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Most factor VIII B domain missense mutations are unlikely to be causative mutations for severe hemophilia A: implications for genotyping.
J Thromb Haemost. 2011 Jun;9(6):1183-90. doi: 10.1111/j.1538-7836.2011.04268.x.
J Thromb Haemost. 2011.
PMID: 21645226
Free PMC article.
Bioengineering of coagulation factor VIII for efficient expression through elimination of a dispensable disulfide loop.
Selvaraj SR, Scheller AN, Miao HZ, Kaufman RJ, Pipe SW.
Selvaraj SR, et al.
J Thromb Haemost. 2012 Jan;10(1):107-15. doi: 10.1111/j.1538-7836.2011.04545.x.
J Thromb Haemost. 2012.
PMID: 22044596
Free PMC article.
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Gene editing in hemophilia: a "CRISPR" choice?
Pipe SW, Selvaraj SR.
Pipe SW, et al. Among authors: selvaraj sr.
Blood. 2019 Jun 27;133(26):2733-2734. doi: 10.1182/blood.2019001180.
Blood. 2019.
PMID: 31248872
Free article.
No abstract available.
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Not in the genotype: can unexplained hemophilia A result from "micro(RNA) management"?
Selvaraj SR, Pipe SW.
Selvaraj SR, et al.
Transfusion. 2020 Feb;60(2):227-228. doi: 10.1111/trf.15668.
Transfusion. 2020.
PMID: 32022934
Free article.
No abstract available.
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Oxidative folding and assembly with transthyretin are sequential events in the biogenesis of retinol binding protein in the endoplasmic reticulum.
Selvaraj SR, Bhatia V, Tatu U.
Selvaraj SR, et al.
Mol Biol Cell. 2008 Dec;19(12):5579-92. doi: 10.1091/mbc.e08-01-0026. Epub 2008 Sep 24.
Mol Biol Cell. 2008.
PMID: 18815280
Free PMC article.
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