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Quoted phrase not found in phrase index: "Alzheimer disease without neurofibrillary tangles"
Page 1
Alzheimer disease.
Knopman DS, Amieva H, Petersen RC, Chételat G, Holtzman DM, Hyman BT, Nixon RA, Jones DT. Knopman DS, et al. Nat Rev Dis Primers. 2021 May 13;7(1):33. doi: 10.1038/s41572-021-00269-y. Nat Rev Dis Primers. 2021. PMID: 33986301 Free PMC article. Review.
Alzheimer disease (AD) is biologically defined by the presence of beta-amyloid-containing plaques and tau-containing neurofibrillary tangles. ...Therapeutic endeavours are still struggling to find targets within this framework that substantially change
Alzheimer disease (AD) is biologically defined by the presence of beta-amyloid-containing plaques and tau-containing neurof
Alzheimer's disease: risk factors and potentially protective measures.
Silva MVF, Loures CMG, Alves LCV, de Souza LC, Borges KBG, Carvalho MDG. Silva MVF, et al. J Biomed Sci. 2019 May 9;26(1):33. doi: 10.1186/s12929-019-0524-y. J Biomed Sci. 2019. PMID: 31072403 Free PMC article. Review.
Alzheimer's disease (AD) is the most common type of dementia and typically manifests through a progressive loss of episodic memory and cognitive function, subsequently causing language and visuospatial skills deficiencies, which are often accompanied by behavioral d
Alzheimer's disease (AD) is the most common type of dementia and typically manifests through a progressive loss of episodic me
Aging and Alzheimer's disease pathology.
Sengoku R. Sengoku R. Neuropathology. 2020 Feb;40(1):22-29. doi: 10.1111/neup.12626. Epub 2019 Dec 21. Neuropathology. 2020. PMID: 31863504 Review.
The number of people with dementia worldwide is predicted to increase to 131.5 million by 2050. When studying dementia, understanding the basis of the neuropathological background is very important. Taking Alzheimer's disease (AD) neuropathology as an example …
The number of people with dementia worldwide is predicted to increase to 131.5 million by 2050. When studying dementia, understanding …
Neuropathology of Alzheimer's Disease.
Trejo-Lopez JA, Yachnis AT, Prokop S. Trejo-Lopez JA, et al. Neurotherapeutics. 2022 Jan;19(1):173-185. doi: 10.1007/s13311-021-01146-y. Epub 2021 Nov 2. Neurotherapeutics. 2022. PMID: 34729690 Free PMC article. Review.
The key pathological hallmarks-extracellular plaques and intracellular neurofibrillary tangles (NFT)-described by Alois Alzheimer in his seminal 1907 article are still central to the postmortem diagnosis of Alzheimer's disease (AD), but major ad …
The key pathological hallmarks-extracellular plaques and intracellular neurofibrillary tangles (NFT)-described by Alois Alz
Molecular biomarkers of Alzheimer's disease: progress and prospects.
Lashley T, Schott JM, Weston P, Murray CE, Wellington H, Keshavan A, Foti SC, Foiani M, Toombs J, Rohrer JD, Heslegrave A, Zetterberg H. Lashley T, et al. Dis Model Mech. 2018 May 8;11(5):dmm031781. doi: 10.1242/dmm.031781. Dis Model Mech. 2018. PMID: 29739861 Free PMC article. Review.
The neurodegenerative disorder Alzheimer's disease is characterised by the formation of beta-amyloid plaques and neurofibrillary tangles in the brain parenchyma, which cause synapse and neuronal loss. ...In this 'At a glance' article and poster, we sum …
The neurodegenerative disorder Alzheimer's disease is characterised by the formation of beta-amyloid plaques and neurofibri
SPECT and PET imaging in Alzheimer's disease.
