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Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1977 1
1978 1
1982 2
1983 3
1984 2
1985 1
1986 3
1987 2
1988 2
1989 1
1990 5
1991 4
1992 7
1993 6
1994 8
1995 13
1996 7
1997 7
1998 13
1999 15
2000 9
2001 19
2002 18
2003 19
2004 19
2005 15
2006 23
2007 17
2008 21
2009 29
2010 29
2011 37
2012 33
2013 48
2014 55
2015 68
2016 50
2017 54
2018 59
2019 64
2020 60
2021 52
2022 49
2023 50
2024 21

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895 results

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Quoted phrase not found in phrase index: "Astrocytoma, increased risk of"
Page 1
Molecular targeted therapy of glioblastoma.
Le Rhun E, Preusser M, Roth P, Reardon DA, van den Bent M, Wen P, Reifenberger G, Weller M. Le Rhun E, et al. Cancer Treat Rev. 2019 Nov;80:101896. doi: 10.1016/j.ctrv.2019.101896. Epub 2019 Sep 11. Cancer Treat Rev. 2019. PMID: 31541850 Free article. Review.
More than 90% of glioblastomas are isocitrate dehydrogenase (IDH)-wildtype tumors. Incidence increases with age, males are more often affected. Beyond rare instances of genetic predisposition and irradiation exposure, there are no known glioblastoma risk factors. .. …
More than 90% of glioblastomas are isocitrate dehydrogenase (IDH)-wildtype tumors. Incidence increases with age, males are more often …
Multifunctional mRNA-Based CAR T Cells Display Promising Antitumor Activity Against Glioblastoma.
Meister H, Look T, Roth P, Pascolo S, Sahin U, Lee S, Hale BD, Snijder B, Regli L, Ravi VM, Heiland DH, Sentman CL, Weller M, Weiss T. Meister H, et al. Clin Cancer Res. 2022 Nov 1;28(21):4747-4756. doi: 10.1158/1078-0432.CCR-21-4384. Clin Cancer Res. 2022. PMID: 36037304
PURPOSE: Most chimeric antigen receptor (CAR) T-cell strategies against glioblastoma have demonstrated only modest therapeutic activity and are based on persistent gene modification strategies that have limited transgene capacity, long manufacturing processes, and the risk …
PURPOSE: Most chimeric antigen receptor (CAR) T-cell strategies against glioblastoma have demonstrated only modest therapeutic activity and …
Benign Glioma.
Wu PB, Filley AC, Miller ML, Bruce JN. Wu PB, et al. Adv Exp Med Biol. 2023;1405:31-71. doi: 10.1007/978-3-031-23705-8_2. Adv Exp Med Biol. 2023. PMID: 37452934
The first group is of focal, well-circumscribed lesions that notably are not associated with an increased risk of malignant transformation. Primarily diagnosed in pediatric patients, these WHO grade I tumors may be cured with surgical resection alone (Sturm e …
The first group is of focal, well-circumscribed lesions that notably are not associated with an increased risk of malig …
Encephalocraniocutaneous Lipomatosis.
Bavle A, Shah R, Gross N, Gavula T, Ruiz-Elizalde A, Wierenga K, McNall-Knapp R. Bavle A, et al. J Pediatr Hematol Oncol. 2018 Oct;40(7):553-554. doi: 10.1097/MPH.0000000000001170. J Pediatr Hematol Oncol. 2018. PMID: 29683947
Magnetic resonance imaging revealed a large suprasellar mass, which was debulked and found to be a pilocytic astrocytoma. Testing was not performed for the BRAF/KIAA1549 fusion or BRAFV600E mutation. ...Targeted sequencing of tissue from the right gluteal mass, revealed a …
Magnetic resonance imaging revealed a large suprasellar mass, which was debulked and found to be a pilocytic astrocytoma. Testing was …
Identification of a single cell-based signature for predicting prognosis risk and immunotherapy response in patients with glioblastoma.
Dai Z, Zhang N, Zhou R, Zhang H, Zhang L, Wang Z, Zeng W, Luo P, Zhang J, Liu Z, Cheng Q. Dai Z, et al. Clin Immunol. 2023 Jun;251:109345. doi: 10.1016/j.clim.2023.109345. Epub 2023 Apr 24. Clin Immunol. 2023. PMID: 37100336
Samples having different malignant biological hallmarks were distinguished by the algorithm, with the high gene pair score group featuring classic copy number variations, oncogenic mutations, and extensive hypomethylation, mediating poor prognosis. The increased gene pair …
Samples having different malignant biological hallmarks were distinguished by the algorithm, with the high gene pair score group featuring c …
Glioblastoma care in the elderly.
