Similar articles for PMID: 11390973

Year	Number of Results
1991	2
1992	3
1993	3
1994	3
1995	15
1996	14
1997	10
1998	17
1999	12
2000	16
2001	17
2002	11
2003	14
2004	8
2005	10
2006	3
2007	4
2008	6
2009	6
2010	4
2011	3
2012	5
2013	5
2014	6
2016	2
2017	4
2018	3
2019	10
2020	6
2021	5
2022	2
2024	0

1

Structural basis for fibroblast growth factor receptor 2 activation in Apert syndrome.

Ibrahimi OA, Eliseenkova AV, Plotnikov AN, Yu K, Ornitz DM, Mohammadi M. Ibrahimi OA, et al. Proc Natl Acad Sci U S A. 2001 Jun 19;98(13):7182-7. doi: 10.1073/pnas.121183798. Epub 2001 Jun 5. Proc Natl Acad Sci U S A. 2001. PMID: 11390973 Free PMC article.

2

Apert syndrome mutations in fibroblast growth factor receptor 2 exhibit increased affinity for FGF ligand.

Anderson J, Burns HD, Enriquez-Harris P, Wilkie AO, Heath JK. Anderson J, et al. Hum Mol Genet. 1998 Sep;7(9):1475-83. doi: 10.1093/hmg/7.9.1475. Hum Mol Genet. 1998. PMID: 9700203

3

Loss of fibroblast growth factor receptor 2 ligand-binding specificity in Apert syndrome.

Yu K, Herr AB, Waksman G, Ornitz DM. Yu K, et al. Proc Natl Acad Sci U S A. 2000 Dec 19;97(26):14536-41. doi: 10.1073/pnas.97.26.14536. Proc Natl Acad Sci U S A. 2000. PMID: 11121055 Free PMC article.

4

Craniosynostosis and related limb anomalies.

Wilkie AO, Oldridge M, Tang Z, Maxson RE Jr. Wilkie AO, et al. Novartis Found Symp. 2001;232:122-33; discussion 133-43. doi: 10.1002/0470846658.ch9. Novartis Found Symp. 2001. PMID: 11277076 Review.

5

A soluble form of fibroblast growth factor receptor 2 (FGFR2) with S252W mutation acts as an efficient inhibitor for the enhanced osteoblastic differentiation caused by FGFR2 activation in Apert syndrome.

Tanimoto Y, Yokozeki M, Hiura K, Matsumoto K, Nakanishi H, Matsumoto T, Marie PJ, Moriyama K. Tanimoto Y, et al. J Biol Chem. 2004 Oct 29;279(44):45926-34. doi: 10.1074/jbc.M404824200. Epub 2004 Aug 13. J Biol Chem. 2004. PMID: 15310757 Free article.

6

Sequence analyses and comparative modeling of fly and worm fibroblast growth factor receptors indicate that the determinants for FGF and heparin binding are retained in evolution.

Nagendra HG, Harrington AE, Harmer NJ, Pellegrini L, Blundell TL, Burke DF. Nagendra HG, et al. FEBS Lett. 2001 Jul 13;501(1):51-8. doi: 10.1016/s0014-5793(01)02603-5. FEBS Lett. 2001. PMID: 11457455 Free article.

7

Negative autoregulation of fibroblast growth factor receptor 2 expression characterizing cranial development in cases of Apert (P253R mutation) and Pfeiffer (C278F mutation) syndromes and suggesting a basis for differences in their cranial phenotypes.

Britto JA, Moore RL, Evans RD, Hayward RD, Jones BM. Britto JA, et al. J Neurosurg. 2001 Oct;95(4):660-73. doi: 10.3171/jns.2001.95.4.0660. J Neurosurg. 2001. PMID: 11596961

8

Understanding the molecular basis of Apert syndrome.

Ibrahimi OA, Chiu ES, McCarthy JG, Mohammadi M. Ibrahimi OA, et al. Plast Reconstr Surg. 2005 Jan;115(1):264-70. Plast Reconstr Surg. 2005. PMID: 15622262 Review.

9

Signaling by fibroblast growth factors (FGF) and fibroblast growth factor receptor 2 (FGFR2)-activating mutations blocks mineralization and induces apoptosis in osteoblasts.

Mansukhani A, Bellosta P, Sahni M, Basilico C. Mansukhani A, et al. J Cell Biol. 2000 Jun 12;149(6):1297-308. doi: 10.1083/jcb.149.6.1297. J Cell Biol. 2000. PMID: 10851026 Free PMC article.

10

Genomic screening of fibroblast growth-factor receptor 2 reveals a wide spectrum of mutations in patients with syndromic craniosynostosis.

Kan SH, Elanko N, Johnson D, Cornejo-Roldan L, Cook J, Reich EW, Tomkins S, Verloes A, Twigg SR, Rannan-Eliya S, McDonald-McGinn DM, Zackai EH, Wall SA, Muenke M, Wilkie AO. Kan SH, et al. Am J Hum Genet. 2002 Feb;70(2):472-86. doi: 10.1086/338758. Epub 2002 Jan 4. Am J Hum Genet. 2002. PMID: 11781872 Free PMC article.

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