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Quoted phrase not found in phrase index: "Decreased 3-hydroxyacyl-CoA dehydrogenase level"
Page 1
Similar metabolic adaptations during exercise after low volume sprint interval and traditional endurance training in humans.
Burgomaster KA, Howarth KR, Phillips SM, Rakobowchuk M, Macdonald MJ, McGee SL, Gibala MJ. Burgomaster KA, et al. J Physiol. 2008 Jan 1;586(1):151-60. doi: 10.1113/jphysiol.2007.142109. Epub 2007 Nov 8. J Physiol. 2008. PMID: 17991697 Free PMC article. Clinical Trial.

Despite these differences, both protocols induced similar increases (P < 0.05) in mitochondrial markers for skeletal muscle CHO (pyruvate dehydrogenase E1alpha protein content) and lipid oxidation (3-hydroxyacyl CoA dehydrogenase maximal acti

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Despite these differences, both protocols induced similar increases (P < 0.05) in mitochondrial markers for skeletal muscle CHO (pyruvate

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Optimal dietary therapy of long-chain 3-hydroxyacyl-CoA dehydrogenase deficiency.
Gillingham MB, Connor WE, Matern D, Rinaldo P, Burlingame T, Meeuws K, Harding CO. Gillingham MB, et al. Mol Genet Metab. 2003 Jun;79(2):114-23. doi: 10.1016/s1096-7192(03)00073-8. Mol Genet Metab. 2003. PMID: 12809642 Free PMC article. Clinical Trial.
Current dietary therapy for long-chain 3-hydroxyacyl-CoA dehydrogenase (LCHAD) or trifunctional protein (TFP) deficiency consists of fasting avoidance, and limiting long-chain fatty acid (LCFA) intake. ...Subjects consumed an average of 11% of caloric …
Current dietary therapy for long-chain 3-hydroxyacyl-CoA dehydrogenase (LCHAD) or trifunctional protein (TFP) de …
Dietary management of long-chain 3-hydroxyacyl-CoA dehydrogenase deficiency (LCHADD). A case report and survey.
Gillingham M, Van Calcar S, Ney D, Wolff J, Harding C. Gillingham M, et al. J Inherit Metab Dis. 1999 Apr;22(2):123-31. doi: 10.1023/a:1005437616934. J Inherit Metab Dis. 1999. PMID: 10234607 Free PMC article.
Current dietary management of long-chain 3-hydroxyacyl-CoA dehydrogenase (LCHAD; long-chain-(S)-3-hydroxyacyl-CoA:NAD+ oxido-reductase, EC 1.1.1.211) deficiency (LCHADD) is based on avoiding fasting, and minimizing energy productio …
Current dietary management of long-chain 3-hydroxyacyl-CoA dehydrogenase (LCHAD; long-chain-(S)-3-hydr …
Effects of immobilization and aerobic training on proteins related to intramuscular substrate storage and metabolism in young and older men.
Vigelsø A, Gram M, Wiuff C, Hansen CN, Prats C, Dela F, Helge JW. Vigelsø A, et al. Eur J Appl Physiol. 2016 Mar;116(3):481-94. doi: 10.1007/s00421-015-3302-x. Epub 2015 Dec 1. Eur J Appl Physiol. 2016. PMID: 26626913
The biopsies were analyzed for muscle substrates; muscle perilipin protein (PLIN), glycogen synthase (GS), synaptosomal-associated protein of 23 kDa (SNAP23) protein content, and muscle 3-hydroxyacyl-CoA dehydrogenase (HAD) activity RESULTS: The older …
The biopsies were analyzed for muscle substrates; muscle perilipin protein (PLIN), glycogen synthase (GS), synaptosomal-associated protein o …
3-Hydroxyacyl-coenzyme A dehydrogenase deficiency and hyperinsulinemic hypoglycemia: characterization of a novel mutation and severe dietary protein sensitivity.
Kapoor RR, James C, Flanagan SE, Ellard S, Eaton S, Hussain K. Kapoor RR, et al. J Clin Endocrinol Metab. 2009 Jul;94(7):2221-5. doi: 10.1210/jc.2009-0423. Epub 2009 May 5. J Clin Endocrinol Metab. 2009. PMID: 19417036 Free PMC article.
BACKGROUND: HADH encodes for the enzyme 3-hydroxyacyl-coenzyme A dehydrogenase (HADH) and catalyses the penultimate reaction in the beta-oxidation of fatty acids. ...Sequencing of the HADH gene identified a homozygous missense mutation (c.562A>G; p.Met188Val). Hydroxyac …
