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Quoted phrase not found in phrase index: "Abnormal circulating aldosterone"
Page 1
Combined Angiotensin Receptor Antagonism and Neprilysin Inhibition.
Hubers SA, Brown NJ. Hubers SA, et al. Circulation. 2016 Mar 15;133(11):1115-24. doi: 10.1161/CIRCULATIONAHA.115.018622. Circulation. 2016. PMID: 26976916 Free PMC article. Review.
Angiotensin-converting enzyme inhibitors, angiotensin receptor blockers, beta-blockers, and aldosterone antagonists have improved mortality in patients with heart failure and reduced ejection fraction, but mortality remains high. ...In the Prospective Comparison of ARNI Wi …
Angiotensin-converting enzyme inhibitors, angiotensin receptor blockers, beta-blockers, and aldosterone antagonists have improved mor …
Obesity and Cardiac Remodeling in Adults: Mechanisms and Clinical Implications.
Alpert MA, Karthikeyan K, Abdullah O, Ghadban R. Alpert MA, et al. Prog Cardiovasc Dis. 2018 Jul-Aug;61(2):114-123. doi: 10.1016/j.pcad.2018.07.012. Epub 2018 Jul 7. Prog Cardiovasc Dis. 2018. PMID: 29990533 Review.
In recent years experimental studies and some studies in humans suggest that certain neurohormonal and metabolic alterations that occur commonly in obesity may contribute to alterations in cardiac structure and function. These include activation of the renin-angiotensin-aldost …
In recent years experimental studies and some studies in humans suggest that certain neurohormonal and metabolic alterations that occur comm …
A Personalized Update on IgA Nephropathy: A New Vision and New Future Challenges.
Gutiérrez E, Carvaca-Fontán F, Luzardo L, Morales E, Alonso M, Praga M. Gutiérrez E, et al. Nephron. 2020;144(11):555-571. doi: 10.1159/000509997. Epub 2020 Aug 20. Nephron. 2020. PMID: 32818944 Free article. Review.
IgAN is characterized by dysregulation of the immune system, which causes an abnormal synthesis of IgA1 that is deglycosylated causing its mesangial deposition. ...Currently, except for the blockade of the renin-angiotensin-aldosterone system, no other therapies are …
IgAN is characterized by dysregulation of the immune system, which causes an abnormal synthesis of IgA1 that is deglycosylated causin …
Vasoactive substances in the circulatory dysfunction of cirrhosis.
Møller S, Bendtsen F, Henriksen JH. Møller S, et al. Scand J Clin Lab Invest. 2001;61(6):421-9. doi: 10.1080/00365510152567059. Scand J Clin Lab Invest. 2001. PMID: 11681531 Review.
Patients with cirrhosis and portal hypertension exhibit characteristic haemodynamic changes with a hyperkinetic systemic circulation, abnormal distribution of the blood volume, and neurohumoral dysregulation. ...Reflex-induced enhanced sympatho-adrenal activi …
Patients with cirrhosis and portal hypertension exhibit characteristic haemodynamic changes with a hyperkinetic systemic circulation, …
Fluid homeostasis in chronic obstructive lung disease.
de Leeuw PW, Dees A. de Leeuw PW, et al. Eur Respir J Suppl. 2003 Nov;46:33s-40s. doi: 10.1183/09031936.03.00000603a. Eur Respir J Suppl. 2003. PMID: 14621105 Free article. Review.
The mechanisms by which patients with COPD retain salt and water are not completely understood. Several abnormalities have been found including reduced renal blood flow with relatively preserved glomerular filtration rate and elevated levels of renin, aldosterone …
The mechanisms by which patients with COPD retain salt and water are not completely understood. Several abnormalities have been found …
Splanchnic and systemic hemodynamic derangement in decompensated cirrhosis.
Møller S, Bendtsen F, Henriksen JH. Møller S, et al. Can J Gastroenterol. 2001 Feb;15(2):94-106. doi: 10.1155/2001/603012. Can J Gastroenterol. 2001. PMID: 11240379 Review.
Patients with cirrhosis and portal hypertension exhibit characteristic hemodynamic changes with hyperkinetic systemic circulation, abnormal distribution of blood volume and neurohumoral dysregulation. Their plasma and noncentral blood volumes are incre …
Patients with cirrhosis and portal hypertension exhibit characteristic hemodynamic changes with hyperkinetic systemic circulation, …
Circulatory abnormalities in cirrhosis with focus on neurohumoral aspects.
Møller S, Henriksen JH. Møller S, et al. Semin Nephrol. 1997 Nov;17(6):505-19. Semin Nephrol. 1997. PMID: 9353862 Review.
Patients with cirrhosis exhibit characteristic hemodynamic changes with a hyperkinetic circulation and an abnormal distribution of the blood volume and neurohumoral regulation. ...Recent research has focused on the assertion that the hemodynamic and neurohumo …
Patients with cirrhosis exhibit characteristic hemodynamic changes with a hyperkinetic circulation and an abnormal distributio …
Urinary renin-angiotensin markers in polycystic kidney disease.
Salih M, Bovée DM, Roksnoer LCW, Casteleijn NF, Bakker SJL, Gansevoort RT, Zietse R, Danser AHJ, Hoorn EJ. Salih M, et al. Am J Physiol Renal Physiol. 2017 Oct 1;313(4):F874-F881. doi: 10.1152/ajprenal.00209.2017. Epub 2017 Jul 26. Am J Physiol Renal Physiol. 2017. PMID: 28747358 Free article.
In autosomal dominant polycystic kidney disease (ADPKD), activation of the renin-angiotensin aldosterone system (RAAS) may contribute to hypertension and disease progression. Although previous studies have focused on circulating RAAS components, preliminary evidence …
In autosomal dominant polycystic kidney disease (ADPKD), activation of the renin-angiotensin aldosterone system (RAAS) may contribute …
Diabetes mellitus and heart failure.
Sander GE, Giles TD. Sander GE, et al. Am Heart Hosp J. 2003 Fall;1(4):273-80. doi: 10.1111/j.1541-9215.2003.02085.x. Am Heart Hosp J. 2003. PMID: 15815121 Review.
Hyperglycemia, insulin resistance, and hypertension, together with activation of both the circulating and the tissue renin-angiotensin-aldosterone systems, contribute to structural fibrosis and autonomic neuropathy. Thus, it becomes imperative to identify cardiac …
Hyperglycemia, insulin resistance, and hypertension, together with activation of both the circulating and the tissue renin-angiotensi …
Congestive heart failure--pathophysiology and medical treatment.
Remme WJ. Remme WJ. J Cardiovasc Pharmacol. 1986;8 Suppl 1:S36-52. J Cardiovasc Pharmacol. 1986. PMID: 2422492 Review.
With ongoing failure, myocardial muscle function further deteriorates because of a disturbance in myocardial energy utilization, abnormalities in sympathetic neurotransmitter metabolism, a reduction in beta-receptor density, and, more importantly, a derangement of intracel …
With ongoing failure, myocardial muscle function further deteriorates because of a disturbance in myocardial energy utilization, abnormal …
36 results