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Progressive glycosylation of the haemagglutinin of avian influenza H5N1 modulates virus replication, virulence and chicken-to-chicken transmission without significant impact on antigenic drift.
Abdelwhab EM, Veits J, Tauscher K, Ziller M, Grund C, Hassan MK, Shaheen M, Harder TC, Teifke J, Stech J, Mettenleiter TC. Abdelwhab EM, et al. Among authors: stech j. J Gen Virol. 2016 Dec;97(12):3193-3204. doi: 10.1099/jgv.0.000648. Epub 2016 Oct 28. J Gen Virol. 2016. PMID: 27902339
A Dual Motif in the Hemagglutinin of H5N1 Goose/Guangdong-Like Highly Pathogenic Avian Influenza Virus Strains Is Conserved from Their Early Evolution and Increases both Membrane Fusion pH and Virulence.
Wessels U, Abdelwhab EM, Veits J, Hoffmann D, Mamerow S, Stech O, Hellert J, Beer M, Mettenleiter TC, Stech J. Wessels U, et al. Among authors: stech j. J Virol. 2018 Aug 16;92(17):e00778-18. doi: 10.1128/JVI.00778-18. Print 2018 Sep 1. J Virol. 2018. PMID: 29899102 Free PMC article.
Acquisition of a polybasic hemagglutinin cleavage site by a low-pathogenic avian influenza virus is not sufficient for immediate transformation into a highly pathogenic strain.
Stech O, Veits J, Weber S, Deckers D, Schröer D, Vahlenkamp TW, Breithaupt A, Teifke J, Mettenleiter TC, Stech J. Stech O, et al. Among authors: stech j. J Virol. 2009 Jun;83(11):5864-8. doi: 10.1128/JVI.02649-08. Epub 2009 Mar 18. J Virol. 2009. PMID: 19297482 Free PMC article.
56 results