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174 results

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Quoted phrase not found in phrase index: "Abnormal renal tubule morphology"
Page 1
KIM-1 mediates fatty acid uptake by renal tubular cells to promote progressive diabetic kidney disease.
Mori Y, Ajay AK, Chang JH, Mou S, Zhao H, Kishi S, Li J, Brooks CR, Xiao S, Woo HM, Sabbisetti VS, Palmer SC, Galichon P, Li L, Henderson JM, Kuchroo VK, Hawkins J, Ichimura T, Bonventre JV. Mori Y, et al. Cell Metab. 2021 May 4;33(5):1042-1061.e7. doi: 10.1016/j.cmet.2021.04.004. Cell Metab. 2021. PMID: 33951465 Free PMC article.
Tubulointerstitial abnormalities are predictive of the progression of diabetic kidney disease (DKD), and their targeting may be an effective means for prevention. Proximal tubular (PT) expression of kidney injury molecule (KIM)-1, as well as blood and …
Tubulointerstitial abnormalities are predictive of the progression of diabetic kidney disease (DKD), and their targetin …
Autosomal dominant polycystic kidney disease.
Torres VE, Harris PC, Pirson Y. Torres VE, et al. Lancet. 2007 Apr 14;369(9569):1287-1301. doi: 10.1016/S0140-6736(07)60601-1. Lancet. 2007. PMID: 17434405 Review.
Autosomal dominant polycystic kidney disease is the most prevalent, potentially lethal, monogenic disorder. ...
Autosomal dominant polycystic kidney disease is the most prevalent, potentially lethal, monogenic disorder. ...
More advantages in detecting bone and soft tissue metastases from prostate cancer using 18F-PSMA PET/CT.
Pianou NK, Stavrou PZ, Vlontzou E, Rondogianni P, Exarhos DN, Datseris IE. Pianou NK, et al. Hell J Nucl Med. 2019 Jan-Apr;22(1):6-9. doi: 10.1967/s002449910952. Epub 2019 Mar 7. Hell J Nucl Med. 2019. PMID: 30843003 Free article.
Sensitivity and specificity however, perform better (96% and 94% respectively) when SPET and SPET/CT techniques are applied. Of course, bone marrow metastases cannot be detected in a WBS. The PSA marker is used to predict the pre-test probability of BM and in case o …
Sensitivity and specificity however, perform better (96% and 94% respectively) when SPET and SPET/CT techniques are applied. Of course
Molecular genetics of renal ciliopathies.
Barroso-Gil M, Olinger E, Sayer JA. Barroso-Gil M, et al. Biochem Soc Trans. 2021 Jun 30;49(3):1205-1220. doi: 10.1042/BST20200791. Biochem Soc Trans. 2021. PMID: 33960378 Review.
Renal ciliopathies are a heterogenous group of inherited disorders leading to an array of phenotypes that include cystic kidney disease and renal interstitial fibrosis leading to progressive chronic kidney disease and end-stage kidney disease. .
Renal ciliopathies are a heterogenous group of inherited disorders leading to an array of phenotypes that include cystic kidney
Genetics and CKD.
Vehaskari VM. Vehaskari VM. Adv Chronic Kidney Dis. 2011 Sep;18(5):317-23. doi: 10.1053/j.ackd.2011.07.001. Adv Chronic Kidney Dis. 2011. PMID: 21896372 Review.
The diagnosis of hereditary monogenic kidney diseases is frequently delayed, in part because of physicians' unfamiliarity with the relatively rare conditions or because of the late onset of symptoms in some patients. ...Accurate diagnosis is important for appropriate treat …
The diagnosis of hereditary monogenic kidney diseases is frequently delayed, in part because of physicians' unfamiliarity with the re …
Mechanisms of compensatory renal growth.
Cleper R. Cleper R. Pediatr Endocrinol Rev. 2012 Nov;10(1):152-63. Pediatr Endocrinol Rev. 2012. PMID: 23469392 Review.
Congenitally reduced renal mass- as with agenesis of one kidney, unilateral multicystic dysplastic kidney or with premature birth with early arrest of nephrogenesis- as well as acquired loss of a significant part of kidney tissue- as with kidney
Congenitally reduced renal mass- as with agenesis of one kidney, unilateral multicystic dysplastic kidney or with prema …
Renal oncocytoma.
Lieber MM. Lieber MM. Urol Clin North Am. 1993 May;20(2):355-9. Urol Clin North Am. 1993. PMID: 8493754 Review.
Renal oncocytomas are believed to arise from the intercalated cells of the renal collecting tubules. Specific chromosomal abnormalities, which differentiate oncocytomas from renal cell carcinomas, have been found. ...
Renal oncocytomas are believed to arise from the intercalated cells of the renal collecting tubules. Specific chromosom
Disease modeling in genetic kidney diseases: mice.
Hofmeister AF, Kömhoff M, Weber S, Grgic I. Hofmeister AF, et al. Cell Tissue Res. 2017 Jul;369(1):159-170. doi: 10.1007/s00441-017-2639-3. Epub 2017 Jun 10. Cell Tissue Res. 2017. PMID: 28601904 Review.
Moreover, knockout and transgenic mouse models can be highly informative with respect to the effects of genetic variations on renal phenotypes. This review focuses on mouse models that have been devised primarily to study monogenic human kidney diseases, which are t …
Moreover, knockout and transgenic mouse models can be highly informative with respect to the effects of genetic variations on renal p …
Endothelin in polycystic kidney disease.
Chang MY, Ong ACM. Chang MY, et al. Contrib Nephrol. 2011;172:200-209. doi: 10.1159/000328701. Epub 2011 Aug 30. Contrib Nephrol. 2011. PMID: 21894000 Review.
Autosomal dominant polycystic kidney disease (ADPKD) is the most common genetic kidney disease in man and is caused by germline mutations in PKD1 or PKD2. ...Current evidence suggests that ET-1 and its receptors act as major modifying genes for renal disease …
Autosomal dominant polycystic kidney disease (ADPKD) is the most common genetic kidney disease in man and is caused by germlin …
Proximal tubular injury in myeloma.
Batuman V. Batuman V. Contrib Nephrol. 2007;153:87-104. doi: 10.1159/000096762. Contrib Nephrol. 2007. PMID: 17075225 Review.
Renal involvement is common in multiple myeloma and implies much worse prognosis. ...In early stages of myeloma, light chain nephrotoxicity often presents with proximal tubular functional abnormalities, such as Fanconi syndrome. These proximal tubule a
Renal involvement is common in multiple myeloma and implies much worse prognosis. ...In early stages of myeloma, light chain n
174 results