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Page 1
Coxsackievirus B3 replication and pathogenesis.
Garmaroudi FS, Marchant D, Hendry R, Luo H, Yang D, Ye X, Shi J, McManus BM. Garmaroudi FS, et al. Among authors: yang d. Future Microbiol. 2015;10(4):629-53. doi: 10.2217/fmb.15.5. Future Microbiol. 2015. PMID: 25865198 Free article. Review.
Evolution has equipped this pico-rna-virus, CVB3, to use different strategies, including CVB3-induced direct damage to host cells followed by a host inflammatory response to CVB3 infection, and cell death to super-additively promote target organ tissue injury …
Evolution has equipped this pico-rna-virus, CVB3, to use different strategies, including CVB3-induced direct damage to host ce …
Heart-specific NFAT5 knockout suppresses type I interferon signaling and aggravates coxsackievirus-induced myocarditis.
Zhao G, Zhang HM, Nasseri AR, Yip F, Telkar N, Chen YT, Aghakeshmiri S, Küper C, Lam W, Yang W, Zhao J, Luo H, McManus BM, Yang D. Zhao G, et al. Among authors: yang d. Basic Res Cardiol. 2024 Dec;119(6):1075-1092. doi: 10.1007/s00395-024-01058-w. Epub 2024 Jun 5. Basic Res Cardiol. 2024. PMID: 38834767
Although the association of Coxsackievirus B3 (CVB3) with viral myocarditis is well-established, the role of NFAT5 in this disease remains largely unexplored. Previous research has demonstrated that NFAT5 restricts CVB3 replication yet is susceptible to cleavage by …
Although the association of Coxsackievirus B3 (CVB3) with viral myocarditis is well-established, the role of NFAT5 in this disease re …
Poly(rC) binding protein 1 benefits coxsackievirus B3 infection via suppressing the translation of p62/SQSTM1.
He HY, You Z, Ouyang T, Zhao G, Chen LJ, Wang Q, Li JY, Ye X, Zhang MH, Yang D, Ge XY, Qiu Y. He HY, et al. Among authors: yang d. Virus Res. 2022 Sep;318:198851. doi: 10.1016/j.virusres.2022.198851. Epub 2022 Jun 25. Virus Res. 2022. PMID: 35764193
Coxsackievirus B3 (CVB3) is a positive single-strand RNA virus causing myocarditis, pancreatitis and meningitis. ...In summary, our studies have identified PCBP1 as a beneficial factor for CVB3 infection. These findings may deepen the understanding of host-virus int …
Coxsackievirus B3 (CVB3) is a positive single-strand RNA virus causing myocarditis, pancreatitis and meningitis. ...In summary, our s …
The impact of CVB3 infection on host cell biology.
Marchant D, Si X, Luo H, McManus B, Yang D. Marchant D, et al. Among authors: yang d. Curr Top Microbiol Immunol. 2008;323:177-98. doi: 10.1007/978-3-540-75546-3_8. Curr Top Microbiol Immunol. 2008. PMID: 18357770 Review.
CVB3 myocarditis can lead to dilated cardiomyopath (DCM). DCM is one of the leading causes of the need for heart transplantation, so it is important to understand the life cycle of CVB3 and its interactions with the host cell. ...Interactions of host cell proteins w …
CVB3 myocarditis can lead to dilated cardiomyopath (DCM). DCM is one of the leading causes of the need for heart transplantation, so …
Role of Coxsackievirus B3-Induced Immune Responses in the Transition from Myocarditis to Dilated Cardiomyopathy and Heart Failure.
Yip F, Lai B, Yang D. Yip F, et al. Among authors: yang d. Int J Mol Sci. 2023 Apr 23;24(9):7717. doi: 10.3390/ijms24097717. Int J Mol Sci. 2023. PMID: 37175422 Free PMC article. Review.
However, it is unclear which alterations within host immune profiles are crucial to dictating the outcomes of myocarditis. Coxsackievirus B3 (CVB3) is a well-studied virus that has been identified as a causal agent of myocarditis in various models, along with other viruses …
However, it is unclear which alterations within host immune profiles are crucial to dictating the outcomes of myocarditis. Coxsackievirus B3 …
Genetic determinants of coxsackievirus B3 pathogenesis.
