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Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1957 1
1970 1
1971 1
1972 1
1973 2
1974 1
1975 2
1976 5
1977 5
1978 2
1979 3
1980 6
1981 7
1982 5
1983 5
1984 6
1985 9
1986 3
1987 9
1988 4
1989 8
1990 9
1991 1
1992 5
1993 7
1994 6
1995 10
1996 13
1997 10
1998 13
1999 12
2000 19
2001 4
2002 10
2003 13
2004 12
2005 15
2006 21
2007 14
2008 11
2009 12
2010 17
2011 17
2012 25
2013 15
2014 20
2015 17
2016 13
2017 13
2018 15
2019 12
2020 23
2021 13
2022 11
2023 6
2024 5

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486 results

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Quoted phrase not found in phrase index: "Abnormal nephron morphology"
Page 1
Podocytopathies.
Kopp JB, Anders HJ, Susztak K, Podestà MA, Remuzzi G, Hildebrandt F, Romagnani P. Kopp JB, et al. Nat Rev Dis Primers. 2020 Aug 13;6(1):68. doi: 10.1038/s41572-020-0196-7. Nat Rev Dis Primers. 2020. PMID: 32792490 Free PMC article. Review.
In children and young adults, genetic variants in >50 podocyte-expressed genes, syndromal non-podocyte-specific genes and phenocopies with other underlying genetic abnormalities cause podocytopathies associated with steroid-resistant nephrotic syndrome or severe protein …
In children and young adults, genetic variants in >50 podocyte-expressed genes, syndromal non-podocyte-specific genes and phenocopies wit …
IgA Nephropathy: Core Curriculum 2021.
Pattrapornpisut P, Avila-Casado C, Reich HN. Pattrapornpisut P, et al. Am J Kidney Dis. 2021 Sep;78(3):429-441. doi: 10.1053/j.ajkd.2021.01.024. Epub 2021 Jul 9. Am J Kidney Dis. 2021. PMID: 34247883 Review.
Given substantial heterogeneity in the clinical course of disease, online risk calculators are available that may assist in prognostication and inform discussions with patients. ...In this installment of the AJKD Core Curriculum in Nephrology, IgAN pathogenesis, cli …
Given substantial heterogeneity in the clinical course of disease, online risk calculators are available that may assist in progno …
A Primer on Congenital Anomalies of the Kidneys and Urinary Tracts (CAKUT).
Murugapoopathy V, Gupta IR. Murugapoopathy V, et al. Clin J Am Soc Nephrol. 2020 May 7;15(5):723-731. doi: 10.2215/CJN.12581019. Epub 2020 Mar 18. Clin J Am Soc Nephrol. 2020. PMID: 32188635 Free PMC article. Review.
Congenital anomalies of the kidneys and urinary tracts (CAKUT) are disorders caused by defects in the development of the kidneys and their outflow tracts. ...Low nephron number appears to arise by the same mechanisms as CAKUT, but it differs in terms of the magnitud …
Congenital anomalies of the kidneys and urinary tracts (CAKUT) are disorders caused by defects in the development of the kidneys and …
KIM-1 mediates fatty acid uptake by renal tubular cells to promote progressive diabetic kidney disease.
Mori Y, Ajay AK, Chang JH, Mou S, Zhao H, Kishi S, Li J, Brooks CR, Xiao S, Woo HM, Sabbisetti VS, Palmer SC, Galichon P, Li L, Henderson JM, Kuchroo VK, Hawkins J, Ichimura T, Bonventre JV. Mori Y, et al. Cell Metab. 2021 May 4;33(5):1042-1061.e7. doi: 10.1016/j.cmet.2021.04.004. Cell Metab. 2021. PMID: 33951465 Free PMC article.
Tubulointerstitial abnormalities are predictive of the progression of diabetic kidney disease (DKD), and their targeting may be an effective means for prevention. Proximal tubular (PT) expression of kidney injury molecule (KIM)-1, as well as blood and urinary levels …
Tubulointerstitial abnormalities are predictive of the progression of diabetic kidney disease (DKD), and their targeting may b …
Pathophysiology of proteinuria.
