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Involvement of nitric oxide synthase and ROS-mediated activation of L-type voltage-gated Ca2+ channels in NMDA-induced DPYSL3 degradation.
Kowara R, Moraleja KL, Chakravarthy B. Kowara R, et al. Among authors: chakravarthy b. Brain Res. 2006 Nov 13;1119(1):40-9. doi: 10.1016/j.brainres.2006.08.047. Epub 2006 Sep 20. Brain Res. 2006. PMID: 16987501
While NMDA-induced DPYSL3 truncation was blocked by both NMDA receptor antagonist (MK801) [Kowara, R., Chen, Q., Milliken, M., Chakravarthy, B., 2005. Calpain-mediated degradation of dihydropyrimidinase-like 3 protein (DPYSL3) in response to NMDA and H(2)O(2) toxici …
While NMDA-induced DPYSL3 truncation was blocked by both NMDA receptor antagonist (MK801) [Kowara, R., Chen, Q., Milliken, M., Chakravart …
PLA(2) signaling is involved in calpain-mediated degradation of synaptic dihydropyrimidinase-like 3 protein in response to NMDA excitotoxicity.
Kowara R, Moraleja KL, Chakravarthy B. Kowara R, et al. Among authors: chakravarthy b. Neurosci Lett. 2008 Jan 17;430(3):197-202. doi: 10.1016/j.neulet.2007.10.036. Epub 2007 Nov 5. Neurosci Lett. 2008. PMID: 18053648
Recently we have shown that glutamate excitotoxicity and oxidative stress result in calpain-dependent cleavage of DPYSL3, and that NOS plays a role in this process [R. Kowara, Q. Chen, M. Milliken, B. Chakravarthy, Calpain-mediated truncation of dihydropyrimidinase- …
Recently we have shown that glutamate excitotoxicity and oxidative stress result in calpain-dependent cleavage of DPYSL3, and that NOS plays …
189 results