Rhabdomyolysis in pontocerebellar hypoplasia type 2 (PCH-2)

Neuromuscul Disord. 2008 Jan;18(1):52-8. doi: 10.1016/j.nmd.2007.08.001. Epub 2007 Sep 6.

Abstract

Pontocerebellar hypoplasia type 2, an autosomal recessive neurodegeneration with prenatal onset, is characterised by progressive microcephaly and chorea/dystonia and has not previously been associated with muscular involvement. The gene associated with PCH-2 is unknown. An episode of rhabdomyolysis is reported in two non-related children with PCH-2, fatal in one, precipitated by intercurrent disease. Muscle biopsies in two other PCH-2 patients, and in one rhabdomyolysis patient whose biopsy antedated this complication showed areas of myofibrillar disruption or necrosis. Postmortem muscle sampled in another case without neuromuscular symptoms revealed focal necrosis, regenerating small fibres and upregulation of HLA-ABC. Random serum creatine kinase values in six other PCH-2 patients without clinical signs of neuromuscular involvement were increased in four. Collected data provide preliminary evidence of a subclinical myopathy associated with PCH-2.

Publication types

  • Case Reports

MeSH terms

  • Adult
  • Cerebellum / abnormalities*
  • Child, Preschool
  • Chromosome Disorders / genetics
  • Chromosome Disorders / pathology
  • Chromosome Disorders / physiopathology
  • Creatine Kinase / blood
  • Female
  • Genes, Recessive / genetics
  • HLA Antigens / analysis
  • HLA Antigens / metabolism
  • Humans
  • Infant
  • Infant, Newborn
  • Male
  • Microscopy, Electron, Transmission
  • Muscle Fibers, Skeletal / metabolism
  • Muscle Fibers, Skeletal / pathology
  • Muscle, Skeletal / metabolism
  • Muscle, Skeletal / pathology*
  • Muscle, Skeletal / physiopathology
  • Necrosis / genetics
  • Necrosis / pathology
  • Necrosis / physiopathology
  • Olivopontocerebellar Atrophies / complications*
  • Olivopontocerebellar Atrophies / pathology
  • Olivopontocerebellar Atrophies / physiopathology
  • Pons / abnormalities*
  • Rhabdomyolysis / genetics
  • Rhabdomyolysis / pathology*
  • Rhabdomyolysis / physiopathology

Substances

  • HLA Antigens
  • Creatine Kinase