Jordan JT, Gerstner ER, Batchelor TT, Cahill DP, Plotkin SR. Jordan JT, et al. Cancer. 2016 Jan 15;122(2):189-97. doi: 10.1002/cncr.29742. Epub 2015 Nov 30. Cancer. 2016. PMID: 26618888 Free article. Review.
In elderly patients with glioblastoma, there is a survival advantage for those who undergo maximal safe resection, which likely includes an incremental benefit with increasing completeness of resection. Radiotherapy extends survival in selected patients, and hypofractionat …
In elderly patients with glioblastoma, there is a survival advantage for those who undergo maximal safe resection, which likely includes an …
The clinical and molecular characteristics of progressive hypothalamic/optic pathway pilocytic astrocytoma.
Li X, Moreira DC, Bag AK, Qaddoumi I, Acharya S, Chiang J. Li X, et al. Neuro Oncol. 2023 Apr 6;25(4):750-760. doi: 10.1093/neuonc/noac241. Neuro Oncol. 2023. PMID: 36260562 Free PMC article.
BACKGROUND: Unresectable hypothalamic/optic pathway pilocytic astrocytoma (PA) often progresses despite multiple therapies. Identifying clinical and molecular characteristics of progressive tumors may aid in prognostication and treatment. ...CONCLUSIONS: We identified r …
BACKGROUND: Unresectable hypothalamic/optic pathway pilocytic astrocytoma (PA) often progresses despite multiple therapies. Identifyi …
Multicentric Low-Grade Gliomas.
Sridharan V, Urbanski LM, Bi WL, Thistle K, Miller MB, Ramkissoon S, Reardon DA, Dunn IF. Sridharan V, et al. World Neurosurg. 2015 Oct;84(4):1045-50. doi: 10.1016/j.wneu.2015.05.021. Epub 2015 May 22. World Neurosurg. 2015. PMID: 26004698
Three months later, the cerebellar lesion also acquired new contrast enhancement and was found to be a grade II astrocytoma following a supracerebellar infratentorial approach for resection. ...PDGFRA amplifications and IDH1 wild-type status may act in a concerted fashion …
Three months later, the cerebellar lesion also acquired new contrast enhancement and was found to be a grade II astrocytoma following …
Identification and validation of a novel prognostic signature based on mitochondria and oxidative stress related genes for glioblastoma.
Tong S, Xia M, Xu Y, Sun Q, Ye L, Yuan F, Wang Y, Cai J, Ye Z, Tian D. Tong S, et al. J Transl Med. 2023 Feb 22;21(1):136. doi: 10.1186/s12967-023-03970-6. J Transl Med. 2023. PMID: 36814293 Free PMC article.
BACKGROUND: Mitochondria represent a major source of reactive oxygen species (ROS) in cells, and the direct increase in ROS content is the primary cause of oxidative stress, which plays an important role in tumor proliferation, invasion, angiogenesis, and treatment. ...The …
BACKGROUND: Mitochondria represent a major source of reactive oxygen species (ROS) in cells, and the direct increase in ROS content i …
Glioblastoma cell populations with distinct oncogenic programs release podoplanin as procoagulant extracellular vesicles.
Tawil N, Bassawon R, Meehan B, Nehme A, Montermini L, Gayden T, De Jay N, Spinelli C, Chennakrishnaiah S, Choi D, Adnani L, Zeinieh M, Jabado N, Kleinman CL, Witcher M, Riazalhosseini Y, Key NS, Schiff D, Grover SP, Mackman N, Couturier CP, Petrecca K, Suvà ML, Patel A, Tirosh I, Najafabadi H, Rak J. Tawil N, et al. Blood Adv. 2021 Mar 23;5(6):1682-1694. doi: 10.1182/bloodadvances.2020002998. Blood Adv. 2021. PMID: 33720339 Free PMC article.
Although the molecular effectors of these changes are poorly understood, the upregulation of podoplanin (PDPN) by cancer cells has recently been linked to an increased risk for venous thromboembolism (VTE) in GBM patients. Therefore, regulation of this platelet-acti …
Although the molecular effectors of these changes are poorly understood, the upregulation of podoplanin (PDPN) by cancer cells has recently …
895 results