Chang M - Search Results

1

Expression and purification of hexahistidine-tagged human glutathione S-transferase P1-1 in Escherichia coli.

Chang M, Bolton JL, Blond SY. Chang M, et al. Protein Expr Purif. 1999 Dec;17(3):443-8. doi: 10.1006/prep.1999.1149. Protein Expr Purif. 1999. PMID: 10600464

2

Inhibition of cellular enzymes by equine catechol estrogens in human breast cancer cells: specificity for glutathione S-transferase P1-1.

Yao J, Chang M, Li Y, Pisha E, Liu X, Yao D, Elguindi EC, Blond SY, Bolton JL. Yao J, et al. Among authors: chang m. Chem Res Toxicol. 2002 Jul;15(7):935-42. doi: 10.1021/tx020018i. Chem Res Toxicol. 2002. PMID: 12119004

We have previously shown that human GST P1-1, which is the most widely distributed extrahepatic isozyme, could be inactivated by the catechol estrogen metabolite 4-hydroxyequilenin (4-OHEN) in vitro [Chang, M., Shin, Y. G., van Breemen, R. B., Blond, S. Y., and Bolt …

We have previously shown that human GST P1-1, which is the most widely distributed extrahepatic isozyme, could be inactivated by the catecho …

3

Structural and functional consequences of inactivation of human glutathione S-transferase P1-1 mediated by the catechol metabolite of equine estrogens, 4-hydroxyequilenin.

Chang M, Shin YG, van Breemen RB, Blond SY, Bolton JL. Chang M, et al. Biochemistry. 2001 Apr 17;40(15):4811-20. doi: 10.1021/bi002513o. Biochemistry. 2001. PMID: 11294649

4

Inhibition of glutathione S-transferase activity by the quinoid metabolites of equine estrogens.

Chang M, Zhang F, Shen L, Pauss N, Alam I, van Breemen RB, Blond SY, Bolton JL. Chang M, et al. Chem Res Toxicol. 1998 Jul;11(7):758-65. doi: 10.1021/tx9702190. Chem Res Toxicol. 1998. PMID: 9671538

5

Quinoids as reactive intermediates in estrogen carcinogenesis.

Bolton JL, Chang M. Bolton JL, et al. Among authors: chang m. Adv Exp Med Biol. 2001;500:497-507. doi: 10.1007/978-1-4615-0667-6_75. Adv Exp Med Biol. 2001. PMID: 11764987 Review. No abstract available.

6

Catechol estrogen 4-hydroxyequilenin is a substrate and an inhibitor of catechol-O-methyltransferase.

Yao J, Li Y, Chang M, Wu H, Yang X, Goodman JE, Liu X, Liu H, Mesecar AD, Van Breemen RB, Yager JD, Bolton JL. Yao J, et al. Among authors: chang m. Chem Res Toxicol. 2003 May;16(5):668-75. doi: 10.1021/tx0340549. Chem Res Toxicol. 2003. PMID: 12755597

7

Functional and structural comparisons of cysteine residues in the Val108 wild type and Met108 variant of human soluble catechol O-methyltransferase.

Li Y, Yang X, Chang M, Yager JD, van Breemen RB, Bolton JL. Li Y, et al. Among authors: chang m. Chem Biol Interact. 2005 Apr 15;152(2-3):151-63. doi: 10.1016/j.cbi.2005.03.001. Chem Biol Interact. 2005. PMID: 15840388

8

Equine catechol estrogen 4-hydroxyequilenin is a more potent inhibitor of the variant form of catechol-O-methyltransferase.

Li Y, Yao J, Chang M, Nikolic D, Yu L, Yager JD, Mesecar AD, van Breemen RB, Bolton JL. Li Y, et al. Among authors: chang m. Chem Res Toxicol. 2004 Apr;17(4):512-20. doi: 10.1021/tx0342464. Chem Res Toxicol. 2004. PMID: 15089093

9

Altered apoptotic response in MCF 10A cells treated with the equine estrogen metabolite, 4-hydroxyequilenin.

Li Y, Yao J, Chang M, Cuendet M, Bolton JL. Li Y, et al. Among authors: chang m. Toxicol Lett. 2004 Dec 30;154(3):225-33. doi: 10.1016/j.toxlet.2004.08.006. Toxicol Lett. 2004. PMID: 15501614

10

Unexpected hormonal activity of a catechol equine estrogen metabolite reveals reversible glutathione conjugation.

Peng KW, Chang M, Wang YT, Wang Z, Qin Z, Bolton JL, Thatcher GR. Peng KW, et al. Among authors: chang m. Chem Res Toxicol. 2010 Aug 16;23(8):1374-83. doi: 10.1021/tx100129h. Chem Res Toxicol. 2010. PMID: 20540524 Free PMC article.

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