McManus BM, Yanagawa B, Rezai N, Luo H, Taylor L, Zhang M, Yuan J, Buckley J, Triche T, Schreiner G, Yang D. McManus BM, et al. Among authors: yang d. Ann N Y Acad Sci. 2002 Dec;975:169-79. doi: 10.1111/j.1749-6632.2002.tb05950.x. Ann N Y Acad Sci. 2002. PMID: 12538163
We first elucidated up- or downregulated transcriptional activities in CVB3-infected hearts by mRNA differential display. Further characterization of these regulated genes including Nip21, IP10, and IGTPase, and study of their role in CVB3-infection are underway. .. …
We first elucidated up- or downregulated transcriptional activities in CVB3-infected hearts by mRNA differential display. Further cha …
Cleavage and degradation of EDEM1 promotes coxsackievirus B3 replication via ATF6a-mediated unfolded protein response signalling.
Zhang HM, Qiu Y, Zhao G, Wang H, Chen YT, Aghakeshmiri S, Hanson P, Yang D. Zhang HM, et al. Among authors: yang d. Cell Microbiol. 2020 Jul;22(7):e13198. doi: 10.1111/cmi.13198. Epub 2020 Mar 9. Cell Microbiol. 2020. PMID: 32083795
Our previous study of coxsackievirus B3 (CVB3)-induced unfolded protein responses (UPR) found that overexpression of ATF6a enhances CVB3 VP1 capsid protein production and increases viral particle formation. ...Furthermore, we found that EDEM1 was cleaved by both …
Our previous study of coxsackievirus B3 (CVB3)-induced unfolded protein responses (UPR) found that overexpression of ATF6a enhances …
Coxsackievirus B3-Induced m6A Modification of RNA Enhances Viral Replication via Suppression of YTHDF-Mediated Stress Granule Formation.
Zhao G, Zhang HM, Chen YT, Shi K, Aghakeshmiri S, Yip F, Luo H, McManus B, Yang D. Zhao G, et al. Among authors: yang d. Microorganisms. 2024 Oct 26;12(11):2152. doi: 10.3390/microorganisms12112152. Microorganisms. 2024. PMID: 39597541 Free PMC article.
As a positive-sense single-stranded RNA virus, CVB3 replicates exclusively in the cytoplasm through a cap-independent translation initiation mechanism. ...This study provides a potential therapeutic strategy for CVB3-induced myocarditis....
As a positive-sense single-stranded RNA virus, CVB3 replicates exclusively in the cytoplasm through a cap-independent translation ini …
Myocarditis as systemic disease: new perspectives on pathogenesis.
Carthy CM, Yang D, Anderson DR, Wilson JE, McManus BM. Carthy CM, et al. Among authors: yang d. Clin Exp Pharmacol Physiol. 1997 Dec;24(12):997-1003. doi: 10.1111/j.1440-1681.1997.tb02739.x. Clin Exp Pharmacol Physiol. 1997. PMID: 9406676 Review.
Many competing hypotheses exist as to the pathogenesis of CVB3-induced myocarditis, including direct virus-induced myocyte damage and immunopathological disease with autoimmune sequelae. ...Most apoptotic bodies in cardiac tissue are derived from immune cells. 4. Beyond in …
Many competing hypotheses exist as to the pathogenesis of CVB3-induced myocarditis, including direct virus-induced myocyte damage and …
MicroRNA-203 enhances coxsackievirus B3 replication through targeting zinc finger protein-148.
Hemida MG, Ye X, Zhang HM, Hanson PJ, Liu Z, McManus BM, Yang D. Hemida MG, et al. Among authors: yang d. Cell Mol Life Sci. 2013 Jan;70(2):277-91. doi: 10.1007/s00018-012-1104-4. Epub 2012 Jul 29. Cell Mol Life Sci. 2013. PMID: 22842794 Free PMC article.
Coxsackievirus B3 (CVB3) is the primary causal agent of viral myocarditis. During infection, it hijacks host genes to favour its own replication. ...Ectopic expression of miR-203 downregulated ZFP-148 translation, increased cell viability and subsequently enhanced CVB3 …
Coxsackievirus B3 (CVB3) is the primary causal agent of viral myocarditis. During infection, it hijacks host genes to favour its own …
58 results