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62 results

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Quoted phrase not found in phrase index: "Abnormal circulating aldosterone"
Page 1
Combined Angiotensin Receptor Antagonism and Neprilysin Inhibition.
Hubers SA, Brown NJ. Hubers SA, et al. Circulation. 2016 Mar 15;133(11):1115-24. doi: 10.1161/CIRCULATIONAHA.115.018622. Circulation. 2016. PMID: 26976916 Free PMC article. Review.
Angiotensin-converting enzyme inhibitors, angiotensin receptor blockers, beta-blockers, and aldosterone antagonists have improved mortality in patients with heart failure and reduced ejection fraction, but mortality remains high. ...In the Prospective Comparison of ARNI Wi …
Angiotensin-converting enzyme inhibitors, angiotensin receptor blockers, beta-blockers, and aldosterone antagonists have improved mor …
Mineralocorticoid Receptor Activation in Vascular Insulin Resistance and Dysfunction.
Igbekele AE, Jia G, Hill MA, Sowers JR, Jia G. Igbekele AE, et al. Int J Mol Sci. 2022 Aug 11;23(16):8954. doi: 10.3390/ijms23168954. Int J Mol Sci. 2022. PMID: 36012219 Free PMC article. Review.
Mineralocorticoid receptors (MRs), the principal receptors for the hormone aldosterone, play an important role in regulating renal sodium handling and blood pressure. ...This review highlights recent experimental and clinical data that support the contribution of …
Mineralocorticoid receptors (MRs), the principal receptors for the hormone aldosterone, play an important role in regulating renal so …
Nonmodulation and essential hypertension.
Hollenberg NK, Williams GH. Hollenberg NK, et al. Curr Hypertens Rep. 2006 May;8(2):127-31. doi: 10.1007/s11906-006-0008-9. Curr Hypertens Rep. 2006. PMID: 16672145 Review.
Nonmodulation is a process in which there is a disorder in angiotensin-dependent control of the renal circulation and adrenal aldosterone release. The abnormalities are associated with an inability to handle a sodium load and salt-sensitive hypertension. ...
Nonmodulation is a process in which there is a disorder in angiotensin-dependent control of the renal circulation and adrenal aldo …
The role of 11β-hydroxysteroid dehydrogenase type 2 in human hypertension.
Ferrari P. Ferrari P. Biochim Biophys Acta. 2010 Dec;1802(12):1178-87. doi: 10.1016/j.bbadis.2009.10.017. Epub 2009 Nov 10. Biochim Biophys Acta. 2010. PMID: 19909806 Free article. Review.
Cortisol and aldosterone have the same in vitro affinity for the mineralocorticoid receptor (MR), although in vivo only aldosterone acts as a physiologic agonist of the MR, despite circulating levels of cortisol in humans and corticosterone in rodents being t …
Cortisol and aldosterone have the same in vitro affinity for the mineralocorticoid receptor (MR), although in vivo only aldosteron …
11beta-hydroxysteroid dehydrogenase and its role in the syndrome of apparent mineralocorticoid excess.
White PC. White PC. Am J Med Sci. 2001 Dec;322(6):308-15. doi: 10.1097/00000441-200112000-00003. Am J Med Sci. 2001. PMID: 11780688 Review.
Excess secretion of aldosterone or other mineralocorticoids, or abnormal sensitivity to mineralocorticoids, may result in hypokalemia, suppressed plasma renin activity, and hypertension. ...Because mineralocorticoid receptors themselves have similar affinities for c …
Excess secretion of aldosterone or other mineralocorticoids, or abnormal sensitivity to mineralocorticoids, may result in hypo …
Splanchnic and systemic hemodynamic derangement in decompensated cirrhosis.
Møller S, Bendtsen F, Henriksen JH. Møller S, et al. Can J Gastroenterol. 2001 Feb;15(2):94-106. doi: 10.1155/2001/603012. Can J Gastroenterol. 2001. PMID: 11240379 Review.
Patients with cirrhosis and portal hypertension exhibit characteristic hemodynamic changes with hyperkinetic systemic circulation, abnormal distribution of blood volume and neurohumoral dysregulation. Their plasma and noncentral blood volumes are incre …
Patients with cirrhosis and portal hypertension exhibit characteristic hemodynamic changes with hyperkinetic systemic circulation, …
Normotensive primary aldosteronism.
Shiroto H, Ando H, Ebitani I, Hara M, Numazawa K, Kawamura S, Sasaki H. Shiroto H, et al. Am J Med. 1980 Oct;69(4):603-6. doi: 10.1016/0002-9343(80)90474-x. Am J Med. 1980. PMID: 6999899
After removal of the adenoma, the abnormalities subsided. The lack of hypertension in primary aldosteronism is a rare condition. ...As blood pressure response to the administration of angiotensin II was lower than is typical for primary aldosteronism, …
After removal of the adenoma, the abnormalities subsided. The lack of hypertension in primary aldosteronism is a rare conditio …
Resistant hypertension in the high-risk metabolic patient.
Chaudhary K, Buddineni JP, Nistala R, Whaley-Connell A. Chaudhary K, et al. Curr Diab Rep. 2011 Feb;11(1):41-6. doi: 10.1007/s11892-010-0155-x. Curr Diab Rep. 2011. PMID: 20941645 Review.
The metabolic syndrome is a constellation of metabolic and vascular abnormalities that include insulin resistance with compensatory hyperinsulinemia, central or visceral obesity, hypertension, dyslipidemia, microalbuminuria, and oxidative stress as well as prothrombotic an …
The metabolic syndrome is a constellation of metabolic and vascular abnormalities that include insulin resistance with compensatory h …
Activation and Inhibition of Sodium-Hydrogen Exchanger Is a Mechanism That Links the Pathophysiology and Treatment of Diabetes Mellitus With That of Heart Failure.
Packer M. Packer M. Circulation. 2017 Oct 17;136(16):1548-1559. doi: 10.1161/CIRCULATIONAHA.117.030418. Circulation. 2017. PMID: 29038209 Review.
Activation of the sodium-hydrogen exchanger in the heart and vasculature (NHE1 isoform) and the kidneys (NHE3 isoform) may serve as a common mechanism that links both disorders and may underlie their interplay. Insulin insensitivity and adipokine abnormalities (the hallmar …
Activation of the sodium-hydrogen exchanger in the heart and vasculature (NHE1 isoform) and the kidneys (NHE3 isoform) may serve as a common …
11 beta-Hydroxysteroid dehydrogenase and its role in the syndrome of apparent mineralocorticoid excess.
White PC, Mune T, Rogerson FM, Kayes KM, Agarwal AK. White PC, et al. Pediatr Res. 1997 Jan;41(1):25-9. doi: 10.1203/00006450-199701000-00004. Pediatr Res. 1997. PMID: 8979285 Review.
Excess secretion of aldosterone or other mineralocorticoids, or abnormal sensitivity to mineralocorticoids, may results in hypokalemia, suppressed plasma renin activity, and hypertension. ...Because mineralocorticoid receptors themselves have similar affinities for …
Excess secretion of aldosterone or other mineralocorticoids, or abnormal sensitivity to mineralocorticoids, may results in hyp …
62 results