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Quoted phrase not found in phrase index: "Abnormal platelet phosphatidylserine exposure"
Page 1
Beta-thalassaemia and sickle cell anaemia as paradigms of hypercoagulability.
Ataga KI, Cappellini MD, Rachmilewitz EA. Ataga KI, et al. Br J Haematol. 2007 Oct;139(1):3-13. doi: 10.1111/j.1365-2141.2007.06740.x. Br J Haematol. 2007. PMID: 17854302 Free article. Review.
The abnormal phospholipid membrane assymetry present in the red blood cells of beta-thalassaemia and SCA patients, with resultant phosphatidylserine exposure appears to play a significant role in the aetiology of the observed hypercoagulable state. Thi …
The abnormal phospholipid membrane assymetry present in the red blood cells of beta-thalassaemia and SCA patients, with result …
Procoagulant activity induced by transcatheter closure of atrial septal defects is associated with exposure of phosphatidylserine on microparticles, platelets and red blood cells.
Ding W, Kou J, Meng H, Kou Y, He Z, Cao M, Wang L, Bi Y, Thatte HS, Shi J. Ding W, et al. Thromb Res. 2015 Aug;136(2):354-60. doi: 10.1016/j.thromres.2015.06.015. Epub 2015 Jun 12. Thromb Res. 2015. PMID: 26099643 Clinical Trial.
The mechanism of hypercoagulable state following transcatheter closure of atrial septal defects (ASDs) remains unclear. We evaluated the exposure of phosphatidylserine (PS) on released microparticles (MPs) and also the cells of their origin from peripheral blood …
The mechanism of hypercoagulable state following transcatheter closure of atrial septal defects (ASDs) remains unclear. We evaluated the …
Reversibility of severe metabolic stress in stored platelets after in vitro plasma rescue or in vivo transfusion: restoration of secretory function and maintenance of platelet survival.
Rinder HM, Snyder EL, Tracey JB, Dincecco D, Wang C, Baril L, Rinder CS, Smith BR. Rinder HM, et al. Transfusion. 2003 Sep;43(9):1230-7. doi: 10.1046/j.1537-2995.2003.00484.x. Transfusion. 2003. PMID: 12919425 Clinical Trial.
Whole-blood-derived PCs were transfused into a small number of normal volunteers to determine platelet recovery and survival. ...CONCLUSION: Transient derangement of platelet metabolism, which does not increase membrane phosphatidylserine exposure …
Whole-blood-derived PCs were transfused into a small number of normal volunteers to determine platelet recovery and survival. …