TRIB3-P62 interaction, diabetes and autophagy

Oncotarget. 2015 Oct 27;6(33):34061-2. doi: 10.18632/oncotarget.6108.

Authors

Fang Hua¹, Zhuo-Wei Hu¹

Affiliation

¹ Immunology and Cancer Pharmacology Group, State Key Laboratory of Bioactive Substance and Function of Natural Medicines, Institute of Materia Medica, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, P.R. China.

No abstract available

Keywords: T cell activation; anergy; interleukin-2; mTOR; microRNA.

Publication types

Editorial

MeSH terms

Autophagy / physiology*
Cell Cycle Proteins / metabolism*
Diabetes Mellitus / pathology*
Humans
Hyperinsulinism / blood
Hyperinsulinism / pathology
Insulin / blood
Insulin-Like Growth Factor I / metabolism
Neoplasms / blood
Neoplasms / pathology*
Protein Serine-Threonine Kinases / antagonists & inhibitors*
Protein Serine-Threonine Kinases / metabolism
RNA-Binding Proteins / metabolism*
Repressor Proteins / metabolism*
Stress, Physiological

Substances

Cell Cycle Proteins
Insulin
P62 protein, human
RNA-Binding Proteins
Repressor Proteins
TRIB3 protein, human
Insulin-Like Growth Factor I
Protein Serine-Threonine Kinases