ETO/MTG8 is an inhibitor of C/EBPbeta activity and a regulator of early adipogenesis

Mol Cell Biol. 2004 Nov;24(22):9863-72. doi: 10.1128/MCB.24.22.9863-9872.2004.

Abstract

The putative transcriptional corepressor ETO/MTG8 has been extensively studied due to its involvement in a chromosomal translocation causing the t(8;21) form of acute myeloid leukemia. Despite this, the role of ETO in normal physiology has remained obscure. Here we show that ETO is highly expressed in preadipocytes and acts as an inhibitor of C/EBPbeta during early adipogenesis, contributing to its characteristically delayed activation. ETO prevents both the transcriptional activation of the C/EBPalpha promoter by C/EBPbeta and its concurrent accumulation in centromeric sites during early adipogenesis. ETO expression rapidly reduces after the initiation of adipogenesis, and this is essential to the normal induction of adipogenic gene expression. These findings define, for the first time, a molecular role for ETO in normal physiology as an inhibitor of C/EBPbeta and a novel regulator of early adipogenesis.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • 3T3-L1 Cells
  • Adipocytes / metabolism
  • Adipose Tissue / growth & development*
  • Adipose Tissue / metabolism*
  • Animals
  • CCAAT-Enhancer-Binding Protein-beta / antagonists & inhibitors*
  • Cell Line
  • DNA-Binding Proteins / genetics
  • DNA-Binding Proteins / metabolism*
  • Gene Expression
  • Humans
  • Lipid Metabolism
  • Mice
  • Proto-Oncogene Proteins / genetics
  • Proto-Oncogene Proteins / metabolism*
  • RUNX1 Translocation Partner 1 Protein
  • Recombinant Fusion Proteins / genetics
  • Recombinant Fusion Proteins / metabolism
  • Transcription Factors / genetics
  • Transcription Factors / metabolism*
  • Transfection

Substances

  • CCAAT-Enhancer-Binding Protein-beta
  • DNA-Binding Proteins
  • MTG8 protein, mouse
  • Proto-Oncogene Proteins
  • RUNX1 Translocation Partner 1 Protein
  • RUNX1T1 protein, human
  • Recombinant Fusion Proteins
  • Transcription Factors