Corrigendum: The use of progeroid DNA repair-deficient mice for assessing anti-aging compounds, illustrating the benefits of nicotinamide riboside

Front Aging. 2022 Nov 23:3:1086552. doi: 10.3389/fragi.2022.1086552. eCollection 2022.

María B Birkisdóttir^{1

2}, Ivar van Galen^{1

2}, Renata M C Brandt³, Sander Barnhoorn³, Nicole van Vliet³, Claire van Dijk^{3

4}, Bhawani Nagarajah⁵, Sandra Imholz⁵, Conny T van Oostrom⁵, Erwin Reiling⁵, Ákos Gyenis⁶, Pier G Mastroberardino^{3

7

8}, Dick Jaarsma⁹, Harry van Steeg⁵, Jan H J Hoeijmakers^{1

2

3

6}, Martijn E T Dollé⁵, Wilbert P Vermeij^{1

2}

¹ Princess Máxima Center for Pediatric Oncology, Utrecht, Netherlands.
² Oncode Institute, Utrecht, Netherlands.
³ Department of Molecular Genetics, Erasmus MC Cancer Institute, Erasmus University Medical Center, Rotterdam, Netherlands.
⁴ Department of Hematology, Erasmus University Medical Center, Rotterdam, Netherlands.
⁵ Centre for Health Protection, National Institute for Public Health and the Environment, (RIVM), Bilthoven, Netherlands.
⁶ Faculty of Medicine, CECAD, Institute for Genome Stability in Aging and Disease, University of Cologne, Cologne, Germany.
⁷ IFOM-The FIRC Institute of Molecular Oncology, Milan, Italy.
⁸ Department of Life, Health, and Environmental Sciences, University of L'Aquila, L'Aquila, Italy.
⁹ Department of Neuroscience, Erasmus University Medical Center, Rotterdam, Netherlands.

Abstract

[This corrects the article DOI: 10.3389/fragi.2022.1005322.].

Keywords: DNA damage repair; NAD; aging; anti-aging interventions; dietary restriction mimetics; pharmacological screening; progeria.