoresis. 2006. PMID: 16739229 Review.
Focal adhesion kinase: a potential target in cancer therapy.
van Nimwegen MJ, van de Water B. van Nimwegen MJ, et al. Among authors: van de Water B. Biochem Pharmacol. 2007 Mar 1;73(5):597-609. doi: 10.1016/j.bcp.2006.08.011. Epub 2006 Sep 25. Biochem Pharmacol. 2007. PMID: 16997283 Review.
An improved method to study NK-independent mechanisms of MTLn3 breast cancer lung metastasis.
van Nimwegen MJ, Verkoeijen S, Kuppen PJ, Velthuis JH, van de Water B. van Nimwegen MJ, et al. Among authors: van de Water B. Clin Exp Metastasis. 2007;24(5):379-87. doi: 10.1007/s10585-007-9075-9. Epub 2007 May 31. Clin Exp Metastasis. 2007. PMID: 17541710
This is largely mediated by the perforin/granzyme B pathway in MTLn3 cells in a caspase-dependent manner. Temporal in vivo depletion of NK cells by an antibody-based method, dramatically improved colonization of the lungs by MTLn3 cells, from 5 metastases in the untreated …
This is largely mediated by the perforin/granzyme B pathway in MTLn3 cells in a caspase-dependent manner. Temporal in vivo depletion …
An improved model to study tumor cell autonomous metastasis programs using MTLn3 cells and the Rag2(-/-) gammac (-/-) mouse.
Le Dévédec SE, van Roosmalen W, Maria N, Grimbergen M, Pont C, Lalai R, van de Water B. Le Dévédec SE, et al. Among authors: van Roosmalen W, van de Water B. Clin Exp Metastasis. 2009;26(7):673-84. doi: 10.1007/s10585-009-9267-6. Epub 2009 May 24. Clin Exp Metastasis. 2009. PMID: 19466569 Free PMC article.
Annexin A1 regulates TGF-beta signaling and promotes metastasis formation of basal-like breast cancer cells.
de Graauw M, van Miltenburg MH, Schmidt MK, Pont C, Lalai R, Kartopawiro J, Pardali E, Le Dévédec SE, Smit VT, van der Wal A, Van't Veer LJ, Cleton-Jansen AM, ten Dijke P, van de Water B. de Graauw M, et al. Among authors: van Miltenburg MH, van der Wal A, van de Water B. Proc Natl Acad Sci U S A. 2010 Apr 6;107(14):6340-5. doi: 10.1073/pnas.0913360107. Epub 2010 Mar 22. Proc Natl Acad Sci U S A. 2010. PMID: 20308542 Free PMC article.
Two-photon intravital multicolor imaging combined with inducible gene expression to distinguish metastatic behavior of breast cancer cells in vivo.
Le Dévédec SE, Lalai R, Pont C, de Bont H, van de Water B. Le Dévédec SE, et al. Among authors: de Bont H, van de Water B. Mol Imaging Biol. 2011 Feb;13(1):67-77. doi: 10.1007/s11307-010-0307-z. Mol Imaging Biol. 2011. PMID: 20396956 Free PMC article.
Systems microscopy approaches to understand cancer cell migration and metastasis.
Le Dévédec SE, Yan K, de Bont H, Ghotra V, Truong H, Danen EH, Verbeek F, van de Water B. Le Dévédec SE, et al. Among authors: de Bont H, van de Water B. Cell Mol Life Sci. 2010 Oct;67(19):3219-40. doi: 10.1007/s00018-010-0419-2. Epub 2010 Jun 18. Cell Mol Life Sci. 2010. PMID: 20556632 Free PMC article. Review.
Elevated insulin-like growth factor 1 receptor signaling induces antiestrogen resistance through the MAPK/ERK and PI3K/Akt signaling routes.
Zhang Y, Moerkens M, Ramaiahgari S, de Bont H, Price L, Meerman J, van de Water B. Zhang Y, et al. Among authors: de Bont H, van de Water B. Breast Cancer Res. 2011 May 19;13(3):R52. doi: 10.1186/bcr2883. Breast Cancer Res. 2011. PMID: 21595894 Free PMC article.
This antiestrogen-resistant phenotype involved mitogen-activated protein kinase/extracellular signal-regulated kinase (MAPK/ERK) and phosphatidylinositol 3-kinase/protein kinase B pathways downstream of the IGF-1R signaling hub and was independent of ER signaling. ...
This antiestrogen-resistant phenotype involved mitogen-activated protein kinase/extracellular signal-regulated kinase (MAPK/ERK) and phospha …
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