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Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1959 1
1966 1
1971 1
1972 2
1974 2
1975 7
1976 5
1977 4
1978 2
1979 3
1980 3
1981 3
1982 3
1983 3
1984 5
1985 9
1986 9
1987 6
1988 3
1989 3
1990 9
1991 6
1992 8
1993 7
1994 8
1995 6
1996 6
1997 15
1998 8
1999 12
2000 12
2001 11
2002 8
2003 12
2004 17
2005 23
2006 14
2007 16
2008 20
2009 19
2010 25
2011 33
2012 28
2013 31
2014 28
2015 29
2016 17
2017 29
2018 27
2019 32
2020 27
2021 23
2022 11
2023 18
2024 17

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605 results

Results by year

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Quoted phrase not found in phrase index: "Abnormality of the urinary system physiology"
Page 1
IgA Nephropathy: Core Curriculum 2021.
Pattrapornpisut P, Avila-Casado C, Reich HN. Pattrapornpisut P, et al. Am J Kidney Dis. 2021 Sep;78(3):429-441. doi: 10.1053/j.ajkd.2021.01.024. Epub 2021 Jul 9. Am J Kidney Dis. 2021. PMID: 34247883 Review.
Given substantial heterogeneity in the clinical course of disease, online risk calculators are available that may assist in prognostication and inform discussions with patients. ...Recent advances in our understanding of mucosal immunity and the role of the compleme …
Given substantial heterogeneity in the clinical course of disease, online risk calculators are available that may assist in progno
Podocytopathies.
Kopp JB, Anders HJ, Susztak K, Podestà MA, Remuzzi G, Hildebrandt F, Romagnani P. Kopp JB, et al. Nat Rev Dis Primers. 2020 Aug 13;6(1):68. doi: 10.1038/s41572-020-0196-7. Nat Rev Dis Primers. 2020. PMID: 32792490 Free PMC article. Review.
In children and young adults, genetic variants in >50 podocyte-expressed genes, syndromal non-podocyte-specific genes and phenocopies with other underlying genetic abnormalities cause podocytopathies associated with steroid-resistant nephrotic syndrome or severe protein …
In children and young adults, genetic variants in >50 podocyte-expressed genes, syndromal non-podocyte-specific genes and phenocopies wit …
The prevalence of congenital anomalies in Europe.
Dolk H, Loane M, Garne E. Dolk H, et al. Adv Exp Med Biol. 2010;686:349-64. doi: 10.1007/978-90-481-9485-8_20. Adv Exp Med Biol. 2010. PMID: 20824455 Review.
EUROCAT (European Surveillance of Congenital Anomalies) is the network of population-based registers of congenital anomaly in Europe, with a common protocol and data quality review, covering 1.5 million annual births in 22 countries. ...The prevalence of chromosomal …
EUROCAT (European Surveillance of Congenital Anomalies) is the network of population-based registers of congenital anomaly in …
Combined Angiotensin Receptor Antagonism and Neprilysin Inhibition.
Hubers SA, Brown NJ. Hubers SA, et al. Circulation. 2016 Mar 15;133(11):1115-24. doi: 10.1161/CIRCULATIONAHA.115.018622. Circulation. 2016. PMID: 26976916 Free PMC article. Review.
Thus, this combined angiotensin receptor antagonist and neprilysin inhibitor addresses 2 of the pathophysiological mechanisms of heart failure: activation of the renin-angiotensin-aldosterone system and decreased sensitivity to natriuretic peptides. In the Prospective Comp …
Thus, this combined angiotensin receptor antagonist and neprilysin inhibitor addresses 2 of the pathophysiological mechanisms of heart failu …
AKI in the ICU: definition, epidemiology, risk stratification, and outcomes.
Singbartl K, Kellum JA. Singbartl K, et al. Kidney Int. 2012 May;81(9):819-25. doi: 10.1038/ki.2011.339. Epub 2011 Oct 5. Kidney Int. 2012. PMID: 21975865 Free article. Review.
The detrimental effects of AKI are not limited to classical well-known symptoms such as fluid overload and electrolyte abnormalities. AKI can also lead to problems that are not readily appreciated at the bedside and can extend well beyond the ICU stay, including progressio …
The detrimental effects of AKI are not limited to classical well-known symptoms such as fluid overload and electrolyte abnormalities. …
Alport Syndrome: A Comprehensive Review on Genetics, Pathophysiology, Histology, Clinical and Therapeutic Perspectives.
Pedrosa AL, Bitencourt L, Paranhos RM, Leitáo CA, Ferreira GC, Simões E Silva AC. Pedrosa AL, et al. Curr Med Chem. 2021;28(27):5602-5624. doi: 10.2174/0929867328666210108113500. Curr Med Chem. 2021. PMID: 33423643 Review.
We discuss the genetic and pathophysiology of AS, clinical manifestations, histopathology, diagnostic protocols, conventional treatment and prognostic markers of the disease. In addition, we summarize experimental findings with novel therapeutic perspectives for AS. ...As …
We discuss the genetic and pathophysiology of AS, clinical manifestations, histopathology, diagnostic protocols, conventional treatment and …
Nail-patella syndrome.
Witzgall R. Witzgall R. Pflugers Arch. 2017 Aug;469(7-8):927-936. doi: 10.1007/s00424-017-2013-z. Epub 2017 Jul 5. Pflugers Arch. 2017. PMID: 28681095 Review.
Many of the patients suffer from renal symptoms which also affect their prognosis. In 1998, mutations in the gene encoding the transcription factor LMX1B were identified as underlying this autosomal-dominant disease. ...LMX1B is essential for dorso-ventral pattern formatio …
Many of the patients suffer from renal symptoms which also affect their prognosis. In 1998, mutations in the gene encoding the transc …
UTI in kidney transplant.
Ness D, Olsburgh J. Ness D, et al. World J Urol. 2020 Jan;38(1):81-88. doi: 10.1007/s00345-019-02742-6. Epub 2019 Apr 1. World J Urol. 2020. PMID: 30937570 Review.
Urinary tract infection (UTI) remains the most common type of infection contracted by kidney transplant patients. ...Understanding and managing UTI in transplant patients requires an appreciation of their unique anatomy and physiology. Both the transplant and
Urinary tract infection (UTI) remains the most common type of infection contracted by kidney transplant patients. ...Understan
High-Density Lipoproteins and the Kidney.
Strazzella A, Ossoli A, Calabresi L. Strazzella A, et al. Cells. 2021 Mar 31;10(4):764. doi: 10.3390/cells10040764. Cells. 2021. PMID: 33807271 Free PMC article. Review.
Genetic LCAT deficiency is the most emblematic case and represents a unique tool to evaluate the impact of alterations in the HDL system on the progression of renal disease. Lipid abnormalities detected in LCAT-deficient carriers mirror the ones observed in CKD pati …
Genetic LCAT deficiency is the most emblematic case and represents a unique tool to evaluate the impact of alterations in the HDL system
Molecular genetics of renal ciliopathies.
Barroso-Gil M, Olinger E, Sayer JA. Barroso-Gil M, et al. Biochem Soc Trans. 2021 Jun 30;49(3):1205-1220. doi: 10.1042/BST20200791. Biochem Soc Trans. 2021. PMID: 33960378 Review.
Mutations in genes encoding ciliary proteins involved in the structure and function of primary cilia cause ciliopathy syndromes and affect many organ systems including the kidney. Recognised disease phenotypes associated with primary ciliopathies that have a strong renal c …
Mutations in genes encoding ciliary proteins involved in the structure and function of primary cilia cause ciliopathy syndromes and affect m …
605 results