Abstract
The inability of traditional risk factors such as hypercholesterolemia, hypertension, and smoking to explain the incidence of atherosclerosis (AT) in about 50% of the cases prompted a search for additional putative risk factors involved in the development of the disease. Infectious agents have long been suspected to initiate/contribute to the process of AT. It has also been suggested that inflammation, either related to infectious agents or independent from infection, may mediate the atherogenic process [1, 2].
Publication types
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Research Support, Non-U.S. Gov't
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Review
MeSH terms
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Animals
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Arteriosclerosis / etiology*
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Arteriosclerosis / microbiology
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Arteriosclerosis / virology
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Chlamydophila Infections / complications
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Chlamydophila pneumoniae / pathogenicity
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Chronic Disease
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Cytomegalovirus / pathogenicity
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Cytomegalovirus Infections / complications
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Disease Models, Animal
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Herpes Simplex / complications
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Humans
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Infections / complications*
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Inflammation / complications
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Risk Factors
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Seroepidemiologic Studies
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Simplexvirus / pathogenicity