Valotassiou V, Malamitsi J, Papatriantafyllou J, Dardiotis E, Tsougos I, Psimadas D, Alexiou S, Hadjigeorgiou G, Georgoulias P. Valotassiou V, et al. Ann Nucl Med. 2018 Nov;32(9):583-593. doi: 10.1007/s12149-018-1292-6. Epub 2018 Aug 20. Ann Nucl Med. 2018. PMID: 30128693 Review.
Alzheimer's disease (AD) is a progressive neurodegenerative disorder and the most common cause of dementia. Beta-amyloid (Abeta) deposition and neurofibrillary tangles (NFTs) of abnormal hyperphosphorylated tau protein are the pathological hallmarks of
Alzheimer's disease (AD) is a progressive neurodegenerative disorder and the most common cause of dementia. Beta-amyloid (Abet
Genetics of familial and sporadic Alzheimer's disease.
Piaceri I, Nacmias B, Sorbi S. Piaceri I, et al. Front Biosci (Elite Ed). 2013 Jan 1;5(1):167-77. doi: 10.2741/e605. Front Biosci (Elite Ed). 2013. PMID: 23276979 Free article. Review.
Alzheimer's disease (AD) is an age-related progressive neurodegenerative disorder. ...Genome-wide association studies (GWASs) revealed the genetic complexity of the disease and recent studies suggested that epigenetic mechanisms may play an essential role in
Alzheimer's disease (AD) is an age-related progressive neurodegenerative disorder. ...Genome-wide association studies (GWASs)
Molecular Basis of Familial and Sporadic Alzheimer's Disease.
Dorszewska J, Prendecki M, Oczkowska A, Dezor M, Kozubski W. Dorszewska J, et al. Curr Alzheimer Res. 2016;13(9):952-63. doi: 10.2174/1567205013666160314150501. Curr Alzheimer Res. 2016. PMID: 26971934 Review.
Alzheimer's disease (AD) is a multifactorial disease with genetic (70%) and environmental (30%) causes. ...It seems that a complete understanding of both FAD and SAD pathogenesis may contribute to the search for earlier clinical diagnosis and to an understand
Alzheimer's disease (AD) is a multifactorial disease with genetic (70%) and environmental (30%) causes. ...It seems tha
Prognosis and Neuropathologic Correlation of Clinical Subtypes of Parkinson Disease.
De Pablo-Fernández E, Lees AJ, Holton JL, Warner TT. De Pablo-Fernández E, et al. JAMA Neurol. 2019 Apr 1;76(4):470-479. doi: 10.1001/jamaneurol.2018.4377. JAMA Neurol. 2019. PMID: 30640364 Free PMC article.
IMPORTANCE: Clinical subtyping of Parkinson disease at diagnosis is useful in estimating disease course and survival. ...Severity and distribution of Lewy pathology and Alzheimer disease-related pathology were assessed using staging systems. ...
IMPORTANCE: Clinical subtyping of Parkinson disease at diagnosis is useful in estimating disease course and survival. . …
The spectrum of disease in chronic traumatic encephalopathy.
McKee AC, Stern RA, Nowinski CJ, Stein TD, Alvarez VE, Daneshvar DH, Lee HS, Wojtowicz SM, Hall G, Baugh CM, Riley DO, Kubilus CA, Cormier KA, Jacobs MA, Martin BR, Abraham CR, Ikezu T, Reichard RR, Wolozin BL, Budson AE, Goldstein LE, Kowall NW, Cantu RC. McKee AC, et al. Brain. 2013 Jan;136(Pt 1):43-64. doi: 10.1093/brain/aws307. Epub 2012 Dec 2. Brain. 2013. PMID: 23208308 Free PMC article.
In chronic traumatic encephalopathy, the spectrum of hyperphosphorylated tau pathology ranged in severity from focal perivascular epicentres of neurofibrillary tangles in the frontal neocortex to severe tauopathy affecting widespread brain regions, including the med …
In chronic traumatic encephalopathy, the spectrum of hyperphosphorylated tau pathology ranged in severity from focal perivascular epicentres …
766 results