BACKGROUND: HADH encodes for the enzyme 3-hydroxyacyl-coenzyme A dehydrogenase (HADH) and catalyses the penultimate reaction in the b …
Metabolic control during exercise with and without medium-chain triglycerides (MCT) in children with long-chain 3-hydroxy acyl-CoA dehydrogenase (LCHAD) or trifunctional protein (TFP) deficiency.
Gillingham MB, Scott B, Elliott D, Harding CO. Gillingham MB, et al. Mol Genet Metab. 2006 Sep-Oct;89(1-2):58-63. doi: 10.1016/j.ymgme.2006.06.004. Epub 2006 Jul 27. Mol Genet Metab. 2006. PMID: 16876451 Free PMC article.
Exercise induced rhabdomyolysis is a complication of long-chain 3-hydroxyacyl-CoA dehydrogenase (LCHAD) and mitochondrial trifunctional protein (TFP) deficiency that frequently leads to exercise avoidance. ...Creatine kinase and lactate levels d …
Exercise induced rhabdomyolysis is a complication of long-chain 3-hydroxyacyl-CoA dehydrogenase (LCHAD) and mito …
Exercise training, but not resveratrol, improves metabolic and inflammatory status in skeletal muscle of aged men.
Olesen J, Gliemann L, Biensø R, Schmidt J, Hellsten Y, Pilegaard H. Olesen J, et al. J Physiol. 2014 Apr 15;592(8):1873-86. doi: 10.1113/jphysiol.2013.270256. Epub 2014 Feb 10. J Physiol. 2014. PMID: 24514907 Free PMC article. Clinical Trial.
Exercise training increased skeletal muscle peroxisome proliferator-activated receptor-gamma co-activator-1alpha mRNA ~1.5-fold, cytochrome c protein ~1.3-fold, cytochrome c oxidase I protein ~1.5-fold, citrate synthase activity ~1.3-fold, 3-hydroxyacyl-CoA …
Exercise training increased skeletal muscle peroxisome proliferator-activated receptor-gamma co-activator-1alpha mRNA ~1.5-fold, cytochrome …
Garlic provides protection to mice heart against isoproterenol-induced oxidative damage: role of nitric oxide.
Khatua TN, Padiya R, Karnewar S, Kuncha M, Agawane SB, Kotamraju S, Banerjee SK. Khatua TN, et al. Nitric Oxide. 2012 Jun 30;27(1):9-17. doi: 10.1016/j.niox.2012.03.004. Epub 2012 Mar 28. Nitric Oxide. 2012. PMID: 22484451
While no change in serum SGPT and SGOT level, a significant decrease in serum LDH level was observed after garlic feeding. Garlic-induced NO formation was further confirmed in human aortic endothelial cells (HAEC). Administration of isoproterenol caused a sig …
While no change in serum SGPT and SGOT level, a significant decrease in serum LDH level was observed after garlic feedi …
Gene expression of myogenic factors and phenotype-specific markers in electrically stimulated muscle of paraplegics.
Vissing K, Andersen JL, Harridge SD, Sandri C, Hartkopp A, Kjaer M, Schjerling P. Vissing K, et al. J Appl Physiol (1985). 2005 Jul;99(1):164-72. doi: 10.1152/japplphysiol.01172.2004. Epub 2005 Mar 3. J Appl Physiol (1985). 2005. PMID: 15746295 Free article. Clinical Trial.
The training evoked the expected mRNA increase for slow fiber-specific markers myosin heavy chain I and 3-hydroxyacyl-CoA dehydrogenase A, whereas an mRNA decrease was seen for fast fiber-specific markers myosin heavy chain IIx and glycerol phos …
The training evoked the expected mRNA increase for slow fiber-specific markers myosin heavy chain I and 3-hydroxyacyl-CoA …
Beta-adrenergic blockade and training in human subjects: effects on muscle metabolic capacity.
Svedenhag J, Henriksson J, Juhlin-Dannfelt A. Svedenhag J, et al. Am J Physiol. 1984 Sep;247(3 Pt 1):E305-11. doi: 10.1152/ajpendo.1984.247.3.E305. Am J Physiol. 1984. PMID: 6089581 Clinical Trial.
The activities of the mitochondrial enzymes citrate synthase (CS), succinate dehydrogenase (SDH), cytochrome c oxidase (Cyt-c-ox), and 3-hydroxyacyl-CoA dehydrogenase (HAD) in the quadriceps femoris muscle increased significantly (P less than 0. …
The activities of the mitochondrial enzymes citrate synthase (CS), succinate dehydrogenase (SDH), cytochrome c oxidase (Cyt-c-ox), an …
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