Similar articles for PMID: 11134029

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1

Molecular basis of the Kell-null phenotype: a mutation at the splice site of human KEL gene abolishes the expression of Kell blood group antigens.

Yu LC, Twu YC, Chang CY, Lin M. Yu LC, et al. J Biol Chem. 2001 Mar 30;276(13):10247-52. doi: 10.1074/jbc.M009879200. Epub 2000 Dec 27. J Biol Chem. 2001. PMID: 11134029 Free article.

2

Point mutations characterize KEL10, the KEL3, KEL4, and KEL21 alleles, and the KEL17 and KEL11 alleles.

Lee S, Wu X, Son S, Naime D, Reid M, Okubo Y, Sistonen P, Redman C. Lee S, et al. Transfusion. 1996 Jun;36(6):490-4. doi: 10.1046/j.1537-2995.1996.36696269505.x. Transfusion. 1996. PMID: 8669078

3

Molecular defects underlying the Kell null phenotype.

Lee S, Russo DC, Reiner AP, Lee JH, Sy MY, Telen MJ, Judd WJ, Simon P, Rodrigues MJ, Chabert T, Poole J, Jovanovic-Srzentic S, Levene C, Yahalom V, Redman CM. Lee S, et al. J Biol Chem. 2001 Jul 20;276(29):27281-9. doi: 10.1074/jbc.M103433200. Epub 2001 May 24. J Biol Chem. 2001. PMID: 11375401 Free article.

4

Three uncommon KEL alleles in one family with unusual Kell phenotypes explain a 35-year old conundrum.

Karamatic Crew V, Poole J, Burton N, Daniels G. Karamatic Crew V, et al. Vox Sang. 2014 Apr;106(3):242-7. doi: 10.1111/vox.12094. Vox Sang. 2014. PMID: 24795954

5

Organization of the gene encoding the human Kell blood group protein.

Lee S, Zambas E, Green ED, Redman C. Lee S, et al. Blood. 1995 Mar 1;85(5):1364-70. Blood. 1995. PMID: 7858266 Free article.

6

The KEL24 and KEL14 alleles of the Kell blood group system.

Lee S, Naime D, Reid M, Redman C. Lee S, et al. Transfusion. 1997 Oct;37(10):1035-8. doi: 10.1046/j.1537-2995.1997.371098016441.x. Transfusion. 1997. PMID: 9354821

7

Molecular basis for the high-incidence antigens of the Kell blood group system.

Lee S, Naime DS, Reid ME, Redman CM. Lee S, et al. Transfusion. 1997 Nov-Dec;37(11-12):1117-22. doi: 10.1046/j.1537-2995.1997.37111298088039.x. Transfusion. 1997. PMID: 9426633

8

Heterozygosity for two novel null alleles of the KEL gene causes the Kell-null phenotype in a Japanese woman.

Koda Y, Soejima M, Tsuneoka M, Yasumoto K, Higashitani T, Sagawa K, Kimura H. Koda Y, et al. Br J Haematol. 2002 Apr;117(1):220-5. doi: 10.1046/j.1365-2141.2002.03368.x. Br J Haematol. 2002. PMID: 11918559 Free article.

9

Genetic diversity of KELnull and KELel: a nationwide Austrian survey.

Körmöczi GF, Wagner T, Jungbauer C, Vadon M, Ahrens N, Moll W, Mühlbacher A, Ozgül-Gülce S, Kleinrath T, Kilga-Nogler S, Schönitzer D, Gassner C. Körmöczi GF, et al. Transfusion. 2007 Apr;47(4):703-14. doi: 10.1111/j.1537-2995.2007.01174.x. Transfusion. 2007. PMID: 17381630

10

Point mutations in KEL exon 8 determine a high-incidence (RAZ) and a low-incidence (KEL25, VLAN) antigen of the Kell blood group system.

Lee S, Reid ME, Redman CM. Lee S, et al. Vox Sang. 2001 Nov;81(4):259-63. doi: 10.1046/j.1423-0410.2001.00119.x. Vox Sang. 2001. PMID: 11904003

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