D'Amico G, Bazzi C. D'Amico G, et al. Kidney Int. 2003 Mar;63(3):809-25. doi: 10.1046/j.1523-1755.2003.00840.x. Kidney Int. 2003. PMID: 12631062 Free article. Review.
Proteinuria is consequence of two mechanisms: the abnormal transglomerular passage of proteins due to increased permeability of glomerular capillary wall and their subsequent impaired reabsorption by the epithelial cells of the proximal tubuli. ...Recent clinical studies s …
Proteinuria is consequence of two mechanisms: the abnormal transglomerular passage of proteins due to increased permeability of glome …
Autosomal dominant polycystic kidney disease.
Torres VE, Harris PC, Pirson Y. Torres VE, et al. Lancet. 2007 Apr 14;369(9569):1287-1301. doi: 10.1016/S0140-6736(07)60601-1. Lancet. 2007. PMID: 17434405 Review.
Membranoproliferative glomerulonephritis: no longer the same disease and may need very different treatment.
Noris M, Daina E, Remuzzi G. Noris M, et al. Nephrol Dial Transplant. 2023 Feb 13;38(2):283-290. doi: 10.1093/ndt/gfab281. Nephrol Dial Transplant. 2023. PMID: 34596686
However, this classification leaves a number of issues unresolved. The finding of genetic and acquired complement abnormalities in both C3G and IC-MPGN indicates that they represent a heterogeneous spectrum rather than distinct diseases. ...This creates the need to charact …
However, this classification leaves a number of issues unresolved. The finding of genetic and acquired complement abnormalities in bo …
Update on C3 Glomerulopathy: A Complement-Mediated Disease.
Caravaca-Fontán F, Lucientes L, Cavero T, Praga M. Caravaca-Fontán F, et al. Nephron. 2020;144(6):272-280. doi: 10.1159/000507254. Epub 2020 May 5. Nephron. 2020. PMID: 32369815 Free article. Review.
Thus, a new pathogenic classification based on a cluster analysis of clinical, histologic, and genetic data has recently been proposed, which could also help identify patients at higher risk of progression. Several pathogenic abnormalities in complement genes have been des …
Thus, a new pathogenic classification based on a cluster analysis of clinical, histologic, and genetic data has recently been proposed, whic …
A review of clinical characteristics and genetic backgrounds in Alport syndrome.
Nozu K, Nakanishi K, Abe Y, Udagawa T, Okada S, Okamoto T, Kaito H, Kanemoto K, Kobayashi A, Tanaka E, Tanaka K, Hama T, Fujimaru R, Miwa S, Yamamura T, Yamamura N, Horinouchi T, Minamikawa S, Nagata M, Iijima K. Nozu K, et al. Clin Exp Nephrol. 2019 Feb;23(2):158-168. doi: 10.1007/s10157-018-1629-4. Epub 2018 Aug 20. Clin Exp Nephrol. 2019. PMID: 30128941 Free PMC article. Review.
Alport syndrome (AS) is a progressive hereditary renal disease that is characterized by sensorineural hearing loss and ocular abnormalities. It is divided into three modes of inheritance, namely, X-linked Alport syndrome (XLAS), autosomal recessive AS (ARAS), and autosomal …
Alport syndrome (AS) is a progressive hereditary renal disease that is characterized by sensorineural hearing loss and ocular abnormaliti …
Nail-patella syndrome.
Witzgall R. Witzgall R. Pflugers Arch. 2017 Aug;469(7-8):927-936. doi: 10.1007/s00424-017-2013-z. Epub 2017 Jul 5. Pflugers Arch. 2017. PMID: 28681095 Review.
Many of the patients suffer from renal symptoms which also affect their prognosis. In 1998, mutations in the gene encoding the transcription factor LMX1B were identified as underlying this autosomal-dominant disease. ...
Many of the patients suffer from renal symptoms which also affect their prognosis. In 1998, mutations in the gene encoding the transc …
486 results