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Biomimetic Nanoparticles as a Theranostic Tool for Traumatic Brain Injury.
Zinger A, Soriano S, Baudo G, De Rosa E, Taraballi F, Villapol S. Zinger A, et al. Among authors: villapol s. Adv Funct Mater. 2021 Jul 23;31(30):2100722. doi: 10.1002/adfm.202100722. Epub 2021 Mar 26. Adv Funct Mater. 2021. PMID: 34413716 Free PMC article.
Traumatic brain injury (TBI) triggers both central and peripheral inflammatory responses. Existing pharmacological drugs are unable to effectively and quickly target the brain inflamed regions, setting up a major roadblock towards effective brain
Traumatic brain injury (TBI) triggers both central and peripheral inflammatory responses. Existing pharmacological drug
Traumatic Brain Injury in Mice Induces Acute Bacterial Dysbiosis Within the Fecal Microbiome.
Treangen TJ, Wagner J, Burns MP, Villapol S. Treangen TJ, et al. Among authors: villapol s. Front Immunol. 2018 Nov 27;9:2757. doi: 10.3389/fimmu.2018.02757. eCollection 2018. Front Immunol. 2018. PMID: 30546361 Free PMC article.
The secondary injury cascade that is activated following traumatic brain injury (TBI) induces responses from multiple physiological systems, including the immune system. These responses are not limited to the area of brain injury; they ca …
The secondary injury cascade that is activated following traumatic brain injury (TBI) induces responses from mul …
Polypathology and dementia after brain trauma: Does brain injury trigger distinct neurodegenerative diseases, or should they be classified together as traumatic encephalopathy?
Washington PM, Villapol S, Burns MP. Washington PM, et al. Among authors: villapol s. Exp Neurol. 2016 Jan;275 Pt 3(0 3):381-388. doi: 10.1016/j.expneurol.2015.06.015. Epub 2015 Jun 16. Exp Neurol. 2016. PMID: 26091850 Free PMC article. Review.
Neuropathological studies of human traumatic brain injury (TBI) cases have described amyloid plaques acutely after a single severe TBI, and tau pathology after repeat mild TBI (mTBI). ...These data support the suggestion that the multiple manifestations of TB …
Neuropathological studies of human traumatic brain injury (TBI) cases have described amyloid plaques acutely after a si …
Sexual dimorphism in the inflammatory response to traumatic brain injury.
Villapol S, Loane DJ, Burns MP. Villapol S, et al. Glia. 2017 Sep;65(9):1423-1438. doi: 10.1002/glia.23171. Epub 2017 Jun 13. Glia. 2017. PMID: 28608978 Free PMC article.
The activation of resident microglial cells, alongside the infiltration of peripheral macrophages, are key neuroinflammatory responses to traumatic brain injury (TBI) that are directly associated with neuronal death. ...Understanding how sex affects the cours …
The activation of resident microglial cells, alongside the infiltration of peripheral macrophages, are key neuroinflammatory responses to …
Serum Amyloid A is Expressed in the Brain After Traumatic Brain Injury in a Sex-Dependent Manner.
Soriano S, Moffet B, Wicker E, Villapol S. Soriano S, et al. Among authors: villapol s. Cell Mol Neurobiol. 2020 Oct;40(7):1199-1211. doi: 10.1007/s10571-020-00808-3. Epub 2020 Feb 14. Cell Mol Neurobiol. 2020. PMID: 32060858 Free PMC article.
Serum amyloid A (SAA) is an acute phase protein upregulated in the liver after traumatic brain injury (TBI). So far, it has not been investigated whether SAA expression also occurs in the brain in response to TBI. For this, we performed a moderate cont …
Serum amyloid A (SAA) is an acute phase protein upregulated in the liver after traumatic brain injury (TBI). So far, it …
Neurorestoration after traumatic brain injury through angiotensin II receptor blockage.
Villapol S, Balarezo MG, Affram K, Saavedra JM, Symes AJ. Villapol S, et al. Brain. 2015 Nov;138(Pt 11):3299-315. doi: 10.1093/brain/awv172. Epub 2015 Jun 26. Brain. 2015. PMID: 26115674 Free PMC article.
Traumatic brain injury results in neuronal injury and death, acute and prolonged inflammation and decreased blood flow. ...This study strongly suggests that sartans with dual AT1R blocking and PPARgamma activating properties have therapeutic potential
Traumatic brain injury results in neuronal injury and death, acute and prolonged inflammation and decreased bloo
Apolipoprotein E4 impairs spontaneous blood brain barrier repair following traumatic brain injury.
Main BS, Villapol S, Sloley SS, Barton DJ, Parsadanian M, Agbaegbu C, Stefos K, McCann MS, Washington PM, Rodriguez OC, Burns MP. Main BS, et al. Among authors: villapol s. Mol Neurodegener. 2018 Apr 4;13(1):17. doi: 10.1186/s13024-018-0249-5. Mol Neurodegener. 2018. PMID: 29618365 Free PMC article.
BACKGROUND: Traumatic Brain Injury (TBI) is a major cause of disability and mortality, to which there is currently no comprehensive treatment. Blood Brain Barrier (BBB) dysfunction is well documented in human TBI patients, yet the molecular mechanisms …
BACKGROUND: Traumatic Brain Injury (TBI) is a major cause of disability and mortality, to which there is currently no c …
A Mouse Model of Single and Repetitive Mild Traumatic Brain Injury.
Main BS, Sloley SS, Villapol S, Zapple DN, Burns MP. Main BS, et al. Among authors: villapol s. J Vis Exp. 2017 Jun 20;(124):55713. doi: 10.3791/55713. J Vis Exp. 2017. PMID: 28654066 Free PMC article.
Mild Traumatic Brain Injury (mTBI) can result in the acute loss of brain function, including a period of confusion, a loss of consciousness (LOC), focal neurological deficits and even amnesia. Athletes participating in contact sports are at high risk o …
Mild Traumatic Brain Injury (mTBI) can result in the acute loss of brain function, including a period of confusi …
Serum Amyloid A Protein as a Potential Biomarker for Severity and Acute Outcome in Traumatic Brain Injury.
Wicker E, Benton L, George K, Furlow W, Villapol S. Wicker E, et al. Among authors: villapol s. Biomed Res Int. 2019 Apr 16;2019:5967816. doi: 10.1155/2019/5967816. eCollection 2019. Biomed Res Int. 2019. PMID: 31119176 Free PMC article.
Traumatic brain injury (TBI) causes a wide variety of neuroinflammatory events. ...Our previous studies have shown that the liver produces serum amyloid A (SAA) at high levels in the initial hours after controlled cortical impact (CCI) injury in mice.
Traumatic brain injury (TBI) causes a wide variety of neuroinflammatory events. ...Our previous studies have shown that
Smad3 deficiency increases cortical and hippocampal neuronal loss following traumatic brain injury.
Villapol S, Wang Y, Adams M, Symes AJ. Villapol S, et al. Exp Neurol. 2013 Dec;250:353-65. doi: 10.1016/j.expneurol.2013.10.008. Epub 2013 Oct 9. Exp Neurol. 2013. PMID: 24120438
Transforming growth factor-beta (TGF-beta) signaling is involved in pathological processes following brain injury. TGF-beta signaling through Smad3 contributes significantly to the immune response and glial scar formation after brain injury. However, T …
Transforming growth factor-beta (TGF-beta) signaling is involved in pathological processes following brain injury. TGF-beta si …
24 results