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miR-21-mediated decreased neutrophil apoptosis is a determinant of impaired coronary collateral growth in metabolic syndrome.
Hutcheson R, Terry R, Hutcheson B, Jadhav R, Chaplin J, Smith E, Barrington R, Proctor SD, Rocic P. Hutcheson R, et al. Among authors: hutcheson b. Am J Physiol Heart Circ Physiol. 2015 Jun 1;308(11):H1323-35. doi: 10.1152/ajpheart.00654.2014. Epub 2015 Apr 3. Am J Physiol Heart Circ Physiol. 2015. PMID: 25840830 Free PMC article.
Elevated 20-HETE impairs coronary collateral growth in metabolic syndrome via endothelial dysfunction.
Joseph G, Soler A, Hutcheson R, Hunter I, Bradford C, Hutcheson B, Gotlinger KH, Jiang H, Falck JR, Proctor S, Schwartzman ML, Rocic P. Joseph G, et al. Among authors: hutcheson r, hutcheson b. Am J Physiol Heart Circ Physiol. 2017 Mar 1;312(3):H528-H540. doi: 10.1152/ajpheart.00561.2016. Epub 2016 Dec 23. Am J Physiol Heart Circ Physiol. 2017. PMID: 28011587 Free PMC article.
Elevated 20-HETE in metabolic syndrome regulates arterial stiffness and systolic hypertension via MMP12 activation.
Soler A, Hunter I, Joseph G, Hutcheson R, Hutcheson B, Yang J, Zhang FF, Joshi SR, Bradford C, Gotlinger KH, Maniyar R, Falck JR, Proctor S, Schwartzman ML, Gupte SA, Rocic P. Soler A, et al. Among authors: hutcheson b. J Mol Cell Cardiol. 2018 Apr;117:88-99. doi: 10.1016/j.yjmcc.2018.02.005. Epub 2018 Feb 8. J Mol Cell Cardiol. 2018. PMID: 29428638 Free PMC article.
Corrigendum to "Elevated 20-HETE in metabolic syndrome regulates arterial stiffness and systolic hypertension via MMP12 activation" [J. Mol. Cell. Cardiol. 117 (2018) 88-99].
Soler A, Hunter I, Joseph G, Hutcheson R, Hutcheson B, Yang J, Zhang FF, Joshi SR, Bradford C, Gotlinger KH, Maniyar R, Falck JR, Proctor S, Schwartzman ML, Gupte SA, Rocic P. Soler A, et al. Among authors: hutcheson b. J Mol Cell Cardiol. 2018 Aug;121:308. doi: 10.1016/j.yjmcc.2018.03.015. Epub 2018 Apr 4. J Mol Cell Cardiol. 2018. PMID: 29625729 No abstract available.
21 results