Kong D - Search Results

1

Notch-1 induces epithelial-mesenchymal transition consistent with cancer stem cell phenotype in pancreatic cancer cells.

Bao B, Wang Z, Ali S, Kong D, Li Y, Ahmad A, Banerjee S, Azmi AS, Miele L, Sarkar FH. Bao B, et al. Among authors: kong d. Cancer Lett. 2011 Aug 1;307(1):26-36. doi: 10.1016/j.canlet.2011.03.012. Epub 2011 Apr 3. Cancer Lett. 2011. Retraction in: Cancer Lett. 2018 Jun 1;423:153. doi: 10.1016/j.canlet.2018.03.020. PMID: 21463919 Free PMC article. Retracted.

2

PDGF-D signaling: a novel target in cancer therapy.

Wang Z, Kong D, Li Y, Sarkar FH. Wang Z, et al. Among authors: kong d. Curr Drug Targets. 2009 Jan;10(1):38-41. doi: 10.2174/138945009787122914. Curr Drug Targets. 2009. PMID: 19149534 Review.

3

Acquisition of epithelial-mesenchymal transition phenotype of gemcitabine-resistant pancreatic cancer cells is linked with activation of the notch signaling pathway.

Wang Z, Li Y, Kong D, Banerjee S, Ahmad A, Azmi AS, Ali S, Abbruzzese JL, Gallick GE, Sarkar FH. Wang Z, et al. Among authors: kong d. Cancer Res. 2009 Mar 15;69(6):2400-7. doi: 10.1158/0008-5472.CAN-08-4312. Epub 2009 Mar 10. Cancer Res. 2009. PMID: 19276344 Free PMC article.

4

Inactivation of uPA and its receptor uPAR by 3,3'-diindolylmethane (DIM) leads to the inhibition of prostate cancer cell growth and migration.

Ahmad A, Kong D, Sarkar SH, Wang Z, Banerjee S, Sarkar FH. Ahmad A, et al. Among authors: kong d. J Cell Biochem. 2009 Jun 1;107(3):516-27. doi: 10.1002/jcb.22152. J Cell Biochem. 2009. PMID: 19330806 Free PMC article.

5

3,3'-Diindolylmethane enhances taxotere-induced apoptosis in hormone-refractory prostate cancer cells through survivin down-regulation.

Rahman KM, Banerjee S, Ali S, Ahmad A, Wang Z, Kong D, Sakr WA. Rahman KM, et al. Among authors: kong d. Cancer Res. 2009 May 15;69(10):4468-75. doi: 10.1158/0008-5472.CAN-08-4423. Epub 2009 May 12. Cancer Res. 2009. PMID: 19435906

6

3,3'-Diindolylmethane enhances chemosensitivity of multiple chemotherapeutic agents in pancreatic cancer.

Banerjee S, Wang Z, Kong D, Sarkar FH. Banerjee S, et al. Among authors: kong d. Cancer Res. 2009 Jul 1;69(13):5592-600. doi: 10.1158/0008-5472.CAN-09-0838. Epub 2009 Jun 16. Cancer Res. 2009. Retraction in: Cancer Res. 2018 Sep 15;78(18):5467. doi: 10.1158/0008-5472.CAN-18-1176. PMID: 19531648 Free PMC article. Retracted.

7

miR-200 regulates PDGF-D-mediated epithelial-mesenchymal transition, adhesion, and invasion of prostate cancer cells.

Kong D, Li Y, Wang Z, Banerjee S, Ahmad A, Kim HR, Sarkar FH. Kong D, et al. Stem Cells. 2009 Aug;27(8):1712-21. doi: 10.1002/stem.101. Stem Cells. 2009. PMID: 19544444 Free PMC article.

8

Up-regulation of miR-200 and let-7 by natural agents leads to the reversal of epithelial-to-mesenchymal transition in gemcitabine-resistant pancreatic cancer cells.

Li Y, VandenBoom TG 2nd, Kong D, Wang Z, Ali S, Philip PA, Sarkar FH. Li Y, et al. Among authors: kong d. Cancer Res. 2009 Aug 15;69(16):6704-12. doi: 10.1158/0008-5472.CAN-09-1298. Epub 2009 Aug 4. Cancer Res. 2009. PMID: 19654291 Free PMC article.

9

Down-regulation of uPA and uPAR by 3,3'-diindolylmethane contributes to the inhibition of cell growth and migration of breast cancer cells.

Ahmad A, Kong D, Wang Z, Sarkar SH, Banerjee S, Sarkar FH. Ahmad A, et al. Among authors: kong d. J Cell Biochem. 2009 Nov 1;108(4):916-25. doi: 10.1002/jcb.22323. J Cell Biochem. 2009. Retraction in: J Cell Biochem. 2016 Aug;117(8):1959. doi: 10.1002/jcb.25583. PMID: 19693769 Free PMC article. Retracted.

10

FoxM1 down-regulation leads to inhibition of proliferation, migration and invasion of breast cancer cells through the modulation of extra-cellular matrix degrading factors.

Ahmad A, Wang Z, Kong D, Ali S, Li Y, Banerjee S, Ali R, Sarkar FH. Ahmad A, et al. Among authors: kong d. Breast Cancer Res Treat. 2010 Jul;122(2):337-46. doi: 10.1007/s10549-009-0572-1. Epub 2009 Oct 8. Breast Cancer Res Treat. 2010. Retraction in: Breast Cancer Res Treat. 2016 Aug;158(3):607. doi: 10.1007/s10549-016-3904-y. PMID: 19813088 Retracted.

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