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Interleukin-1beta, transforming growth factor-beta1, and bradykinin attenuate cyclic AMP production by human pulmonary artery smooth muscle cells in response to prostacyclin analogues and prostaglandin E2 by cyclooxygenase-2 induction and downregulation of adenylyl cyclase isoforms 1, 2, and 4.
El-Haroun H, Bradbury D, Clayton A, Knox AJ. El-Haroun H, et al. Among authors: bradbury d. Circ Res. 2004 Feb 20;94(3):353-61. doi: 10.1161/01.RES.0000111801.48626.F4. Epub 2003 Dec 11. Circ Res. 2004. PMID: 14670842
IL-1beta, BK, and TGF-beta1 attenuate PGI2-mediated cAMP formation in human pulmonary artery smooth muscle cells by multiple mechanisms involving p38 MAP kinase and PKA.
El-Haroun H, Clarke DL, Deacon K, Bradbury D, Clayton A, Sutcliffe A, Knox AJ. El-Haroun H, et al. Among authors: bradbury d. Am J Physiol Lung Cell Mol Physiol. 2008 Mar;294(3):L553-62. doi: 10.1152/ajplung.00044.2006. Epub 2007 Dec 21. Am J Physiol Lung Cell Mol Physiol. 2008. PMID: 18156442 Free article.
Cyclooxygenase-2 induction by bradykinin in human pulmonary artery smooth muscle cells is mediated by the cyclic AMP response element through a novel autocrine loop involving endogenous prostaglandin E2, E-prostanoid 2 (EP2), and EP4 receptors.
Bradbury DA, Newton R, Zhu YM, El-Haroun H, Corbett L, Knox AJ. Bradbury DA, et al. J Biol Chem. 2003 Dec 12;278(50):49954-64. doi: 10.1074/jbc.M307964200. Epub 2003 Sep 29. J Biol Chem. 2003. PMID: 14517215 Free article.
96 results