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Traumatic Brain Injury in Mice Induces Acute Bacterial Dysbiosis Within the Fecal Microbiome.
Treangen TJ, Wagner J, Burns MP, Villapol S. Treangen TJ, et al. Among authors: villapol s. Front Immunol. 2018 Nov 27;9:2757. doi: 10.3389/fimmu.2018.02757. eCollection 2018. Front Immunol. 2018. PMID: 30546361 Free PMC article.
The secondary injury cascade that is activated following traumatic brain injury (TBI) induces responses from multiple physiological systems, including the immune system. These responses are not limited to the area of brain injury; they ca …
The secondary injury cascade that is activated following traumatic brain injury (TBI) induces responses from mul …
Apolipoprotein E4 impairs spontaneous blood brain barrier repair following traumatic brain injury.
Main BS, Villapol S, Sloley SS, Barton DJ, Parsadanian M, Agbaegbu C, Stefos K, McCann MS, Washington PM, Rodriguez OC, Burns MP. Main BS, et al. Among authors: villapol s. Mol Neurodegener. 2018 Apr 4;13(1):17. doi: 10.1186/s13024-018-0249-5. Mol Neurodegener. 2018. PMID: 29618365 Free PMC article.
BACKGROUND: Traumatic Brain Injury (TBI) is a major cause of disability and mortality, to which there is currently no comprehensive treatment. Blood Brain Barrier (BBB) dysfunction is well documented in human TBI patients, yet the molecular mechanisms …
BACKGROUND: Traumatic Brain Injury (TBI) is a major cause of disability and mortality, to which there is currently no c …
Polypathology and dementia after brain trauma: Does brain injury trigger distinct neurodegenerative diseases, or should they be classified together as traumatic encephalopathy?
Washington PM, Villapol S, Burns MP. Washington PM, et al. Among authors: villapol s. Exp Neurol. 2016 Jan;275 Pt 3(0 3):381-388. doi: 10.1016/j.expneurol.2015.06.015. Epub 2015 Jun 16. Exp Neurol. 2016. PMID: 26091850 Free PMC article. Review.
Neuropathological studies of human traumatic brain injury (TBI) cases have described amyloid plaques acutely after a single severe TBI, and tau pathology after repeat mild TBI (mTBI). ...Preclinical studies confirm that multiple proteins associated with the d …
Neuropathological studies of human traumatic brain injury (TBI) cases have described amyloid plaques acutely after a si …
Roles of Peroxisome Proliferator-Activated Receptor Gamma on Brain and Peripheral Inflammation.
Villapol S. Villapol S. Cell Mol Neurobiol. 2018 Jan;38(1):121-132. doi: 10.1007/s10571-017-0554-5. Epub 2017 Oct 3. Cell Mol Neurobiol. 2018. PMID: 28975471 Free PMC article. Review.
This review will highlight key advances in the understanding of the PPARγ-related mechanisms responsible for neuroprotection after brain injuries, both ischemia and traumatic brain injury, and it will also cover the natural and synthetic agonist for PP …
This review will highlight key advances in the understanding of the PPARγ-related mechanisms responsible for neuroprotection after brain
Sexual dimorphism in the inflammatory response to traumatic brain injury.
Villapol S, Loane DJ, Burns MP. Villapol S, et al. Glia. 2017 Sep;65(9):1423-1438. doi: 10.1002/glia.23171. Epub 2017 Jun 13. Glia. 2017. PMID: 28608978 Free PMC article.
The activation of resident microglial cells, alongside the infiltration of peripheral macrophages, are key neuroinflammatory responses to traumatic brain injury (TBI) that are directly associated with neuronal death. ...Understanding how sex affects the cours …
The activation of resident microglial cells, alongside the infiltration of peripheral macrophages, are key neuroinflammatory responses to …
Serum Amyloid A Protein as a Potential Biomarker for Severity and Acute Outcome in Traumatic Brain Injury.
Wicker E, Benton L, George K, Furlow W, Villapol S. Wicker E, et al. Among authors: villapol s. Biomed Res Int. 2019 Apr 16;2019:5967816. doi: 10.1155/2019/5967816. eCollection 2019. Biomed Res Int. 2019. PMID: 31119176 Free PMC article.
Traumatic brain injury (TBI) causes a wide variety of neuroinflammatory events. These neuroinflammatory events depend, to a greater extent, on the severity of the damage. ...Clinical studies have reported detectable SAA in the plasma of brain injury
Traumatic brain injury (TBI) causes a wide variety of neuroinflammatory events. These neuroinflammatory events depend,
NOX2 deficiency alters macrophage phenotype through an IL-10/STAT3 dependent mechanism: implications for traumatic brain injury.
Barrett JP, Henry RJ, Villapol S, Stoica BA, Kumar A, Burns MP, Faden AI, Loane DJ. Barrett JP, et al. Among authors: villapol s. J Neuroinflammation. 2017 Mar 24;14(1):65. doi: 10.1186/s12974-017-0843-4. J Neuroinflammation. 2017. PMID: 28340575 Free PMC article.
Excessive ROS production is linked with neuroinflammation and chronic neurodegeneration following traumatic brain injury (TBI). ...Here, we report the signaling pathways that regulate NOX2-dependent macrophage/microglial phenotype switching in the TBI brai
Excessive ROS production is linked with neuroinflammation and chronic neurodegeneration following traumatic brain injury
Neurorestoration after traumatic brain injury through angiotensin II receptor blockage.
Villapol S, Balarezo MG, Affram K, Saavedra JM, Symes AJ. Villapol S, et al. Brain. 2015 Nov;138(Pt 11):3299-315. doi: 10.1093/brain/awv172. Epub 2015 Jun 26. Brain. 2015. PMID: 26115674 Free PMC article.
To test whether these drugs may be effective in treating traumatic brain injury, we selected two sartans, candesartan and telmisartan, of proven therapeutic efficacy in animal models of brain inflammation, neurodegenerative disorders and stroke. ...Thi …
To test whether these drugs may be effective in treating traumatic brain injury, we selected two sartans, candesartan a …
A Mouse Model of Single and Repetitive Mild Traumatic Brain Injury.
Main BS, Sloley SS, Villapol S, Zapple DN, Burns MP. Main BS, et al. Among authors: villapol s. J Vis Exp. 2017 Jun 20;(124):55713. doi: 10.3791/55713. J Vis Exp. 2017. PMID: 28654066 Free PMC article.
Mild Traumatic Brain Injury (mTBI) can result in the acute loss of brain function, including a period of confusion, a loss of consciousness (LOC), focal neurological deficits and even amnesia. ...In terms of the level of injury in a sporting ath …
Mild Traumatic Brain Injury (mTBI) can result in the acute loss of brain function, including a period of confusi …
Serum Amyloid A is Expressed in the Brain After Traumatic Brain Injury in a Sex-Dependent Manner.
Soriano S, Moffet B, Wicker E, Villapol S. Soriano S, et al. Among authors: villapol s. Cell Mol Neurobiol. 2020 Feb 14. doi: 10.1007/s10571-020-00808-3. Online ahead of print. Cell Mol Neurobiol. 2020. PMID: 32060858
Serum amyloid A (SAA) is an acute phase protein upregulated in the liver after traumatic brain injury (TBI). So far, it has not been investigated whether SAA expression also occurs in the brain in response to TBI. For this, we performed a moderate cont …
Serum amyloid A (SAA) is an acute phase protein upregulated in the liver after traumatic brain injury (TBI). So far, it …
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